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Arginase-1 expressing microglia in close proximity to motor neurons were increased early in disease progression in canine degenerative myelopathy, a model of amyotrophic lateral sclerosis

Molecular and cellular neuroscience, 2018-04, Vol.88, p.148-157

2018

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AAV9-mediated delivery of miR-23a reduces disease severity in Smn2B/−SMA model mice

Human molecular genetics, 2019-10, Vol.28 (19), p.3199-3210

2019

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Survival motor neuron deficiency slows myoblast fusion through reduced myomaker and myomixer expression

Journal of cachexia, sarcopenia and muscle, 2021-08, Vol.12 (4), p.1098-1116

2021

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AAV9-DOK7 gene therapy reduces disease severity in Smn SMA model mice

Biochemical and biophysical research communications, 2020-09, Vol.530 (1), p.107-114

2020

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Muscle spindle alterations precede onset of sensorimotor deficits in Charcot–Marie–Tooth type 2E

Genes, brain and behavior, 2017-02, Vol.16 (2), p.260-270

2017

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Differential effects of myostatin deficiency on motor and sensory axons

Muscle & nerve, 2017-12, Vol.56 (6), p.E100-E107

2017

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A combinatorial approach increases SMN level in SMA model mice

Human molecular genetics, 2022-08, Vol.31 (17), p.2989-3000

2022

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Altered white matter microstructure in 22q11.2 deletion syndrome: a multisite diffusion tensor imaging study

Molecular psychiatry, 2020-11, Vol.25 (11), p.2818-2831

2020

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Muscle fiber-type selective propensity to pathology in the nmd mouse model of SMARD1

Biochemical and biophysical research communications, 2019-08, Vol.516 (1), p.313-319

2019

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Selective vulnerability in neuronal populations in nmd/SMARD1 mice

Human molecular genetics, 2018-02, Vol.27 (4), p.679-690

2018

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Motor unit recovery following Smn restoration in mouse models of spinal muscular atrophy

Human molecular genetics, 2022-09, Vol.31 (18), p.3107-3119

2022

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Normal muscle fiber type distribution is recapitulated in aged ephrin-A3 -/- mice that previously lacked most slow myofibers

American Journal of Physiology: Cell Physiology, 2023-03, Vol.324 (3), p.C718-C727

2023

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The Ighmbp2 D564N mouse model is the first SMARD1 model to demonstrate respiratory defects

Human molecular genetics, 2022-04, Vol.31 (8), p.1293-1307

2022

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The Ighmbp2D564N mouse model is the first SMARD1 model to demonstrate respiratory defects

Human molecular genetics, 2022-04, Vol.31 (8), p.1293-1307

2022

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Development of a novel severe mouse model of spinal muscular atrophy with respiratory distress type 1: FVB-nmd

Biochemical and biophysical research communications, 2019-12, Vol.520 (2), p.341-346

2019

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AAV9-DOK7 gene therapy reduces disease severity in Smn2B/- SMA model mice

Biochemical and biophysical research communications, 2020-09, Vol.530 (1), p.107-114

2020

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Defining the optimal dose and therapeutic window in SMA with respiratory distress type I model mice, FVB/NJ- Ighmpb2 nmd-2J

Molecular therapy. Methods & clinical development, 2021-12, Vol.23, p.23-32

2021

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A Direct Comparison of IV and ICV Delivery Methods for Gene Replacement Therapy in a Mouse Model of SMARD1

Molecular therapy. Methods & clinical development, 2018-09, Vol.10, p.348-360

2018

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Internode length is reduced during myelination and remyelination by neurofilament medium phosphorylation in motor axons

Experimental neurology, 2018-08, Vol.306, p.158-168

2018

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Genetic Manipulation of Neurofilament Protein Phosphorylation

Methods in enzymology, 2016, Vol.568, p.461

2016

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Exacerbation of Charcot–Marie–Tooth type 2E neuropathy following traumatic nerve injury

Brain research, 2015-11, Vol.1627, p.143-153

2015

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Functional characterization of SMN evolution in mouse models of SMA

Scientific reports, 2019-07, Vol.9 (1), p.9472-12, Article 9472

2019

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Defining the optimal dose and therapeutic window in SMA with respiratory distress type I model mice, FVB/NJ-Ighmpb2

Molecular therapy. Methods & clinical development, 2021-12, Vol.23, p.23-32

2021

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Astrocyte-produced miR-146a as a mediator of motor neuron loss in spinal muscular atrophy

Human molecular genetics, 2017-09, Vol.26 (17), p.3409-3420

2017

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Plastin-3 extends survival and reduces severity in mouse models of spinal muscular atrophy

JCI insight, 2017-03, Vol.2 (5), p.e89970-e89970

2017

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Arginase-1 expressing microglia in close proximity to motor neurons were increased early in disease progression in canine degenerative myelopathy, a model of amyotrophic lateral sclerosis

AAV9-mediated delivery of miR-23a reduces disease severity in Smn2B/−SMA model mice

Survival motor neuron deficiency slows myoblast fusion through reduced myomaker and myomixer expression

AAV9-DOK7 gene therapy reduces disease severity in Smn SMA model mice

Muscle spindle alterations precede onset of sensorimotor deficits in Charcot–Marie–Tooth type 2E

Differential effects of myostatin deficiency on motor and sensory axons

A combinatorial approach increases SMN level in SMA model mice

Altered white matter microstructure in 22q11.2 deletion syndrome: a multisite diffusion tensor imaging study

Muscle fiber-type selective propensity to pathology in the nmd mouse model of SMARD1

Selective vulnerability in neuronal populations in nmd/SMARD1 mice

Motor unit recovery following Smn restoration in mouse models of spinal muscular atrophy

Normal muscle fiber type distribution is recapitulated in aged ephrin-A3 -/- mice that previously lacked most slow myofibers

The Ighmbp2 D564N mouse model is the first SMARD1 model to demonstrate respiratory defects

The Ighmbp2D564N mouse model is the first SMARD1 model to demonstrate respiratory defects

Development of a novel severe mouse model of spinal muscular atrophy with respiratory distress type 1: FVB-nmd

AAV9-DOK7 gene therapy reduces disease severity in Smn2B/- SMA model mice

Defining the optimal dose and therapeutic window in SMA with respiratory distress type I model mice, FVB/NJ- Ighmpb2 nmd-2J

A Direct Comparison of IV and ICV Delivery Methods for Gene Replacement Therapy in a Mouse Model of SMARD1

Internode length is reduced during myelination and remyelination by neurofilament medium phosphorylation in motor axons

Genetic Manipulation of Neurofilament Protein Phosphorylation

Exacerbation of Charcot–Marie–Tooth type 2E neuropathy following traumatic nerve injury

Functional characterization of SMN evolution in mouse models of SMA

Defining the optimal dose and therapeutic window in SMA with respiratory distress type I model mice, FVB/NJ-Ighmpb2

Astrocyte-produced miR-146a as a mediator of motor neuron loss in spinal muscular atrophy

Plastin-3 extends survival and reduces severity in mouse models of spinal muscular atrophy

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