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ERK-MAPK signaling coordinately regulates activity of Rac1 and RhoA for tumor cell motility

Cancer cell, 2003-07, Vol.4 (1), p.67-79

2003

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Smurf1 regulates tumor cell plasticity and motility through degradation of RhoA leading to localized inhibition of contractility

The Journal of cell biology, 2007-01, Vol.176 (1), p.35-42

2007

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Elevated ERK-MAP kinase activity protects the FOS family member FRA-1 against proteasomal degradation in colon carcinoma cells

Journal of cell science, 2003-12, Vol.116 (Pt 24), p.4957-4963

2003

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Transcription factor ATF3 partially transforms chick embryo fibroblasts by promoting growth factor-independent proliferation

Oncogene, 2001-03, Vol.20 (9), p.1135-1141

2001

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v-Jun downregulates the SPARC target gene by binding to the proximal promoter indirectly through Sp1 3

Oncogene, 2003-06, Vol.22 (26), p.4047-4061

2003

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Regulation of Tumor Cell Motility by ERK Mitogen-Activated Protein Kinases

Annals of the New York Academy of Sciences, 2004-12, Vol.1030 (1), p.208-218

2004

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Polytherapy with a combination of three repurposed drugs (PXT3003) down-regulates Pmp22 over-expression and improves myelination, axonal and functional parameters in models of CMT1A neuropathy

Orphanet journal of rare diseases, 2014-12, Vol.9 (1), p.201, Article 201

2014

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Combining two repurposed drugs as a promising approach for Alzheimer's disease therapy

Scientific reports, 2015-01, Vol.5 (1), p.7608-7608, Article 7608

2015

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Brimonidine displays anti‐inflammatory properties in the skin through the modulation of the vascular barrier function

Experimental dermatology, 2018-12, Vol.27 (12), p.1378-1387

2018

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Sulfoximines as potent RORγ inverse agonists

Bioorganic & medicinal chemistry letters, 2018-05, Vol.28 (8), p.1269-1273

2018

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Protective Effect of Dermal Brimonidine Applications Against UV Radiation-induced Skin Tumors, Epidermal Hyperplasia and Cell Proliferation in the Skin of Hairless Mice

Photochemistry and photobiology, 2015-11, Vol.91 (6), p.1479-1487

2015

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Impact of Minor Structural Modifications on Properties of a Series of mTOR Inhibitors

ACS medicinal chemistry letters, 2019-11, Vol.10 (11), p.1561-1567

2019

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Discovery and Characterization of CD12681, a Potent RORγ Inverse Agonist, Preclinical Candidate for the Topical Treatment of Psoriasis

ChemMedChem, 2018-02, Vol.13 (4), p.321-337

2018

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Major contribution of MEK1 to the activation of ERK1/ERK2 and to the growth of LS174T colon carcinoma cells

Biochemical and biophysical research communications, 2008-08, Vol.372 (4), p.845-849

2008

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Down-regulation of the extracellular matrix protein SPARC in vSrc- and vJun-transformed chick embryo fibroblasts contributes to tumor formation in vivo

Oncogene, 2000-03, Vol.19 (14), p.1772-1782

2000

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Down-regulation of the extracellular matrix protein SPARC in vSrc- and vJun-transformed chick embryo fibroblasts contributes to tumor formation in vivo

Oncogene, 2000 (19), p.1772-1782

2000

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Transcriptional control of SPARC by v-Jun and other members of the AP1 family of transcription factors

Oncogene, 2000-10, Vol.19 (43), p.5020-5029

2000

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Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation

Genes & development, 1998-04, Vol.12 (8), p.1227-1239

1998

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Transcriptional control of SPARC by v-Jun and other members of the AP1 family of transcription factors

Oncogene, 2000 (19), p.5020-5029

2000

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v-Jun downregulates the SPARC target gene by binding to the proximal promoter indirectly through Sp1/3

Oncogene, 2003 (22), p.4047-4061

2003

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Transcription factor ATF3 partially transforms chick embryo fibroblasts by promoting growth factor-independent proliferation

Oncogene, 2001 (20), p.1135-1141

2001

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Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Molecular and Cellular Biology, 1996-05, Vol.16 (5), p.1881-1888

1996

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The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site

Oncogene, 2001-11, Vol.20 (50), p.7425

2001

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ERK-MAPK signaling coordinately regulates activity of Rac1 and RhoA for tumor cell motility

Smurf1 regulates tumor cell plasticity and motility through degradation of RhoA leading to localized inhibition of contractility

Elevated ERK-MAP kinase activity protects the FOS family member FRA-1 against proteasomal degradation in colon carcinoma cells

Transcription factor ATF3 partially transforms chick embryo fibroblasts by promoting growth factor-independent proliferation

A Subset of Type I Conventional Dendritic Cells Controls Cutaneous Bacterial Infections through VEGFα-Mediated Recruitment of Neutrophils

v-Jun downregulates the SPARC target gene by binding to the proximal promoter indirectly through Sp1 3

Regulation of Tumor Cell Motility by ERK Mitogen-Activated Protein Kinases

Polytherapy with a combination of three repurposed drugs (PXT3003) down-regulates Pmp22 over-expression and improves myelination, axonal and functional parameters in models of CMT1A neuropathy

Combining two repurposed drugs as a promising approach for Alzheimer's disease therapy

Brimonidine displays anti‐inflammatory properties in the skin through the modulation of the vascular barrier function

Discovery and process development of a novel TACE inhibitor for the topical treatment of psoriasis

Sulfoximines as potent RORγ inverse agonists

Protective Effect of Dermal Brimonidine Applications Against UV Radiation-induced Skin Tumors, Epidermal Hyperplasia and Cell Proliferation in the Skin of Hairless Mice

Impact of Minor Structural Modifications on Properties of a Series of mTOR Inhibitors

Discovery and Characterization of CD12681, a Potent RORγ Inverse Agonist, Preclinical Candidate for the Topical Treatment of Psoriasis

Major contribution of MEK1 to the activation of ERK1/ERK2 and to the growth of LS174T colon carcinoma cells

Down-regulation of the extracellular matrix protein SPARC in vSrc- and vJun-transformed chick embryo fibroblasts contributes to tumor formation in vivo

Down-regulation of the extracellular matrix protein SPARC in vSrc- and vJun-transformed chick embryo fibroblasts contributes to tumor formation in vivo

Transcriptional control of SPARC by v-Jun and other members of the AP1 family of transcription factors

Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation

Transcriptional control of SPARC by v-Jun and other members of the AP1 family of transcription factors

v-Jun downregulates the SPARC target gene by binding to the proximal promoter indirectly through Sp1/3

Transcription factor ATF3 partially transforms chick embryo fibroblasts by promoting growth factor-independent proliferation

Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site

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