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HPV16 E1 dysregulated cellular genes involved in cell proliferation and host DNA damage: A possible role in cervical carcinogenesis

PloS one, 2021-12, Vol.16 (12), p.e0260841

2021

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BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Cell death and differentiation, 2019-06, Vol.26 (6), p.1037-1047

2019

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Novel roles of RTN4 and CLIMP-63 in regulating mitochondrial structure, bioenergetics and apoptosis

Cell death & disease, 2022-05, Vol.13 (5), p.436-436, Article 436

2022

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Role of HPV16 E1 in cervical carcinogenesis

Frontiers in cellular and infection microbiology, 2022-07, Vol.12, p.955847-955847

2022

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Unexpected inhibition of the lipid kinase PIKfyve reveals an epistatic role for p38 MAPKs in endolysosomal fission and volume control

Cell death & disease, 2024-01, Vol.15 (1), p.80-80, Article 80

2024

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Targeting intermediary metabolism enhances the efficacy of BH3 mimetic therapy in hematologic malignancies

Haematologica (Roma), 2019-05, Vol.104 (5), p.1016-1025

2019

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1108 Novel regulators of ImmTAC-mediated killing of melanoma cancer cells revealed by genome-wide CRISPR-Cas9 screens

Journal for immunotherapy of cancer, 2023-11, Vol.11 (Suppl 1), p.A1220-A1220

2023

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Exploring the potential of BH3 mimetic therapy in squamous cell carcinoma of the head and neck

Cell death & disease, 2019-12, Vol.10 (12), p.912-10, Article 912

2019

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Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis

Neoplasia (New York, N.Y.), 2013-05, Vol.15 (5), p.568-IN22

2013

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Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis

PloS one, 2013-02, Vol.8 (2), p.e56603

2013

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BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L

Cell death and differentiation, 2019-06, Vol.26 (6), p.1037

2019

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BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cells

British journal of cancer, 2016-03, Vol.114 (6), p.638-641

2016

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Abstract 4984: TCR-CD3 bispecifics kill tumor cells in a caspase- and gasdermin-dependent manner by inducing multiple cell death pathways

Cancer research (Chicago, Ill.), 2024-03, Vol.84 (6_Supplement), p.4984-4984

2024

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STINGing Viral Tumors: What We Know from Head and Neck Cancers

Cancer research (Chicago, Ill.), 2021-08, Vol.81 (15), p.3945-3952

2021

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Selective BH3-mimetics targeting BCL-2, BCL-X L or MCL-1 induce severe mitochondrial perturbations

Biological chemistry, 2019-02, Vol.400 (2), p.181-185

2019

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Whole-genome sequencing of chronic lymphocytic leukemia identifies subgroups with distinct biological and clinical features

Nature genetics, 2022-11, Vol.54 (11), p.1675-1689

2022

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Apogossypol-mediated reorganisation of the endoplasmic reticulum antagonises mitochondrial fission and apoptosis

Cell death & disease, 2019-07, Vol.10 (7), p.521-11, Article 521

2019

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Selective BH3-mimetics targeting BCL-2, BCL-XL or MCL-1 induce severe mitochondrial perturbations

Biological chemistry, 2019-01, Vol.400 (2), p.181

2019

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DRP-1 is required for BH3 mimetic-mediated mitochondrial fragmentation and apoptosis

Cell death & disease, 2017-01, Vol.8 (1), p.e2552-e2552

2017

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Maritoclax and dinaciclib inhibit MCL-1 activity and induce apoptosis in both a MCL-1-dependent and -independent manner

Oncotarget, 2015-05, Vol.6 (14), p.12668-12681

2015

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DRP-1 functions independently of mitochondrial structural perturbations to facilitate BH3 mimetic-mediated apoptosis

Cell death discovery, 2019-07, Vol.5 (1), p.117-11, Article 117

2019

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Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis12

Neoplasia (New York, N.Y.), 2013-05, Vol.15 (5), p.568-578

2013

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The small molecule dispergo tubulates the endoplasmic reticulum and inhibits export

Molecular biology of the cell, 2013-04, Vol.24 (7), p.1020-1029

2013

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Drosophila Omi, a mitochondrial-localized IAP antagonist and proapoptotic serine protease

The EMBO journal, 2007-07, Vol.26 (13), p.3144-3156

2007

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p38 MAPKs coordinately regulate distinct phases of autophagy and lysosomal biogenesis

Varadarajan, Shankar

2008

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HPV16 E1 dysregulated cellular genes involved in cell proliferation and host DNA damage: A possible role in cervical carcinogenesis

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Novel roles of RTN4 and CLIMP-63 in regulating mitochondrial structure, bioenergetics and apoptosis

Role of HPV16 E1 in cervical carcinogenesis

Unexpected inhibition of the lipid kinase PIKfyve reveals an epistatic role for p38 MAPKs in endolysosomal fission and volume control

Targeting intermediary metabolism enhances the efficacy of BH3 mimetic therapy in hematologic malignancies

1108 Novel regulators of ImmTAC-mediated killing of melanoma cancer cells revealed by genome-wide CRISPR-Cas9 screens

Exploring the potential of BH3 mimetic therapy in squamous cell carcinoma of the head and neck

Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis

Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L

BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cells

Abstract 4984: TCR-CD3 bispecifics kill tumor cells in a caspase- and gasdermin-dependent manner by inducing multiple cell death pathways

STINGing Viral Tumors: What We Know from Head and Neck Cancers

Selective BH3-mimetics targeting BCL-2, BCL-X L or MCL-1 induce severe mitochondrial perturbations

Whole-genome sequencing of chronic lymphocytic leukemia identifies subgroups with distinct biological and clinical features

Apogossypol-mediated reorganisation of the endoplasmic reticulum antagonises mitochondrial fission and apoptosis

Selective BH3-mimetics targeting BCL-2, BCL-XL or MCL-1 induce severe mitochondrial perturbations

DRP-1 is required for BH3 mimetic-mediated mitochondrial fragmentation and apoptosis

Maritoclax and dinaciclib inhibit MCL-1 activity and induce apoptosis in both a MCL-1-dependent and -independent manner

DRP-1 functions independently of mitochondrial structural perturbations to facilitate BH3 mimetic-mediated apoptosis

Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis12

The small molecule dispergo tubulates the endoplasmic reticulum and inhibits export

Drosophila Omi, a mitochondrial-localized IAP antagonist and proapoptotic serine protease

p38 MAPKs coordinately regulate distinct phases of autophagy and lysosomal biogenesis

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