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Loss of the RNA-binding protein TACO1 causes late-onset mitochondrial dysfunction in mice

Nature communications, 2016-06, Vol.7 (1), p.11884-11884, Article 11884

2016

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Cytoskeletal disarray increases arrhythmogenic vulnerability during sympathetic stimulation in a model of hypertrophic cardiomyopathy

Scientific reports, 2023-07, Vol.13 (1), p.11296-11296, Article 11296

2023

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Cellular and Molecular Changes to Cortical Neurons Following Low Intensity Repetitive Magnetic Stimulation at Different Frequencies

Brain stimulation, 2015, Vol.8 (1), p.114-123

2015

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Impaired functional communication between the L-type calcium channel and mitochondria contributes to metabolic inhibition in the mdx heart

Proceedings of the National Academy of Sciences - PNAS, 2014-07, Vol.111 (28), p.E2905-E2914

2014

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Transient Exposure to Hydrogen Peroxide Causes an Increase in Mitochondria-Derived Superoxide As a Result of Sustained Alteration in L-Type Ca2+ Channel Function in the Absence of Apoptosis in Ventricular Myocytes

Circulation research, 2007-04, Vol.100 (7), p.1036-1044

2007

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Cav1.2 calcium channel is glutathionylated during oxidative stress in guinea pig and ischemic human heart

Free radical biology & medicine, 2011-10, Vol.51 (8), p.1501-1511

2011

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PTCD1 Is Required for 16S rRNA Maturation Complex Stability and Mitochondrial Ribosome Assembly

Cell reports (Cambridge), 2018-04, Vol.23 (1), p.127-142

2018

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Mutation in MRPS34 compromises protein synthesis and causes mitochondrial dysfunction

PLoS genetics, 2015-03, Vol.11 (3), p.e1005089-e1005089

2015

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Evidence for regulation of mitochondrial function by the L-type Ca2+ channel in ventricular myocytes

Journal of molecular and cellular cardiology, 2009-06, Vol.46 (6), p.1016-1026

2009

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Qo site of mitochondrial complex III is the source of increased superoxide after transient exposure to hydrogen peroxide

Journal of molecular and cellular cardiology, 2010-11, Vol.49 (5), p.875-885

2010

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How Does Calcium Regulate Mitochondrial Energetics in the Heart? – New Insights

Heart, lung & circulation, 2014-07, Vol.23 (7), p.602-609

2014

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Targeting calcium and the mitochondria in prevention of pathology in the heart

Current drug targets, 2011-05, Vol.12 (5), p.748-760

2011

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Examining Efficacy of “TAT-less” Delivery of a Peptide against the L‑Type Calcium Channel in Cardiac Ischemia–Reperfusion Injury

ACS nano, 2013-03, Vol.7 (3), p.2212-2220

2013

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A maladaptive feedback mechanism between the extracellular matrix and cytoskeleton contributes to hypertrophic cardiomyopathy pathophysiology

Communications biology, 2023-01, Vol.6 (1), p.4-4, Article 4

2023

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Impaired calcium handling and mitochondrial metabolic dysfunction as early markers of hypertrophic cardiomyopathy

Archives of biochemistry and biophysics, 2019-04, Vol.665, p.166-174

2019

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Fidelity and coordination of mitochondrial protein synthesis in health and disease

The Journal of physiology, 2021-07, Vol.599 (14), p.3449-3462

2021

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Real-Time Bioimpedance Sensing of Antifibrotic Drug Action in Primary Human Cells

ACS sensors, 2017-10, Vol.2 (10), p.1482-1490

2017

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Concerted regulation of mitochondrial and nuclear non‐coding RNAs by a dual‐targeted RNase Z

EMBO reports, 2018-10, Vol.19 (10), p.n/a

2018

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A Morpholino Oligomer Therapy Regime That Restores Mitochondrial Function and Prevents mdx Cardiomyopathy

JACC. Basic to translational science, 2018-06, Vol.3 (3), p.391-402

2018

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Characterization and validation of a preventative therapy for hypertrophic cardiomyopathy in a murine model of the disease

Proceedings of the National Academy of Sciences - PNAS, 2020-09, Vol.117 (37), p.23113-23124

2020

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Nanoparticle-Mediated Dual Delivery of an Antioxidant and a Peptide against the L‑Type Ca2+ Channel Enables Simultaneous Reduction of Cardiac Ischemia-Reperfusion Injury

ACS nano, 2015-01, Vol.9 (1), p.279-289

2015

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Mitochondrial mistranslation modulated by metabolic stress causes cardiovascular disease and reduced lifespan

Aging cell, 2021-07, Vol.20 (7), p.e13408-n/a

2021

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A dendronized polymer variant that facilitates safe delivery of a calcium channel antagonist to the heart

Nanomedicine, 2020-10, Vol.29, p.102264, Article 102264

2020

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Verapamil is Less Effective than Triamcinolone for Prevention of Keloid Scar Recurrence After Excision in a Randomized Controlled Trial

Acta dermato-venereologica, 2016-08, Vol.96 (6), p.774-778

2016

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L-type Ca(2+) channel contributes to alterations in mitochondrial calcium handling in the mdx ventricular myocyte

American journal of physiology. Heart and circulatory physiology, 2013-03, Vol.304 (6), p.H767-H775

2013

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Loss of the RNA-binding protein TACO1 causes late-onset mitochondrial dysfunction in mice

Cytoskeletal disarray increases arrhythmogenic vulnerability during sympathetic stimulation in a model of hypertrophic cardiomyopathy

Cellular and Molecular Changes to Cortical Neurons Following Low Intensity Repetitive Magnetic Stimulation at Different Frequencies

Impaired functional communication between the L-type calcium channel and mitochondria contributes to metabolic inhibition in the mdx heart

Transient Exposure to Hydrogen Peroxide Causes an Increase in Mitochondria-Derived Superoxide As a Result of Sustained Alteration in L-Type Ca2+ Channel Function in the Absence of Apoptosis in Ventricular Myocytes

Cav1.2 calcium channel is glutathionylated during oxidative stress in guinea pig and ischemic human heart

PTCD1 Is Required for 16S rRNA Maturation Complex Stability and Mitochondrial Ribosome Assembly

Mutation in MRPS34 compromises protein synthesis and causes mitochondrial dysfunction

Evidence for regulation of mitochondrial function by the L-type Ca2+ channel in ventricular myocytes

Qo site of mitochondrial complex III is the source of increased superoxide after transient exposure to hydrogen peroxide

How Does Calcium Regulate Mitochondrial Energetics in the Heart? – New Insights

Targeting calcium and the mitochondria in prevention of pathology in the heart

Examining Efficacy of “TAT-less” Delivery of a Peptide against the L‑Type Calcium Channel in Cardiac Ischemia–Reperfusion Injury

A maladaptive feedback mechanism between the extracellular matrix and cytoskeleton contributes to hypertrophic cardiomyopathy pathophysiology

Impaired calcium handling and mitochondrial metabolic dysfunction as early markers of hypertrophic cardiomyopathy

Fidelity and coordination of mitochondrial protein synthesis in health and disease

Real-Time Bioimpedance Sensing of Antifibrotic Drug Action in Primary Human Cells

Concerted regulation of mitochondrial and nuclear non‐coding RNAs by a dual‐targeted RNase Z

A Morpholino Oligomer Therapy Regime That Restores Mitochondrial Function and Prevents mdx Cardiomyopathy

Characterization and validation of a preventative therapy for hypertrophic cardiomyopathy in a murine model of the disease

Nanoparticle-Mediated Dual Delivery of an Antioxidant and a Peptide against the L‑Type Ca2+ Channel Enables Simultaneous Reduction of Cardiac Ischemia-Reperfusion Injury

Mitochondrial mistranslation modulated by metabolic stress causes cardiovascular disease and reduced lifespan

A dendronized polymer variant that facilitates safe delivery of a calcium channel antagonist to the heart

Verapamil is Less Effective than Triamcinolone for Prevention of Keloid Scar Recurrence After Excision in a Randomized Controlled Trial

L-type Ca(2+) channel contributes to alterations in mitochondrial calcium handling in the mdx ventricular myocyte

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