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Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart

American journal of physiology. Heart and circulatory physiology, 2016-09, Vol.311 (3), p.H713-H724

2016

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Stimulation of NOX2 in isolated hearts reversibly sensitizes RyR2 channels to activation by cytoplasmic calcium

Journal of molecular and cellular cardiology, 2014-03, Vol.68, p.38-46

2014

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Phospholamban phosphorylation by CaMKII under pathophysiological conditions

Frontiers in bioscience, 2008-05, Vol.13, p.5988

2008

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Increased Na⁺/Ca²⁺ exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive rats

PloS one, 2014-04, Vol.9 (4), p.e96400-e96400

2014

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Time Course and Mechanisms of Phosphorylation of Phospholamban Residues in Ischemia-reperfused Rat Hearts. Dissociation of Phospholamban Phosphorylation Pathways

Journal of molecular and cellular cardiology, 2002-01, Vol.34 (1), p.39-50

2002

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Increased propensity to cardiac alternans in spontaneously hypertensive rats at early stages is associated to prolonged sarcoplasmic reticulum Ca release refractoriness and t-tubule remodeling

Journal of molecular and cellular cardiology, 2022-12, Vol.173, p.28-29

2022

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Frequency-dependent acceleration of relaxation in mammalian heart: a property not relying on phospholamban and SERCA2a phosphorylation

The Journal of physiology, 2005-02, Vol.562 (3), p.801-813

2005

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Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca2+ handling and contractile recovery of stunned myocardium

European journal of pharmacology, 2022-01, Vol.914, p.174665-174665, Article 174665

2022

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Immunodetection of Phosphorylation Sites Gives New Insights into the Mechanisms Underlying Phospholamban Phosphorylation in the Intact Heart

The Journal of biological chemistry, 1996-12, Vol.271 (52), p.33561-33567

1996

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Mechanisms Involved in the Acidosis Enhancement of the Isoproterenol-induced Phosphorylation of Phospholamban in the Intact Heart

The Journal of biological chemistry, 1998-04, Vol.273 (16), p.9804-9811

1998

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Chemical chaperones improve the functional recovery of stunned myocardium by attenuating the endoplasmic reticulum stress

Acta Physiologica, 2020-02, Vol.228 (2), p.e13358-n/a

2020

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Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca 2+ handling and contractile recovery of stunned myocardium

European journal of pharmacology, 2022-01, Vol.914, p.174665

2022

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Role of CaMKII in post acidosis arrhythmias: A simulation study using a human myocyte model

Journal of molecular and cellular cardiology, 2013-07, Vol.60, p.172-183

2013

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Ca2+/calmodulin-dependent protein kinase: a key component in the contractile recovery from acidosis

Cardiovascular research, 2007-03, Vol.73 (4), p.648-656

2007

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Endoplasmic reticulum contribution to the relaxant effect of cGMP- and cAMP-elevating agents in feline aorta

American journal of physiology. Heart and circulatory physiology, 2000-06, Vol.278 (6), p.H1856-H1865

2000

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Ca2+ /calmodulin kinase II increases ryanodine binding and Ca2+ -induced sarcoplasmic reticulum Ca2+ release kinetics during β-adrenergic stimulation

Journal of molecular and cellular cardiology, 2007-09, Vol.43 (3), p.281-291

2007

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Ca 2+ -Calmodulin–Dependent Protein Kinase Phosphorylation of Ryanodine Receptor May Contribute to the β-Adrenergic Regulation of Myocardial Contractility Independently of Increases in Heart Rate

Circulation research, 2008-07, Vol.103 (1)

2008

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Increased [Na.sup.+]/[Ca.sup.2+] exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive rats

PloS one, 2014-04, Vol.9 (4)

2014

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Ca2+-Calmodulin–Dependent Protein Kinase Phosphorylation of Ryanodine Receptor May Contribute to the β-Adrenergic Regulation of Myocardial Contractility Independently of Increases in Heart Rate

Circulation research, 2008-07, Vol.103 (1), p.e10-e11

2008

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Ca 2+ /calmodulin-dependent protein kinase: A key component in the contractile recovery from acidosis

Cardiovascular research, 2007-03, Vol.73 (4), p.648-656

2007

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Subchronic microcystin-lr exposure increased hepatic apoptosis and induced compensatory mechanisms in mice

Journal of biochemical and molecular toxicology, 2012-03, Vol.26 (4), p.131-138

2012

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Ca2+ /calmodulin -dependent protein kinase : A key component in the contractile recovery from acidosis : Calmodulin and Ca2+/calmodulin kinase in the heart

Cardiovascular research, 2007, Vol.73 (4), p.648-656

2007

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β2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heart

Naunyn-Schmiedeberg's archives of pharmacology, 2006-04, Vol.373 (1), p.60-70

2006

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beta 2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heart

Naunyn-Schmiedeberg's archives of pharmacology, 2006-04, Vol.373 (1), p.60-70

2006

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Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosis

Cardiovascular research, 2005-04, Vol.66 (1), p.114-122

2005

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Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart

Stimulation of NOX2 in isolated hearts reversibly sensitizes RyR2 channels to activation by cytoplasmic calcium

Phospholamban phosphorylation by CaMKII under pathophysiological conditions

Increased Na⁺/Ca²⁺ exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive rats

Time Course and Mechanisms of Phosphorylation of Phospholamban Residues in Ischemia-reperfused Rat Hearts. Dissociation of Phospholamban Phosphorylation Pathways

Increased propensity to cardiac alternans in spontaneously hypertensive rats at early stages is associated to prolonged sarcoplasmic reticulum Ca release refractoriness and t-tubule remodeling

Frequency-dependent acceleration of relaxation in mammalian heart: a property not relying on phospholamban and SERCA2a phosphorylation

Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca2+ handling and contractile recovery of stunned myocardium

Immunodetection of Phosphorylation Sites Gives New Insights into the Mechanisms Underlying Phospholamban Phosphorylation in the Intact Heart

Mechanisms Involved in the Acidosis Enhancement of the Isoproterenol-induced Phosphorylation of Phospholamban in the Intact Heart

Chemical chaperones improve the functional recovery of stunned myocardium by attenuating the endoplasmic reticulum stress

Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca 2+ handling and contractile recovery of stunned myocardium

Role of CaMKII in post acidosis arrhythmias: A simulation study using a human myocyte model

Ca2+/calmodulin-dependent protein kinase: a key component in the contractile recovery from acidosis

Endoplasmic reticulum contribution to the relaxant effect of cGMP- and cAMP-elevating agents in feline aorta

Ca2+ /calmodulin kinase II increases ryanodine binding and Ca2+ -induced sarcoplasmic reticulum Ca2+ release kinetics during β-adrenergic stimulation

Ca 2+ -Calmodulin–Dependent Protein Kinase Phosphorylation of Ryanodine Receptor May Contribute to the β-Adrenergic Regulation of Myocardial Contractility Independently of Increases in Heart Rate

Increased [Na.sup.+]/[Ca.sup.2+] exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive rats

Ca2+-Calmodulin–Dependent Protein Kinase Phosphorylation of Ryanodine Receptor May Contribute to the β-Adrenergic Regulation of Myocardial Contractility Independently of Increases in Heart Rate

Ca 2+ /calmodulin-dependent protein kinase: A key component in the contractile recovery from acidosis

Subchronic microcystin-lr exposure increased hepatic apoptosis and induced compensatory mechanisms in mice

Ca2+ /calmodulin -dependent protein kinase : A key component in the contractile recovery from acidosis : Calmodulin and Ca2+/calmodulin kinase in the heart

β2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heart

beta 2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heart

Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosis

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