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KRAS Oncogenic Signaling Extends beyond Cancer Cells to Orchestrate the Microenvironment

Cancer research (Chicago, Ill.), 2018-01, Vol.78 (1), p.7-14

2018

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Stimuli-responsive hydrogels for intratumoral drug delivery

Drug discovery today, 2021-10, Vol.26 (10), p.2397-2405

2021

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Causes and consequences of microsatellite instability in gastric carcinogenesis

World journal of gastroenterology : WJG, 2014-11, Vol.20 (44), p.16433-16442

2014

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Lipid Nanoparticles Functionalized with Antibodies for Anticancer Drug Therapy

Pharmaceutics, 2023-01, Vol.15 (1), p.216

2023

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Injectable Poloxamer Hydrogels for Local Cancer Therapy

Gels, 2023-07, Vol.9 (7), p.593

2023

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The immunosuppressive and pro-tumor functions of CCL18 at the tumor microenvironment

Cytokine & growth factor reviews, 2021-08, Vol.60, p.107-119

2021

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Rheological and Injectability Evaluation of Sterilized Poloxamer-407-Based Hydrogels Containing Docetaxel-Loaded Lipid Nanoparticles

Gels, 2024-05, Vol.10 (5), p.307

2024

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Mutant KRAS modulates colorectal cancer cells invasive response to fibroblast-secreted factors through the HGF/C-MET axis

International journal of cancer, 2022-11, Vol.151 (10), p.1810-1823

2022

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The mechanism of sitagliptin inhibition of colorectal cancer cell lines' metastatic functionalities

IUBMB life, 2021-05, Vol.73 (5), p.761-773

2021

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Analytical Techniques for Characterizing Tumor-Targeted Antibody-Functionalized Nanoparticles

Life (Basel, Switzerland), 2024-04, Vol.14 (4), p.489

2024

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Modulation of Fibroblast Phenotype by Colorectal Cancer Cell-Secreted Factors Is Mostly Independent of Oncogenic KRAS

Cells (Basel, Switzerland), 2022-08, Vol.11 (16), p.2490

2022

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BRAF, KRAS and PIK3CA mutations in colorectal serrated polyps and cancer: primary or secondary genetic events in colorectal carcinogenesis?

BMC cancer, 2008-09, Vol.8 (1), p.255-255, Article 255

2008

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Intricate Macrophage-Colorectal Cancer Cell Communication in Response to Radiation

PloS one, 2016-08, Vol.11 (8), p.e0160891-e0160891

2016

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Mutant KRAS-Associated Proteome Is Mainly Controlled by Exogenous Factors

Cells (Basel, Switzerland), 2022-06, Vol.11 (13), p.1988

2022

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Mutant N-RAS protects colorectal cancer cells from stress-induced apoptosis and contributes to cancer development and progression

Cancer discovery, 2013-03, Vol.3 (3), p.294-307

2013

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S100P is a molecular determinant of E-cadherin function in gastric cancer

Cell communication and signaling, 2019-11, Vol.17 (1), p.155-155, Article 155

2019

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Targeting the Tumor Microenvironment: An Unexplored Strategy for Mutant KRAS Tumors

Cancers, 2019-12, Vol.11 (12), p.2010

2019

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Oncogenic mutations in gastric cancer with microsatellite instability

European journal of cancer (1990), 2011-02, Vol.47 (3), p.443-451

2011

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The prevalence of PIK3CA mutations in gastric and colon cancer

European journal of cancer (1990), 2005-07, Vol.41 (11), p.1649-1654

2005

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Fibroblasts Promote Resistance to KRAS Silencing in Colorectal Cancer Cells

Cancers, 2024-07, Vol.16 (14), p.2595

2024

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Decellularized Colorectal Cancer Matrices as Bioactive Scaffolds for Studying Tumor-Stroma Interactions

Cancers, 2022-01, Vol.14 (2), p.359

2022

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Specific inhibition of p110α subunit of PI3K: putative therapeutic strategy for KRAS mutant colorectal cancers

Oncotarget, 2016-10, Vol.7 (42), p.68546-68558

2016

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B-Raf(V600E) cooperates with alternative spliced Rac1b to sustain colorectal cancer cell survival

Gastroenterology (New York, N.Y. 1943), 2008-09, Vol.135 (3), p.899-906

2008

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KRAS mutations and anti-epidermal growth factor receptor therapy in colorectal cancer with lymph node metastases

Journal of clinical oncology, 2009-01, Vol.27 (1), p.158-159

2009

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Dissecting the signaling pathways associated with the oncogenic activity of MLK3 P252H mutation

BMC cancer, 2014-03, Vol.14 (1), p.182-182, Article 182

2014

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KRAS Oncogenic Signaling Extends beyond Cancer Cells to Orchestrate the Microenvironment

Stimuli-responsive hydrogels for intratumoral drug delivery

Causes and consequences of microsatellite instability in gastric carcinogenesis

Lipid Nanoparticles Functionalized with Antibodies for Anticancer Drug Therapy

Injectable Poloxamer Hydrogels for Local Cancer Therapy

The immunosuppressive and pro-tumor functions of CCL18 at the tumor microenvironment

Rheological and Injectability Evaluation of Sterilized Poloxamer-407-Based Hydrogels Containing Docetaxel-Loaded Lipid Nanoparticles

Mutant KRAS modulates colorectal cancer cells invasive response to fibroblast-secreted factors through the HGF/C-MET axis

The mechanism of sitagliptin inhibition of colorectal cancer cell lines' metastatic functionalities

Analytical Techniques for Characterizing Tumor-Targeted Antibody-Functionalized Nanoparticles

Modulation of Fibroblast Phenotype by Colorectal Cancer Cell-Secreted Factors Is Mostly Independent of Oncogenic KRAS

BRAF, KRAS and PIK3CA mutations in colorectal serrated polyps and cancer: primary or secondary genetic events in colorectal carcinogenesis?

Intricate Macrophage-Colorectal Cancer Cell Communication in Response to Radiation

Mutant KRAS-Associated Proteome Is Mainly Controlled by Exogenous Factors

Mutant N-RAS protects colorectal cancer cells from stress-induced apoptosis and contributes to cancer development and progression

S100P is a molecular determinant of E-cadherin function in gastric cancer

Targeting the Tumor Microenvironment: An Unexplored Strategy for Mutant KRAS Tumors

Oncogenic mutations in gastric cancer with microsatellite instability

The prevalence of PIK3CA mutations in gastric and colon cancer

Fibroblasts Promote Resistance to KRAS Silencing in Colorectal Cancer Cells

Decellularized Colorectal Cancer Matrices as Bioactive Scaffolds for Studying Tumor-Stroma Interactions

Specific inhibition of p110α subunit of PI3K: putative therapeutic strategy for KRAS mutant colorectal cancers

B-Raf(V600E) cooperates with alternative spliced Rac1b to sustain colorectal cancer cell survival

KRAS mutations and anti-epidermal growth factor receptor therapy in colorectal cancer with lymph node metastases

Dissecting the signaling pathways associated with the oncogenic activity of MLK3 P252H mutation

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