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The Physiological and Pathological Implications of the Formation of Hydrogels, with a Specific Focus on Amyloid Polypeptides

Biomolecules (Basel, Switzerland), 2017-09, Vol.7 (4), p.70

2017

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Amyloid Fibril Formation by a Synthetic Peptide from a Region of Human Acetylcholinesterase that Is Homologous to the Alzheimer's Amyloid-β Peptide

Biochemistry (Easton), 2002-11, Vol.41 (46), p.13539-13547

2002

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Rapid method for measurement of surface tension in multiwell plates

Laboratory investigation, 2004-04, Vol.84 (4), p.523-529

2004

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The Intact Human Acetylcholinesterase C-Terminal Oligomerization Domain Is α-Helical in Situ and in Isolation, but a Shorter Fragment Forms β-Sheet-Rich Amyloid Fibrils and Protofibrillar Oligomers

Biochemistry (Easton), 2003-09, Vol.42 (36), p.10863-10873

2003

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Spike-nucleocapsid interaction in Semliki Forest virus reconstructed using network antibodies

Nature (London), 1988-11, Vol.336 (6194), p.36-42

1988

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Vaccinia Virus Utilizes Microtubules for Movement to the Cell Surface

The Journal of cell biology, 2001-07, Vol.154 (2), p.389-402

2001

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Monoclonal antibody defines a macrophage intracellular Ca 2+ -binding protein which is phosphorylated by phagocytosis

Nature (London), 1982-09, Vol.299 (5878), p.70-72

1982

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Intracellular antigens associated with cytoplasmic surface of phagolysosomes

Journal of cell science, 1985-08, Vol.77 (1), p.109-127

1985

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Monoclonal antibody defines a macrophage intracellular Ca2+-binding protein which is phosphorylated by phagocytosis

Nature (London), 1982-09, Vol.299 (5878), p.70-72

1982

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Reproducible Research Practices and Transparency across the Biomedical Literature

PLoS biology, 2016-01, Vol.14 (1), p.e1002333-e1002333

2016

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A critical evaluation of the algorithm behind the Relative Citation Ratio (RCR)

PLoS biology, 2017-10, Vol.15 (10), p.e2002536-e2002536

2017

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The nucleoplasmic reticulum: form and function

Trends in cell biology, 2011-06, Vol.21 (6), p.362-373

2011

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Liquid–liquid phase separation of type II diabetes-associated IAPP initiates hydrogelation and aggregation

Proceedings of the National Academy of Sciences - PNAS, 2020-06, Vol.117 (22), p.12050-12061

2020

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Enrichment of Amyloidogenesis at an Air-Water Interface

Biophysical journal, 2012-03, Vol.102 (5), p.1154-1162

2012

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IAPs, RINGs and ubiquitylation

Nature reviews. Molecular cell biology, 2005-04, Vol.6 (4), p.287-297

2005

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In vivo localization of human acetylcholinesterase-derived species in a β-sheet conformation at the core of senile plaques in Alzheimer's disease

The Journal of biological chemistry, 2019-04, Vol.294 (16), p.6253-6272

2019

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Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming

EMBO reports, 2014-09, Vol.15 (9), p.982-990

2014

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cIAPs and XIAP regulate myelopoiesis through cytokine production in an RIPK1- and RIPK3-dependent manner

Blood, 2014-04, Vol.123 (16), p.2562-2572

2014

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XIAP Loss Triggers RIPK3- and Caspase-8-Driven IL-1β Activation and Cell Death as a Consequence of TLR-MyD88-Induced cIAP1-TRAF2 Degradation

Cell reports (Cambridge), 2017-07, Vol.20 (3), p.668-682

2017

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The enigma of phosphoinositides and their derivatives: Their role in regulation of subcellular compartment morphology

Biochimica et biophysica acta. Biomembranes, 2022-02, Vol.1864 (1), p.183780-183780, Article 183780

2022

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Cell Death in Development

Cell, 1999-01, Vol.96 (2), p.245-254

1999

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The kinetics of islet amyloid polypeptide phase-separated system and hydrogel formation are critically influenced by macromolecular crowding

Biochemical journal, 2021-08, Vol.478 (15), p.3025-3046

2021

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The Physiological and Pathological Implications of the Formation of Hydrogels, with a Specific Focus on Amyloid Polypeptides

Amyloid Fibril Formation by a Synthetic Peptide from a Region of Human Acetylcholinesterase that Is Homologous to the Alzheimer's Amyloid-β Peptide

Rapid method for measurement of surface tension in multiwell plates

The Intact Human Acetylcholinesterase C-Terminal Oligomerization Domain Is α-Helical in Situ and in Isolation, but a Shorter Fragment Forms β-Sheet-Rich Amyloid Fibrils and Protofibrillar Oligomers

Spike-nucleocapsid interaction in Semliki Forest virus reconstructed using network antibodies

Vaccinia Virus Utilizes Microtubules for Movement to the Cell Surface

Monoclonal antibody defines a macrophage intracellular Ca 2+ -binding protein which is phosphorylated by phagocytosis

Intracellular antigens associated with cytoplasmic surface of phagolysosomes

Monoclonal antibody defines a macrophage intracellular Ca2+-binding protein which is phosphorylated by phagocytosis

Reproducible Research Practices and Transparency across the Biomedical Literature

A critical evaluation of the algorithm behind the Relative Citation Ratio (RCR)

Invasive non‐native species likely to threaten biodiversity and ecosystems in the Antarctic Peninsula region

The nucleoplasmic reticulum: form and function

Liquid–liquid phase separation of type II diabetes-associated IAPP initiates hydrogelation and aggregation

Enrichment of Amyloidogenesis at an Air-Water Interface

IAPs, RINGs and ubiquitylation

In vivo localization of human acetylcholinesterase-derived species in a β-sheet conformation at the core of senile plaques in Alzheimer's disease

RIPK1 Regulates RIPK3-MLKL-Driven Systemic Inflammation and Emergency Hematopoiesis

Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming

cIAPs and XIAP regulate myelopoiesis through cytokine production in an RIPK1- and RIPK3-dependent manner

XIAP Loss Triggers RIPK3- and Caspase-8-Driven IL-1β Activation and Cell Death as a Consequence of TLR-MyD88-Induced cIAP1-TRAF2 Degradation

The enigma of phosphoinositides and their derivatives: Their role in regulation of subcellular compartment morphology

Cell Death in Development

The kinetics of islet amyloid polypeptide phase-separated system and hydrogel formation are critically influenced by macromolecular crowding

TNFR1-dependent cell death drives inflammation in Sharpin-deficient mice

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