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Cyclic nucleotide phosphodiesterases as therapeutic targets in cardiac hypertrophy and heart failure

Nature reviews cardiology, 2023-02, Vol.20 (2), p.90-108

2023

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Investigating cardiac β-adrenergic nuclear signaling with FRET-based biosensors

Annales d'endocrinologie, 2021-06, Vol.82 (3-4), p.198-200

2021

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Cyclic nucleotide signalling compartmentation by PDEs in cultured vascular smooth muscle cells

British journal of pharmacology, 2019-06, Vol.176 (11), p.1780-1792

2019

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Cyclic Adenosine Monophosphate Phosphodiesterase Type 4 Protects Against Atrial Arrhythmias

Journal of the American College of Cardiology, 2012-06, Vol.59 (24), p.2182-2190

2012

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Phosphodiesterase 2 Protects Against Catecholamine-Induced Arrhythmia and Preserves Contractile Function After Myocardial Infarction

Circulation research, 2017-01, Vol.120 (1), p.120-132

2017

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PDEs create local domains of cAMP signaling

Journal of molecular and cellular cardiology, 2012-02, Vol.52 (2), p.323-329

2012

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Selective changes in cytosolic β-adrenergic cAMP signals and L-type Calcium Channel regulation by Phosphodiesterases during cardiac hypertrophy

Journal of molecular and cellular cardiology, 2021-01, Vol.150, p.109-121

2021

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Calmodulin kinase II inhibition limits the pro-arrhythmic Ca2+ waves induced by cAMP-phosphodiesterase inhibitors

Cardiovascular research, 2016-05, Vol.110 (1), p.151-161

2016

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Cardiac Overexpression of PDE4B Blunts β-Adrenergic Response and Maladaptive Remodeling in Heart Failure

Circulation (New York, N.Y.), 2020-07, Vol.142 (2), p.161-174

2020

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Cyclic nucleotide phosphodiesterases: therapeutic targets in cardiac hypertrophy and failure

M.S. Médecine sciences, 2024-07, Vol.40 (6-7), p.534

2024

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Compartmentation of Cyclic Nucleotide Signaling in the Heart: The Role of Cyclic Nucleotide Phosphodiesterases

Circulation research, 2006-10, Vol.99 (8), p.816-828

2006

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Response by Vandecasteele et al to Letter Regarding Article, "Cardiac Overexpression of PDE4B Blunts β-Adrenergic Response and Maladaptive Remodeling in Heart Failure"

Circulation (New York, N.Y.), 2021-01, Vol.143 (4), p.e26-e27

2021

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Phosphodiesterase-2 Is Up-Regulated in Human Failing Hearts and Blunts β-Adrenergic Responses in Cardiomyocytes

Journal of the American College of Cardiology, 2013-10, Vol.62 (17), p.1596-1606

2013

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Recombinant Expression of the Voltage-Dependent Anion Channel Enhances the Transfer of Ca2+ Microdomains to Mitochondria

The Journal of cell biology, 2002-11, Vol.159 (4), p.613-624

2002

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Cyclic nucleotide phosphodiesterases in heart and vessels: A therapeutic perspective

Archives of cardiovascular diseases, 2016-06, Vol.109 (6-7), p.431-443

2016

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Cardiac Phosphodiesterases Are Differentially Increased in Diabetic Cardiomyopathy

Life sciences (1973), 2021-10, Vol.283, p.119857-119857, Article 119857

2021

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Decreased Expression and Activity of cAMP Phosphodiesterases in Cardiac Hypertrophy and Its Impact on β-Adrenergic cAMP Signals

Circulation research, 2009-10, Vol.105 (8), p.784-792

2009

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cAMP-Binding Protein Epac Induces Cardiomyocyte Hypertrophy

Circulation research, 2005-12, Vol.97 (12), p.1296-1304

2005

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PDE2 regulates membrane potential, respiration and permeability transition of rodent subsarcolemmal cardiac mitochondria

Mitochondrion, 2019-07, Vol.47, p.64-75

2019

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Phosphodiesterase type 4 anchoring regulates cAMP signaling to Popeye domain-containing proteins

Journal of molecular and cellular cardiology, 2022-04, Vol.165, p.86-102

2022

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Cardiac adenylyl cyclase overexpression precipitates and aggravates age-related myocardial dysfunction

Cardiovascular research, 2019-10, Vol.115 (12), p.1778-1790

2019

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Differential regulation of cardiac excitation-contraction coupling by cAMP phosphodiesterase subtypes

Cardiovascular research, 2013-11, Vol.100 (2), p.336-346

2013

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Cyclic AMP compartmentation due to increased cAMP‐phosphodiesterase activity in transgenic mice with a cardiac‐directed expression of the human adenylyl cyclase type 8 (AC8)

The FASEB journal, 2003-08, Vol.17 (11), p.1380-1391

2003

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Phosphodiesterase 11A (PDE11A) Gene Defects in Patients with ACTH-Independent Macronodular Adrenal Hyperplasia (AIMAH): Functional Variants May Contribute to Genetic Susceptibility of Bilateral Adrenal Tumors

The journal of clinical endocrinology and metabolism, 2012-11, Vol.97 (11), p.E2063-E2069

2012

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Differential changes in cyclic adenosine 3′‐5′ monophosphate (cAMP) effectors and major Ca2+ handling proteins during diabetic cardiomyopathy

Journal of cellular and molecular medicine, 2023-05, Vol.27 (9), p.1277-1289

2023

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Cyclic nucleotide phosphodiesterases as therapeutic targets in cardiac hypertrophy and heart failure

Investigating cardiac β-adrenergic nuclear signaling with FRET-based biosensors

Cyclic nucleotide signalling compartmentation by PDEs in cultured vascular smooth muscle cells

Cyclic Adenosine Monophosphate Phosphodiesterase Type 4 Protects Against Atrial Arrhythmias

Phosphodiesterase 2 Protects Against Catecholamine-Induced Arrhythmia and Preserves Contractile Function After Myocardial Infarction

PDEs create local domains of cAMP signaling

Selective changes in cytosolic β-adrenergic cAMP signals and L-type Calcium Channel regulation by Phosphodiesterases during cardiac hypertrophy

Calmodulin kinase II inhibition limits the pro-arrhythmic Ca2+ waves induced by cAMP-phosphodiesterase inhibitors

Cardiac Overexpression of PDE4B Blunts β-Adrenergic Response and Maladaptive Remodeling in Heart Failure

Cyclic nucleotide phosphodiesterases: therapeutic targets in cardiac hypertrophy and failure

Compartmentation of Cyclic Nucleotide Signaling in the Heart: The Role of Cyclic Nucleotide Phosphodiesterases

Response by Vandecasteele et al to Letter Regarding Article, "Cardiac Overexpression of PDE4B Blunts β-Adrenergic Response and Maladaptive Remodeling in Heart Failure"

Phosphodiesterase-2 Is Up-Regulated in Human Failing Hearts and Blunts β-Adrenergic Responses in Cardiomyocytes

Recombinant Expression of the Voltage-Dependent Anion Channel Enhances the Transfer of Ca2+ Microdomains to Mitochondria

Cyclic nucleotide phosphodiesterases in heart and vessels: A therapeutic perspective

Cardiac Phosphodiesterases Are Differentially Increased in Diabetic Cardiomyopathy

Decreased Expression and Activity of cAMP Phosphodiesterases in Cardiac Hypertrophy and Its Impact on β-Adrenergic cAMP Signals

cAMP-Binding Protein Epac Induces Cardiomyocyte Hypertrophy

PDE2 regulates membrane potential, respiration and permeability transition of rodent subsarcolemmal cardiac mitochondria

Phosphodiesterase type 4 anchoring regulates cAMP signaling to Popeye domain-containing proteins

Cardiac adenylyl cyclase overexpression precipitates and aggravates age-related myocardial dysfunction

Differential regulation of cardiac excitation-contraction coupling by cAMP phosphodiesterase subtypes

Cyclic AMP compartmentation due to increased cAMP‐phosphodiesterase activity in transgenic mice with a cardiac‐directed expression of the human adenylyl cyclase type 8 (AC8)

Phosphodiesterase 11A (PDE11A) Gene Defects in Patients with ACTH-Independent Macronodular Adrenal Hyperplasia (AIMAH): Functional Variants May Contribute to Genetic Susceptibility of Bilateral Adrenal Tumors

Differential changes in cyclic adenosine 3′‐5′ monophosphate (cAMP) effectors and major Ca2+ handling proteins during diabetic cardiomyopathy

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