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Cerebrospinal fluid oxytocin, life history of aggression, and personality disorder

Psychoneuroendocrinology, 2009-11, Vol.34 (10), p.1567-1573

2009

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Estrogen treatment increases the levels of regulator of G protein signaling-Z1 in the hypothalamic paraventricular nucleus: Possible role in desensitization of 5-hydroxytryptamine1A receptors

Neuroscience, 2004, Vol.127 (2), p.261-267

2004

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Estradiol induces partial desensitization of serotonin 1A receptor signaling in the paraventricular nucleus of the hypothalamus and alters expression and interaction of RGSZ1 and Gαz

Neuropharmacology, 2012-04, Vol.62 (5-6), p.2040-2049

2012

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Amygdaloid lesions: differential effect on conditioned stress and immobilization-induced increases in corticosterone and renin secretion

Neuroendocrinology, 1991-08, Vol.54 (2), p.89-95

1991

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Serotonin and the Neuroendocrine Regulation of the Hypothalamic–Pituitary–Adrenal Axis in Health and Disease

Vitamins and Hormones, 2003, Vol.66, p.189-255

2003

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Cocaine-mediated supersensitivity of 5-HT2A receptors in hypothalamic paraventricular nucleus is a withdrawal-induced phenomenon

Neuroscience, 2006-11, Vol.143 (1), p.7-13

2006

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Chronic olanzapine activates the Stat3 signal transduction pathway and alters expression of components of the 5-HT2A receptor signaling system in rat frontal cortex

Neuropharmacology, 2007-09, Vol.53 (4), p.552-562

2007

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Differential serotonergic innervation of individual hypothalamic nuclei and other forebrain regions by the dorsal and median midbrain raphe nuclei

Brain research, 1979-02, Vol.162 (1), p.45-54

1979

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Cocaine-mediated supersensitivity of 5-HT 2A receptors in hypothalamic paraventricular nucleus is a withdrawal-induced phenomenon

Neuroscience, 2006, Vol.143 (1), p.7-13

2006

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Treatment of cycling female rats with fluoxetine induces desensitization of hypothalamic 5-HT1A receptors with no change in 5-HT2A receptors

Neuropharmacology, 2002-07, Vol.43 (1), p.45-54

2002

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Chronic olanzapine activates the Stat3 signal transduction pathway and alters expression of components of the 5-HT 2A receptor signaling system in rat frontal cortex

Neuropharmacology, 2007, Vol.53 (4), p.552-562

2007

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d-Fenfluramine induces serotonin-mediated Fos expression in corticotropin-releasing factor and oxytocin neurons of the hypothalamus, and serotonin-independent Fos expression in enkephalin and neurotensin neurons of the amygdala

Neuroscience, 1999-03, Vol.90 (3), p.851-858

1999

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Estrogen treatment increases the levels of regulator of G protein signaling-Z1 in the hypothalamic paraventricular nucleus: possible role in desensitization of 5-hydroxytryptamine 1A receptors

Neuroscience, 2004, Vol.127 (2), p.261-267

2004

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P.1.17 Separate and combined effects of fluoxetine and a 5-HT 1A receptor agonist or antagonist on 5-HT 1A and 5-HT 1B autoreceptor activity in the rat brain

European neuropsychopharmacology, 2004, Vol.14, p.S15-S16

2004

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P.1.17 Separate and combined effects of fluoxetine and a 5-HT1A receptor agonist or antagonist on 5-HT1A and 5-HT1B autoreceptor activity in the rat brain

European neuropsychopharmacology, 2004-01, Vol.14, p.S15-S16

2004

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Treatment of cycling female rats with fluoxetine induces desensitization of hypothalamic 5-HT 1A receptors with no change in 5-HT 2A receptors

Neuropharmacology, 2002, Vol.43 (1), p.45-54

2002

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The 5-HT1A and 5-HT2A/2C receptor antagonists WAY-100635 and ritanserin do not attenuate D-fenfluramine-induced Fos expression in the brain

Brain research, 1998-04, Vol.791 (1-2), p.67-74

1998

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p-Chlorophenylalanine and fluoxetine inhibit d-fenfluramine-induced Fos expression in the paraventricular nucleus, cingulate cortex and frontal cortex but not in other forebrain and brainstem regions

Brain research, 1997-11, Vol.774 (1), p.94-105

1997

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The 5-HT 1A and 5-HT 2A/2C receptor antagonists WAY-100635 and ritanserin do not attenuate d-fenfluramine-induced Fos expression in the brain

Brain research, 1998, Vol.791 (1), p.67-74

1998

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Fluoxetine gradually increases [ 125 I]DOI-labelled 5-HT 2A/2C receptors in the hypothalamus without changing the levels of G q - and G 11 -proteins

Brain research, 1997-11, Vol.775 (1-2), p.225-228

1997

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Repeated cocaine exposure inhibits the adrenocorticotropic hormone response to the serotonin releaser d-fenfluramine and the 5-HT1A agonist, 8-OH-DPAT

Neuropharmacology, 1994-03, Vol.33 (3-4), p.335-342

1994

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Fluoxetine gradually increases [125I]DOI-labelled 5-HT2A/2C receptors in the hypothalamus without changing the levels of Gq- and G11-proteins

Brain research, 1997-11, Vol.775 (1-2), p.225-228

1997

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Serotonin and stress-induced increases in renin secretion are not blocked by sympathectomy/adrenal medullectomy but are blocked by β antagonists

Brain research, 1995-11, Vol.698 (1), p.185-192

1995

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Prenatal exposure to fluoxetine (Prozac®) alters brain serotonergic systems in offspring

Neurotoxicology and teratology, 1997-05, Vol.19 (3), p.246-246

1997

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Dose-response of fluoxetine-induced desensitization of hypothalamic 5-HT5A receptors

Biological psychiatry (1969), 1997-07, Vol.42 (1), p.67

1997

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Cerebrospinal fluid oxytocin, life history of aggression, and personality disorder

Estrogen treatment increases the levels of regulator of G protein signaling-Z1 in the hypothalamic paraventricular nucleus: Possible role in desensitization of 5-hydroxytryptamine1A receptors

Estradiol induces partial desensitization of serotonin 1A receptor signaling in the paraventricular nucleus of the hypothalamus and alters expression and interaction of RGSZ1 and Gαz

Amygdaloid lesions: differential effect on conditioned stress and immobilization-induced increases in corticosterone and renin secretion

Serotonin and the Neuroendocrine Regulation of the Hypothalamic–Pituitary–Adrenal Axis in Health and Disease

Cocaine-mediated supersensitivity of 5-HT2A receptors in hypothalamic paraventricular nucleus is a withdrawal-induced phenomenon

Chronic olanzapine activates the Stat3 signal transduction pathway and alters expression of components of the 5-HT2A receptor signaling system in rat frontal cortex

Differential serotonergic innervation of individual hypothalamic nuclei and other forebrain regions by the dorsal and median midbrain raphe nuclei

Cocaine-mediated supersensitivity of 5-HT 2A receptors in hypothalamic paraventricular nucleus is a withdrawal-induced phenomenon

Treatment of cycling female rats with fluoxetine induces desensitization of hypothalamic 5-HT1A receptors with no change in 5-HT2A receptors

Chronic olanzapine activates the Stat3 signal transduction pathway and alters expression of components of the 5-HT 2A receptor signaling system in rat frontal cortex

d-Fenfluramine induces serotonin-mediated Fos expression in corticotropin-releasing factor and oxytocin neurons of the hypothalamus, and serotonin-independent Fos expression in enkephalin and neurotensin neurons of the amygdala

Estrogen treatment increases the levels of regulator of G protein signaling-Z1 in the hypothalamic paraventricular nucleus: possible role in desensitization of 5-hydroxytryptamine 1A receptors

P.1.17 Separate and combined effects of fluoxetine and a 5-HT 1A receptor agonist or antagonist on 5-HT 1A and 5-HT 1B autoreceptor activity in the rat brain

P.1.17 Separate and combined effects of fluoxetine and a 5-HT1A receptor agonist or antagonist on 5-HT1A and 5-HT1B autoreceptor activity in the rat brain

Treatment of cycling female rats with fluoxetine induces desensitization of hypothalamic 5-HT 1A receptors with no change in 5-HT 2A receptors

The 5-HT1A and 5-HT2A/2C receptor antagonists WAY-100635 and ritanserin do not attenuate D-fenfluramine-induced Fos expression in the brain

p-Chlorophenylalanine and fluoxetine inhibit d-fenfluramine-induced Fos expression in the paraventricular nucleus, cingulate cortex and frontal cortex but not in other forebrain and brainstem regions

The 5-HT 1A and 5-HT 2A/2C receptor antagonists WAY-100635 and ritanserin do not attenuate d-fenfluramine-induced Fos expression in the brain

Fluoxetine gradually increases [ 125 I]DOI-labelled 5-HT 2A/2C receptors in the hypothalamus without changing the levels of G q - and G 11 -proteins

Repeated cocaine exposure inhibits the adrenocorticotropic hormone response to the serotonin releaser d-fenfluramine and the 5-HT1A agonist, 8-OH-DPAT

Fluoxetine gradually increases [125I]DOI-labelled 5-HT2A/2C receptors in the hypothalamus without changing the levels of Gq- and G11-proteins

Serotonin and stress-induced increases in renin secretion are not blocked by sympathectomy/adrenal medullectomy but are blocked by β antagonists

Prenatal exposure to fluoxetine (Prozac®) alters brain serotonergic systems in offspring

Dose-response of fluoxetine-induced desensitization of hypothalamic 5-HT5A receptors

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