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Mutation in glycerol-3-phosphate dehydrogenase 1-like gene (GPD1-L) decreases cardiac Na+ current and causes inherited arrhythmias

Circulation (New York, N.Y.), 2007-11, Vol.116 (20), p.2260-2268

2007

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Functional Interactions between Distinct Sodium Channel Cytoplasmic Domains through the Action of Calmodulin

The Journal of biological chemistry, 2009-03, Vol.284 (13), p.8846-8854

2009

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Two Distinct Congenital Arrhythmias Evoked by a Multidysfunctional Na+ Channel

Circulation research, 2000-05, Vol.86 (9), p.e91-e97

2000

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Oxidative Mediated Lipid Peroxidation Recapitulates Proarrhythmic Effects on Cardiac Sodium Channels

Circulation research, 2005-12, Vol.97 (12), p.1262-1269

2005

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Cellular arrhythmogenic effects of congenital and acquired long-QT syndrome in the heterogeneous myocardium

Circulation (New York, N.Y.), 2000-03, Vol.101 (10), p.1192-1198

2000

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Genetics of acquired long QT syndrome

The Journal of clinical investigation, 2005-08, Vol.115 (8), p.2025-2032

2005

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A sodium channel pore mutation causing Brugada syndrome

Heart rhythm, 2007-01, Vol.4 (1), p.46-53

2007

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Sex, Age, and Regional Differences in L-Type Calcium Current Are Important Determinants of Arrhythmia Phenotype in Rabbit Hearts With Drug-Induced Long QT Type 2

Circulation research, 2008-05, Vol.102 (9), p.e86-e100

2008

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Effects of I(Kr) and I(Ks) heterogeneity on action potential duration and its rate dependence: A simulation study

Circulation (New York, N.Y.), 1999-05, Vol.99 (18), p.2466

1999

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Allelic variants in long-QT disease genes in patients with drug-associated torsades de pointes

Circulation (New York, N.Y.), 2002-04, Vol.105 (16), p.1943-1948

2002

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Compound-specific Na+ channel pore conformational changes induced by local anaesthetics

The Journal of physiology, 2005-04, Vol.564 (1), p.21-31

2005

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Recreating an artificial biological pacemaker: Insights from a theoretical model

Heart rhythm, 2006-07, Vol.3 (7), p.824-831

2006

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A sodium-channel mutation causes isolated cardiac conduction disease

Nature (London), 2001-02, Vol.409 (6823), p.1043-1047

2001

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Clinical, genetic, and biophysical characterization of SCN5A mutations associated with atrioventricular conduction block

Circulation (New York, N.Y.), 2002-01, Vol.105 (3), p.341-346

2002

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Quantitation of protein kinase A-mediated trafficking of cardiac sodium channels in living cells

Cardiovascular research, 2006-11, Vol.72 (2), p.250-261

2006

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Inherited Sodium Channelopathies A Continuum of Channel Dysfunction

Trends in cardiovascular medicine, 2004, Vol.14 (1), p.28-35

2004

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New mechanism contributing to drug-induced arrhythmia : Rescue of a misprocessed LQT3 mutant

Circulation (New York, N.Y.), 2005-11, Vol.112 (21), p.3239-3246

2005

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HERG Is Protected from Pharmacological Block by α-1,2-Glucosyltransferase Function

The Journal of biological chemistry, 2007-02, Vol.282 (8), p.5506-5513

2007

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Gating-dependent mechanisms for flecainide action in SCN5A-linked arrhythmia syndromes

Circulation (New York, N.Y.), 2001-09, Vol.104 (10), p.1200-1205

2001

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Potential Role of Isoketals Formed Via the Isoprostane Pathway of Lipid Peroxidation in Ischemic Arrhythmias

Journal of cardiovascular pharmacology, 2007-11, Vol.50 (5), p.480-486

2007

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SCN5A– Linked Disease Syndromes: Complex Monogenic Disorders of Cardiac Rhythm

Future cardiology, 2005-01, Vol.1 (1), p.29-38

Viswanathan, Prakash C

2005

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Pause induced early afterdepolarizations in the long QT syndrome: a simulation study

Cardiovascular research, 1999-05, Vol.42 (2), p.530-542

1999

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Pacemaker Channels and Sinus Node Arrhythmia

Trends in cardiovascular medicine, 2004, Vol.14 (1), p.23-28

2004

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Allelic variants in long-QT disease genes in patients with drug-associated torsades de pointes

Circulation (New York, N.Y.), 2002, Vol.105 (16), p.1943-1948

2002

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Abstract 18586: GPD1-L Inhibits the Cardiac Sodium Channel Nav1.5 by decreasing SIRTUIN1 Activity

Circulation (New York, N.Y.), 2013-11, Vol.128 (suppl_22)

2013

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Mutation in glycerol-3-phosphate dehydrogenase 1-like gene (GPD1-L) decreases cardiac Na+ current and causes inherited arrhythmias

Functional Interactions between Distinct Sodium Channel Cytoplasmic Domains through the Action of Calmodulin

Two Distinct Congenital Arrhythmias Evoked by a Multidysfunctional Na+ Channel

Oxidative Mediated Lipid Peroxidation Recapitulates Proarrhythmic Effects on Cardiac Sodium Channels

Cellular arrhythmogenic effects of congenital and acquired long-QT syndrome in the heterogeneous myocardium

Genetics of acquired long QT syndrome

A sodium channel pore mutation causing Brugada syndrome

Sex, Age, and Regional Differences in L-Type Calcium Current Are Important Determinants of Arrhythmia Phenotype in Rabbit Hearts With Drug-Induced Long QT Type 2

Effects of I(Kr) and I(Ks) heterogeneity on action potential duration and its rate dependence: A simulation study

Allelic variants in long-QT disease genes in patients with drug-associated torsades de pointes

Compound-specific Na+ channel pore conformational changes induced by local anaesthetics

Recreating an artificial biological pacemaker: Insights from a theoretical model

A sodium-channel mutation causes isolated cardiac conduction disease

Clinical, genetic, and biophysical characterization of SCN5A mutations associated with atrioventricular conduction block

Quantitation of protein kinase A-mediated trafficking of cardiac sodium channels in living cells

Inherited Sodium Channelopathies A Continuum of Channel Dysfunction

New mechanism contributing to drug-induced arrhythmia : Rescue of a misprocessed LQT3 mutant

HERG Is Protected from Pharmacological Block by α-1,2-Glucosyltransferase Function

Gating-dependent mechanisms for flecainide action in SCN5A-linked arrhythmia syndromes

Potential Role of Isoketals Formed Via the Isoprostane Pathway of Lipid Peroxidation in Ischemic Arrhythmias

SCN5A– Linked Disease Syndromes: Complex Monogenic Disorders of Cardiac Rhythm

Pause induced early afterdepolarizations in the long QT syndrome: a simulation study

Pacemaker Channels and Sinus Node Arrhythmia

Allelic variants in long-QT disease genes in patients with drug-associated torsades de pointes

Abstract 18586: GPD1-L Inhibits the Cardiac Sodium Channel Nav1.5 by decreasing SIRTUIN1 Activity

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