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Rapamycin decelerates cellular senescence

Cell cycle (Georgetown, Tex.), 2009-06, Vol.8 (12), p.1888-1895

2009

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Pseudo-DNA damage response in senescent cells

Cell cycle (Georgetown, Tex.), 2009-12, Vol.8 (24), p.4112-4118

2009

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Isolation and Characterization of Human Colon Adenocarcinoma Stem-Like Cells Based on the Endogenous Expression of the Stem Markers

International journal of molecular sciences, 2021-04, Vol.22 (9), p.4682

2021

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Suppression of replicative senescence by rapamycin in rodent embryonic cells

Cell cycle (Georgetown, Tex.), 2012-06, Vol.11 (12), p.2402-2407

2012

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G1 checkpoint is compromised in mouse ESCs due to functional uncoupling of p53-p21Waf1 signaling

Cell cycle (Georgetown, Tex.), 2016-01, Vol.15 (1), p.52-63

2016

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Activation of DNA damage response signaling in mouse embryonic stem cells

Cell cycle (Georgetown, Tex.), 2008-09, Vol.7 (18), p.2922-2928

2008

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AMPK/Ulk1-dependent autophagy as a key mTOR regulator in the context of cell pluripotency

Cell death & disease, 2019-03, Vol.10 (4), p.260-260, Article 260

2019

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Resveratrol-induced p53 activation is associated with autophagy in mouse embryonic stem cells

Biochemical and biophysical research communications, 2018-09, Vol.503 (3), p.2180-2185

2018

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Approaches and Protocols to Analyze Autophagy and Its Role in Death of Apoptosis-Resistant Senescent Tumor Cells

Methods in molecular biology (Clifton, N.J.), 2022, Vol.2445, p.139

2022

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Sustained activation of DNA damage response in irradiated apoptosis-resistant cells induces reversible senescence associated with mTOR downregulation and expression of stem cell markers

Cell cycle (Georgetown, Tex.), 2014-05, Vol.13 (9), p.1424-1439

2014

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HDAC inhibitor sodium butyrate sensitizes E1A+Ras-transformed cells to DNA damaging agents by facilitating formation and persistence of γH2AX foci

Cancer biology & therapy, 2011-12, Vol.12 (12), p.1069-1077

2011

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β-Catenin in pluripotency: adhering to self-renewal or Wnting to differentiate?

International review of cell and molecular biology, 2014, Vol.312, p.53-78

2014

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Targeted elimination of senescent Ras-transformed cells by suppression of MEK/ERK pathway

Aging (Albany, NY.), 2017-11, Vol.9 (11), p.2352-2375

2017

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Resveratrol enhances pluripotency of mouse embryonic stem cells by activating AMPK/Ulk1 pathway

Cell death discovery, 2019-02, Vol.5 (1), p.61-61, Article 61

2019

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Rapamycin induces pluripotent genes associated with avoidance of replicative senescence

Cell cycle (Georgetown, Tex.), 2013-12, Vol.12 (24), p.3841-3851

2013

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Latest progress in tyrosine kinase inhibitors

Oncotarget, 2014-03, Vol.5 (5), p.1157-1161

2014

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mTOR kinase inhibitor pp242 causes mitophagy terminated by apoptotic cell death in E1A-Ras transformed cells

Oncotarget, 2015-12, Vol.6 (42), p.44905-44926

2015

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New Insights into Cell Cycle Regulation and DNA Damage Response in Embryonic Stem Cells

International Review of Cell and Molecular Biology, 2012, Vol.299, p.161-198

2012

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Novel mechanism of JNK pathway activation by adenoviral E1A

Oncotarget, 2014-04, Vol.5 (8), p.2176-2186

2014

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G1/S Arrest Induced by Histone Deacetylase Inhibitor Sodium Butyrate in E1A + Ras-transformed Cells Is Mediated through Down-regulation of E2F Activity and Stabilization of β-Catenin

The Journal of biological chemistry, 2006-07, Vol.281 (30), p.21040-21051

2006

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Restoration of G1/S Arrest in E1A+c-Ha-ras-Transformed Cells by Bcl-2 Overexpression

Cell cycle (Georgetown, Tex.), 2004-11, Vol.3 (11), p.1427-1432

2004

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e2f1 gene is a new member of Wnt/β-catenin/Tcf-regulated genes

Biochemical and biophysical research communications, 2010-01, Vol.391 (1), p.142-146

2010

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An integrated approach for monitoring cell senescence

Methods in molecular biology (Clifton, N.J.), 2013, Vol.965, p.383

2013

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p21Waf1 is required for complete oncogenic transformation of mouse embryo fibroblasts by E1Aad5 and c-Ha-ras oncogenes

Biochimie, 2011-09, Vol.93 (9), p.1408-1414

2011

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Phosphoinositide 3-kinase inhibitor LY294002 but not serum withdrawal suppresses proliferation of murine embryonic stem cells

Cell biology international, 2007-04, Vol.31 (4), p.330-337

2007

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Rapamycin decelerates cellular senescence

Pseudo-DNA damage response in senescent cells

Isolation and Characterization of Human Colon Adenocarcinoma Stem-Like Cells Based on the Endogenous Expression of the Stem Markers

Suppression of replicative senescence by rapamycin in rodent embryonic cells

G1 checkpoint is compromised in mouse ESCs due to functional uncoupling of p53-p21Waf1 signaling

Activation of DNA damage response signaling in mouse embryonic stem cells

AMPK/Ulk1-dependent autophagy as a key mTOR regulator in the context of cell pluripotency

Resveratrol-induced p53 activation is associated with autophagy in mouse embryonic stem cells

Approaches and Protocols to Analyze Autophagy and Its Role in Death of Apoptosis-Resistant Senescent Tumor Cells

Sustained activation of DNA damage response in irradiated apoptosis-resistant cells induces reversible senescence associated with mTOR downregulation and expression of stem cell markers

HDAC inhibitor sodium butyrate sensitizes E1A+Ras-transformed cells to DNA damaging agents by facilitating formation and persistence of γH2AX foci

β-Catenin in pluripotency: adhering to self-renewal or Wnting to differentiate?

Targeted elimination of senescent Ras-transformed cells by suppression of MEK/ERK pathway

Resveratrol enhances pluripotency of mouse embryonic stem cells by activating AMPK/Ulk1 pathway

Rapamycin induces pluripotent genes associated with avoidance of replicative senescence

Latest progress in tyrosine kinase inhibitors

mTOR kinase inhibitor pp242 causes mitophagy terminated by apoptotic cell death in E1A-Ras transformed cells

New Insights into Cell Cycle Regulation and DNA Damage Response in Embryonic Stem Cells

Novel mechanism of JNK pathway activation by adenoviral E1A

G1/S Arrest Induced by Histone Deacetylase Inhibitor Sodium Butyrate in E1A + Ras-transformed Cells Is Mediated through Down-regulation of E2F Activity and Stabilization of β-Catenin

Restoration of G1/S Arrest in E1A+c-Ha-ras-Transformed Cells by Bcl-2 Overexpression

e2f1 gene is a new member of Wnt/β-catenin/Tcf-regulated genes

An integrated approach for monitoring cell senescence

p21Waf1 is required for complete oncogenic transformation of mouse embryo fibroblasts by E1Aad5 and c-Ha-ras oncogenes

Phosphoinositide 3-kinase inhibitor LY294002 but not serum withdrawal suppresses proliferation of murine embryonic stem cells

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