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Calcium channel ITPR2 and mitochondria-ER contacts promote cellular senescence and aging

Nature communications, 2021-02, Vol.12 (1), p.720-720, Article 720

2021

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Endoplasmic reticulum calcium release through ITPR2 channels leads to mitochondrial calcium accumulation and senescence

Nature communications, 2014-05, Vol.5 (1), p.3792-3792, Article 3792

2014

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Emerging roles of chemokines in prostate cancer

Endocrine-related cancer, 2009-09, Vol.16 (3), p.663-673

2009

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Targeting the phospholipase A2 receptor ameliorates premature aging phenotypes

Aging cell, 2018-12, Vol.17 (6), p.e12835-n/a

2018

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mTOR pathway activation drives lung cell senescence and emphysema

JCI insight, 2018-02, Vol.3 (3)

2018

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Potential role of estrogen receptor beta as a tumor suppressor of epithelial ovarian cancer

PloS one, 2012-09, Vol.7 (9), p.e44787

2012

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Generation of a conditional transgenic mouse model expressing human Phospholipase A2 Receptor 1

Scientific reports, 2020-05, Vol.10 (1), p.8190-8190, Article 8190

2020

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PLA2R1 Mediates Tumor Suppression by Activating JAK2

Cancer research (Chicago, Ill.), 2013-10, Vol.73 (20), p.6334-6345

2013

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Lysyl oxidase family activity promotes resistance of pancreatic ductal adenocarcinoma to chemotherapy by limiting the intratumoral anticancer drug distribution

Oncotarget, 2016-05, Vol.7 (22), p.32100-32112

2016

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Potassium Channel KCNA1 Modulates Oncogene-Induced Senescence and Transformation

Cancer research (Chicago, Ill.), 2013-08, Vol.73 (16), p.5253-5265

2013

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PLA2R1 promotes DNA damage and inhibits spontaneous tumor formation during aging

Cell death & disease, 2021-02, Vol.12 (2), p.190-190, Article 190

2021

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PLA2R1: Expression and function in cancer

Biochimica et biophysica acta, 2014-08, Vol.1846 (1), p.40-44

2014

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Targeting chemoresistant senescent pancreatic cancer cells improves conventional treatment efficacy

Molecular biomedicine, 2023-02, Vol.4 (1), p.4-4, Article 4

2023

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Repression of PLA2R1 by c-MYC and HIF-2alpha promotes cancer growth

Oncotarget, 2014-02, Vol.5 (4), p.1004-1013

2014

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Coxsackie and adenovirus receptor is a target and a mediator of estrogen action in breast cancer

Endocrine-related cancer, 2011-06, Vol.18 (3), p.311-321

2011

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Down-regulation of DcR2 sensitizes androgen-dependent prostate cancer LNCaP cells to TRAIL-induced apoptosis

Cancer cell international, 2011-12, Vol.11 (1), p.42-42, Article 42

2011

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Transformed cells after senescence give rise to more severe tumor phenotypes than transformed non-senescent cells

Cancer letters, 2022-10, Vol.546, p.215850, Article 215850

2022

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Transformed cells after senescence give rise to more severe tumor phenotypes than transformed non-senescent cells

Cancer letters, 2022-10, Vol.546, p.215850-215850, Article 215850

2022

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Phospholipase A2 receptor 1 promotes lung cell senescence and emphysema in obstructive lung disease

The European respiratory journal, 2021-08, Vol.58 (2), p.2000752

2021

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NF‐κB‐dependent secretome of senescent cells can trigger neuroendocrine transdifferentiation of breast cancer cells

Aging cell, 2022-07, Vol.21 (7)

2022

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Benidipine calcium channel blocker promotes the death of cigarette smoke-induced senescent cells and improves lung emphysema

Aging (Albany, NY.), 2023-12, Vol.15 (23), p.13581-13592

2023

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Loss of Pla2r1 decreases cellular senescence and age‐related alterations caused by aging and Western diets

Aging cell, 2023-11, Vol.22 (11), p.e13971-e13971

2023

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Screening of a kinase library reveals novel pro-senescence kinases and their common NF-κB-dependent transcriptional program

Aging (Albany, NY.), 2015-11, Vol.7 (11), p.986-999

2015

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NF‐κB‐dependent secretome of senescent cells can trigger neuroendocrine transdifferentiation of breast cancer cells

Aging cell, 2022-07, Vol.21 (7), p.e13632-n/a

2022

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MUC1 drives epithelial–mesenchymal transition in renal carcinoma through Wnt/β-catenin pathway and interaction with SNAIL promoter

Cancer letters, 2014-05, Vol.346 (2), p.225-236

2014

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Calcium channel ITPR2 and mitochondria-ER contacts promote cellular senescence and aging

Endoplasmic reticulum calcium release through ITPR2 channels leads to mitochondrial calcium accumulation and senescence

Emerging roles of chemokines in prostate cancer

Targeting the phospholipase A2 receptor ameliorates premature aging phenotypes

mTOR pathway activation drives lung cell senescence and emphysema

Potential role of estrogen receptor beta as a tumor suppressor of epithelial ovarian cancer

Generation of a conditional transgenic mouse model expressing human Phospholipase A2 Receptor 1

PLA2R1 Mediates Tumor Suppression by Activating JAK2

Lysyl oxidase family activity promotes resistance of pancreatic ductal adenocarcinoma to chemotherapy by limiting the intratumoral anticancer drug distribution

Potassium Channel KCNA1 Modulates Oncogene-Induced Senescence and Transformation

PLA2R1 promotes DNA damage and inhibits spontaneous tumor formation during aging

PLA2R1: Expression and function in cancer

Targeting chemoresistant senescent pancreatic cancer cells improves conventional treatment efficacy

Repression of PLA2R1 by c-MYC and HIF-2alpha promotes cancer growth

Coxsackie and adenovirus receptor is a target and a mediator of estrogen action in breast cancer

Down-regulation of DcR2 sensitizes androgen-dependent prostate cancer LNCaP cells to TRAIL-induced apoptosis

Transformed cells after senescence give rise to more severe tumor phenotypes than transformed non-senescent cells

Transformed cells after senescence give rise to more severe tumor phenotypes than transformed non-senescent cells

Phospholipase A2 receptor 1 promotes lung cell senescence and emphysema in obstructive lung disease

NF‐κB‐dependent secretome of senescent cells can trigger neuroendocrine transdifferentiation of breast cancer cells

Benidipine calcium channel blocker promotes the death of cigarette smoke-induced senescent cells and improves lung emphysema

Loss of Pla2r1 decreases cellular senescence and age‐related alterations caused by aging and Western diets

Screening of a kinase library reveals novel pro-senescence kinases and their common NF-κB-dependent transcriptional program

NF‐κB‐dependent secretome of senescent cells can trigger neuroendocrine transdifferentiation of breast cancer cells

MUC1 drives epithelial–mesenchymal transition in renal carcinoma through Wnt/β-catenin pathway and interaction with SNAIL promoter

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