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Hepatocellular carcinoma: old friends and new tricks

Experimental and Molecular Medicine, 2020, 52(0), , pp.1-10

2020

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Promotion of growth factor signaling as a critical function of β-catenin during HCC progression

Nature communications, 2019-04, Vol.10 (1), p.1909-1909, Article 1909

2019

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Identification of monocyte-like precursors of granulocytes in cancer as a mechanism for accumulation of PMN-MDSCs

The Journal of experimental medicine, 2019-09, Vol.216 (9), p.2150-2169

2019

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Newly identified aspects of tumor suppression by RB

Disease models & mechanisms, 2011-09, Vol.4 (5), p.581-585

2011

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Direct Comparison of Mononucleated and Binucleated Cardiomyocytes Reveals Molecular Mechanisms Underlying Distinct Proliferative Competencies

Cell reports (Cambridge), 2020-03, Vol.30 (9), p.3105-3116.e4

2020

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Notch signaling inhibits hepatocellular carcinoma following inactivation of the RB pathway

The Journal of experimental medicine, 2011-09, Vol.208 (10), p.1963-1976

2011

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Recruitment of Pontin/Reptin by E2f1 amplifies E2f transcriptional response during cancer progression

Nature communications, 2015-12, Vol.6 (1), p.10028-10028, Article 10028

2015

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Abstract B06: A SOX9+ bile duct progenitor as a cell of origin of hepatocellular carcinoma

Cancer research (Chicago, Ill.), 2018-05, Vol.78 (10_Supplement), p.B06-B06

Viatour, Patrick

2018

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Hematopoietic stem cell quiescence is maintained by compound contributions of the retinoblastoma gene family

Cell stem cell, 2008-10, Vol.3 (4), p.416-428

2008

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Rb family proteins enforce the homeostasis of quiescent hematopoietic stem cells by repressing Socs3 expression

The Journal of experimental medicine, 2017-07, Vol.214 (7), p.1901-1912

2017

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Inactivation of the RB family prevents thymus involution and promotes thymic function by direct control of Foxn1 expression

The Journal of experimental medicine, 2013-06, Vol.210 (6), p.1087-1097

2013

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RB family proteins ensure the homeostasis of quiescent hematopoietic stem cells by preserving intracellular cytokine signaling

Experimental hematology, 2016-09, Vol.44 (9), p.S106-S106

Viatour, Patrick

2016

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MicroRNA programs in normal and aberrant stem and progenitor cells

Genome research, 2011-05, Vol.21 (5), p.798

2011

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Regulation of RB Transcription In Vivo by RB Family Members

Molecular and Cellular Biology, 2010-04, Vol.30 (7), p.1729-1745

2010

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GSK3-Mediated BCL-3 Phosphorylation Modulates Its Degradation and Its Oncogenicity

Molecular cell, 2004-10, Vol.16 (1), p.35-45

2004

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The Repressing Function of the Oncoprotein BCL-3 Requires CtBP, while Its Polyubiquitination and Degradation Involve the E3 Ligase TBLR1

Molecular and Cellular Biology, 2010-08, Vol.30 (16), p.4006-4021

2010

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Phosphorylation of NF-κB and IκB proteins: implications in cancer and inflammation

Trends in biochemical sciences (Amsterdam. Regular ed.), 2005, Vol.30 (1), p.43-52

2005

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Inhibition of the NF-kappa B transcription factor increases Bax expression in cancer cell lines

Oncogene, 2001-05, Vol.20 (22), p.2805-2813

2001

Volltextzugriff (PDF)

Bridges between Cell Cycle Regulation and Self-Renewal Maintenance

Genes & cancer, 2012-11, Vol.3 (11-12), p.670-677

Viatour, Patrick

2012

Volltextzugriff (PDF)

GFP reporter mice for the retinoblastoma-related cell cycle regulator p107

Cell cycle (Georgetown, Tex.), 2008-08, Vol.7 (16), p.2544-2552

2008

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Protein phosphorylation as a key mechanism for the regulation of BCL-3 activity

Cell cycle (Georgetown, Tex.), 2004-12, Vol.3 (12), p.1498-1501

2004

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In vivo disruption of an Rb-E2F-Ezh2 signaling loop causes bladder cancer

Cancer research (Chicago, Ill.), 2014-11, Vol.74 (22), p.6565-6577

2014

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Abstract PR07: Recruitment of Pontin/Reptin by E2F1 amplifies E2F transcriptional response during cancer progression

Molecular cancer research, 2016-11, Vol.14 (11_Supplement), p.PR07-PR07

2016

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Deregulated NF-κB activity in haematological malignancies

Biochemical pharmacology, 2006-10, Vol.72 (9), p.1069-1080

2006

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NF-κB2/p100 induces Bcl-2 expression

Leukemia, 2003-07, Vol.17 (7), p.1349-1356

2003

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Hepatocellular carcinoma: old friends and new tricks

Promotion of growth factor signaling as a critical function of β-catenin during HCC progression

Identification of monocyte-like precursors of granulocytes in cancer as a mechanism for accumulation of PMN-MDSCs

Newly identified aspects of tumor suppression by RB

Direct Comparison of Mononucleated and Binucleated Cardiomyocytes Reveals Molecular Mechanisms Underlying Distinct Proliferative Competencies

Notch signaling inhibits hepatocellular carcinoma following inactivation of the RB pathway

Recruitment of Pontin/Reptin by E2f1 amplifies E2f transcriptional response during cancer progression

Abstract B06: A SOX9+ bile duct progenitor as a cell of origin of hepatocellular carcinoma

Hematopoietic stem cell quiescence is maintained by compound contributions of the retinoblastoma gene family

Rb family proteins enforce the homeostasis of quiescent hematopoietic stem cells by repressing Socs3 expression

Inactivation of the RB family prevents thymus involution and promotes thymic function by direct control of Foxn1 expression

RB family proteins ensure the homeostasis of quiescent hematopoietic stem cells by preserving intracellular cytokine signaling

MicroRNA programs in normal and aberrant stem and progenitor cells

Regulation of RB Transcription In Vivo by RB Family Members

GSK3-Mediated BCL-3 Phosphorylation Modulates Its Degradation and Its Oncogenicity

The Repressing Function of the Oncoprotein BCL-3 Requires CtBP, while Its Polyubiquitination and Degradation Involve the E3 Ligase TBLR1

Phosphorylation of NF-κB and IκB proteins: implications in cancer and inflammation

Inhibition of the NF-kappa B transcription factor increases Bax expression in cancer cell lines

Bridges between Cell Cycle Regulation and Self-Renewal Maintenance

GFP reporter mice for the retinoblastoma-related cell cycle regulator p107

Protein phosphorylation as a key mechanism for the regulation of BCL-3 activity

In vivo disruption of an Rb-E2F-Ezh2 signaling loop causes bladder cancer

Abstract PR07: Recruitment of Pontin/Reptin by E2F1 amplifies E2F transcriptional response during cancer progression

Deregulated NF-κB activity in haematological malignancies

NF-κB2/p100 induces Bcl-2 expression

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