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Philip D. Lawley (1927-2011)

Carcinogenesis (New York), 2012-04, Vol.33 (4), p.947-947

2012

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Mutations in the p53 gene in schistosomal bladder cancer: a study of 92 tumours from Egyptian patients and a comparison between mutational spectra from schistosomal and non-schistosomal urothelial tumours

Carcinogenesis (New York), 1995-05, Vol.16 (5), p.1181-1189

1995

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Randomized, placebo-controlled trial of gastric acid-lowering therapy on duodenal polyposis and relative adduct labeling in familial adenomatous polyposis

Diseases of the colon & rectum, 2001-11, Vol.44 (11), p.1585-1589

2001

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The dubious evidence for smokeless tobacco

British Journal of Addiction, 1990-09, Vol.85 (9), p.1100-1102

Venitt, S.

1990

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Philip D. Lawley (1927-2011)

Mutagenesis, 2012-05, Vol.27 (3), p.255-256

2012

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Lack of telomerase in desmoids occurring sporadically and in association with familial adenomatous polyposis

British journal of surgery, 1998-07, Vol.85 (7), p.965-969

1998

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Use of mutagenicity assays in studies of human cancer

Anticancer research, 1987-09, Vol.7 (5B), p.949

Venitt, S

1987

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Agents of change

Nature (London), 1985-05, Vol.315 (6018), p.434-434

Venitt, S

1985

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Carcinogenicity and mutagenicity of chromium compounds: the association between bronchial metaplasia and neoplasia

Carcinogenesis (New York), 1986-05, Vol.7 (5), p.831-835

1986

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High pH reduces DNA damage caused by bile from patients with familial adenomatous polyposis: antacids may attenuate duodenal polyposis

Gut, 1995-06, Vol.36 (6), p.918-921

1995

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Nucleic acids change

Nature (London), 1984-08, Vol.310 (5977), p.520-520

Venitt, S

1984

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Bile acids do not form adducts when incubated with DNA in vitro

Carcinogenesis (New York), 1994-12, Vol.15 (12), p.2945-2948

1994

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Mechanisms of Spontaneous Human Cancers

Environmental health perspectives, 1996-05, Vol.104 (Suppl 3), p.633-637

Venitt, Stanley

1996

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BACTERIAL MUTATION AS AN INDICATOR OF CARCINOGENICITY

British medical bulletin, 1980-01, Vol.36 (1), p.57-62

Venitt, S

1980

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Mechanisms of carcinogenesis and individual susceptibility to cancer

Clinical chemistry (Baltimore, Md.), 1994-07, Vol.40 (7 Pt 2), p.1421-1425

Venitt, S

1994

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Mutagenicity-New Horizons in Genetic Toxicology

British Journal of Cancer, 1983-05, Vol.47 (5), p.735-736

Venitt, S

1983

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Mechanisms of Spontaneous Human Cancers

Environmental health perspectives, 1996-05, Vol.104, p.633

Venitt, Stanley

1996

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Appearance of artefacts when using 32P-postlabelling to investigate DNA adduct formation by bile acids in vitro: lack of evidence for covalent binding

Carcinogenesis (New York), 1995-07, Vol.16 (7), p.1489-1491

1995

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Detection of telomerase activity in human prostate: a diagnostic marker for prostatic cancer?

British Journal of Urology, 1997-08, Vol.80 (2), p.263-268

1997

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Differences in the levels and pattern of DNA-adduct labelling in human cell lines MCL-5 and CCRF, proficient or deficient in carcinogen-metabolism, treated in vitro with bile from familial adenomatous polyposis patients and from unaffected controls

Carcinogenesis (New York), 1996-04, Vol.17 (4), p.707-713

1996

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Mutagenicity of bile and duodenal adenomas in familial adenomatous polyposis

British journal of surgery, 1990-08, Vol.77 (8), p.878-881

1990

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Rat, but not human, sulfotransferase activates a tamoxifen metabolite to produce DNA adducts and gene mutations in bacteria and mammalian cells in culture

Carcinogenesis (New York), 1998-10, Vol.19 (10), p.1709-1713

1998

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The use of bacterial mutagenicity tests in carcinogenicity studies

British journal of cancer, 1980-03, Vol.41 (3), p.502-503

Venitt, S

1980

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IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Man. Vol. 15: Some Aromatic Amines and Related Nitro Compounds-Hair Dyes, Colouring Agents and Miscellaneous Industrial Chemicals

British Journal of Cancer, 1979-02, Vol.39 (2), p.209-210

Venitt, S

1979

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Heterocyclic aromatic amines induce DNA strand breaks and cell transformation

Carcinogenesis (New York), 1999-04, Vol.20 (4), p.545-551

1999

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Philip D. Lawley (1927-2011)

Mutations in the p53 gene in schistosomal bladder cancer: a study of 92 tumours from Egyptian patients and a comparison between mutational spectra from schistosomal and non-schistosomal urothelial tumours

Randomized, placebo-controlled trial of gastric acid-lowering therapy on duodenal polyposis and relative adduct labeling in familial adenomatous polyposis

The dubious evidence for smokeless tobacco

Philip D. Lawley (1927-2011)

Lack of telomerase in desmoids occurring sporadically and in association with familial adenomatous polyposis

Use of mutagenicity assays in studies of human cancer

Agents of change

Carcinogenicity and mutagenicity of chromium compounds: the association between bronchial metaplasia and neoplasia

High pH reduces DNA damage caused by bile from patients with familial adenomatous polyposis: antacids may attenuate duodenal polyposis

Nucleic acids change

Bile acids do not form adducts when incubated with DNA in vitro

Mechanisms of Spontaneous Human Cancers

BACTERIAL MUTATION AS AN INDICATOR OF CARCINOGENICITY

Mechanisms of carcinogenesis and individual susceptibility to cancer

Mutagenicity-New Horizons in Genetic Toxicology

Mechanisms of Spontaneous Human Cancers

Appearance of artefacts when using 32P-postlabelling to investigate DNA adduct formation by bile acids in vitro: lack of evidence for covalent binding

Detection of telomerase activity in human prostate: a diagnostic marker for prostatic cancer?

Differences in the levels and pattern of DNA-adduct labelling in human cell lines MCL-5 and CCRF, proficient or deficient in carcinogen-metabolism, treated in vitro with bile from familial adenomatous polyposis patients and from unaffected controls

Mutagenicity of bile and duodenal adenomas in familial adenomatous polyposis

Rat, but not human, sulfotransferase activates a tamoxifen metabolite to produce DNA adducts and gene mutations in bacteria and mammalian cells in culture

The use of bacterial mutagenicity tests in carcinogenicity studies

IARC Monographs on the Evaluation of the Carcinogenic Risk of Chemicals to Man. Vol. 15: Some Aromatic Amines and Related Nitro Compounds-Hair Dyes, Colouring Agents and Miscellaneous Industrial Chemicals

Heterocyclic aromatic amines induce DNA strand breaks and cell transformation

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