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Incomplete inhibition by eculizumab: mechanistic evidence for residual C5 activity during strong complement activation

Blood, 2017-02, Vol.129 (8), p.970-980

2017

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RhCE protein variants in Southwestern Germany detected by serologic routine testing

Transfusion (Philadelphia, Pa.), 2009-09, Vol.49 (9), p.1793-1802

2009

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Six years' experience performing RHD genotyping to confirm D− red blood cell units in Germany for preventing anti-D immunizations

Transfusion (Philadelphia, Pa.), 2009-03, Vol.49 (3), p.465-471

2009

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Complement activation by human red blood cell antibodies: hemolytic potential of antibodies and efficacy of complement inhibitors assessed by a sensitive flow cytometric assay

Transfusion (Philadelphia, Pa.), 2018-12, Vol.58 (12), p.2992-3002

2018

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D variants at the RhD vestibule in the weak D type 4 and Eurasian D clusters

Transfusion (Philadelphia, Pa.), 2009-06, Vol.49 (6), p.1059-1069

2009

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Specific amino acid substitutions cause distinct expression of JAL (RH48) and JAHK (RH53) antigens in RhCE and not in RhD

Transfusion (Philadelphia, Pa.), 2010-01, Vol.50 (1), p.267-269

2010

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The Bloodgen Project of the European Union, 2003–2009

Transfusion medicine and hemotherapy, 2009-01, Vol.36 (3), p.162-167

2009

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D category IV: a group of clinically relevant and phylogenetically diverse partial D

Transfusion (Philadelphia, Pa.), 2013-11, Vol.53 (11pt2), p.2960-2973

2013

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An update on the CD59 blood group system

Immunohematology, 2019-01, Vol.35 (1), p.7-8

2019

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A new blood group antigen is defined by anti-CD59, detected in a CD59-deficient patient

Transfusion (Philadelphia, Pa.), 2014-07, Vol.54 (7), p.1817-1822

2014

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CD59: A long-known complement inhibitor has advanced to a blood group system

Immunohematology, 2015-01, Vol.31 (4), p.145-151

2015

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A new blood group antigen is defined by anti‐ CD 59, detected in a CD 59‐deficient patient

Transfusion (Philadelphia, Pa.), 2014-07, Vol.54 (7), p.1817-1822

2014

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D category IV: a group of clinically relevant and phylogenetically diverse partial D : Twenty Years since the Cloning of the Blood Group Genes

Transfusion (Philadelphia, Pa.), 2013, Vol.53 (11), p.2960-2973

2013

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Molecular basis of two novel and related high-prevalence antigens in the Kell blood group system, KUCI and KANT, and their serologic and spatial association with K11 and KETI

Transfusion (Philadelphia, Pa.), 2013-11, Vol.53 (11pt2), p.2872-2881

2013

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Evaluation of factor H (FH), miniFH and the Fc-fusion protein Fc-miniFH in pharmacokinetic studies and different in vitro models of complement mediated diseases

Immunobiology (1979), 2016-10, Vol.221 (10), p.1217-1218

2016

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Mechanistic evidence for incomplete terminal pathway inhibition under eculizumab during strong complement activation

Immunobiology (1979), 2016-10, Vol.221 (10), p.1216-1216

2016

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IVS5-38del4 deletion in the RHD gene does not cause a DEL phenotype: relevance for RHD alleles including DFR-3

Transfusion (Philadelphia, Pa.), 2007-08, Vol.47 (8), p.1552-1555

2007

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Molecular basis of two novel and related high‐prevalence antigens in the K ell blood group system, KUCI and KANT , and their serologic and spatial association with K 11 and KETI

Transfusion (Philadelphia, Pa.), 2013-11, Vol.53 (11pt2), p.2872-2881

2013

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The BloodGen project: toward mass-scale comprehensive genotyping of blood donors in the European Union and beyond

Transfusion (Philadelphia, PA), 2007-07, Vol.47 (s1), p.40S-46S

2007

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Molecular basis of two novel and related high-prevalence antigens in the Kell blood group system, KUCI and KANT, and their serological and spatial association with K11 and KETI

Transfusion (Philadelphia, Pa.), 2013-04, Vol.53 (11 0 2)

2013

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Molecular basis of two novel and related high-prevalence antigens in the Kell blood group system, KUCI and KANT, and their serologic and spatial association with K11 and KETI : Twenty Years since the Cloning of the Blood Group Genes

Transfusion (Philadelphia, Pa.), 2013, Vol.53 (11), p.2872-2881

2013

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DCS-1, DCS-2, and DFV share amino acid substitutions at the extracellular RhD protein vestibule

Transfusion (Philadelphia, Pa.), 2008-01, Vol.48 (1), p.25-33

2008

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The Amino‐Acid Sequence of Porcine Adenylate Kinase from Skeletal Muscle

European journal of biochemistry, 1974-03, Vol.43 (1), p.131-144

1974

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Activation of the alternative pathway of human complement by sulfhydryl compounds of analytic and therapeutic use

International archives of allergy and applied immunology, 1987-01, Vol.84 (2), p.178-184

1987

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Crystalline Adenylate Kinase from Carp Muscle

European journal of biochemistry, 1975-02, Vol.51 (1), p.229-235

1975

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Incomplete inhibition by eculizumab: mechanistic evidence for residual C5 activity during strong complement activation

RhCE protein variants in Southwestern Germany detected by serologic routine testing

Six years' experience performing RHD genotyping to confirm D− red blood cell units in Germany for preventing anti-D immunizations

Complement activation by human red blood cell antibodies: hemolytic potential of antibodies and efficacy of complement inhibitors assessed by a sensitive flow cytometric assay

D variants at the RhD vestibule in the weak D type 4 and Eurasian D clusters

Specific amino acid substitutions cause distinct expression of JAL (RH48) and JAHK (RH53) antigens in RhCE and not in RhD

The Bloodgen Project of the European Union, 2003–2009

D category IV: a group of clinically relevant and phylogenetically diverse partial D

An update on the CD59 blood group system

A new blood group antigen is defined by anti-CD59, detected in a CD59-deficient patient

CD59: A long-known complement inhibitor has advanced to a blood group system

A new blood group antigen is defined by anti‐ CD 59, detected in a CD 59‐deficient patient

D category IV: a group of clinically relevant and phylogenetically diverse partial D : Twenty Years since the Cloning of the Blood Group Genes

Molecular basis of two novel and related high-prevalence antigens in the Kell blood group system, KUCI and KANT, and their serologic and spatial association with K11 and KETI

Evaluation of factor H (FH), miniFH and the Fc-fusion protein Fc-miniFH in pharmacokinetic studies and different in vitro models of complement mediated diseases

Mechanistic evidence for incomplete terminal pathway inhibition under eculizumab during strong complement activation

IVS5-38del4 deletion in the RHD gene does not cause a DEL phenotype: relevance for RHD alleles including DFR-3

Molecular basis of two novel and related high‐prevalence antigens in the K ell blood group system, KUCI and KANT , and their serologic and spatial association with K 11 and KETI

The BloodGen project: toward mass-scale comprehensive genotyping of blood donors in the European Union and beyond

Molecular basis of two novel and related high-prevalence antigens in the Kell blood group system, KUCI and KANT, and their serological and spatial association with K11 and KETI

Molecular basis of two novel and related high-prevalence antigens in the Kell blood group system, KUCI and KANT, and their serologic and spatial association with K11 and KETI : Twenty Years since the Cloning of the Blood Group Genes

DCS-1, DCS-2, and DFV share amino acid substitutions at the extracellular RhD protein vestibule

The Amino‐Acid Sequence of Porcine Adenylate Kinase from Skeletal Muscle

Activation of the alternative pathway of human complement by sulfhydryl compounds of analytic and therapeutic use

Crystalline Adenylate Kinase from Carp Muscle

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