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Evaluation of the Combined Effect of p53 Codon 72 Polymorphism and Hotspot Mutations in Response to Anticancer Drugs

Clinical cancer research, 2005-06, Vol.11 (12), p.4348-4356

2005

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Characterization of the 5'flanking Region of the Human chk1 Gene: Identification of E2F1 Functional Sites

Cell cycle (Georgetown, Tex.), 2003-11, Vol.2 (6), p.603-608

2003

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p73 overexpression increases VEGF and reduces thrombospondin-1 production: implications for tumor angiogenesis

Oncogene, 2001-11, Vol.20 (50), p.7293-7300

2001

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Cisplatinum and Taxol Induce Different Patterns of p53 Phosphorylation

Neoplasia (New York, N.Y.), 2001-01, Vol.3 (1), p.10-16

2001

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Inactivation of p53 in a Human Ovarian Cancer Cell Line Increases the Sensitivity to Paclitaxel by Inducing G2/M Arrest and Apoptosis

Experimental cell research, 1998-05, Vol.241 (1), p.96-101

1998

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Cancer-derived p53 mutants suppress p53-target gene expression--potential mechanism for gain of function of mutant p53

Nucleic acids research, 2007-03, Vol.35 (6), p.2093

2007

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Cooperation between p53 and hMLH1 in a Human Colocarcinoma Cell Line in Response to DNA Damage

Clinical cancer research, 1999-04, Vol.5 (4), p.937-941

1999

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Introduction of wild-type p53 in a human ovarian cancer cell line not expressing endogenous p53

Nucleic acids research, 1994-03, Vol.22 (6), p.1012-1017

1994

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p73: A chiaroscuro gene in cancer

European journal of cancer (1990), 2007-06, Vol.43 (9), p.1361-1372

2007

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p73 supports cellular growth through c-Jun-dependent AP-1 transactivation

Nature cell biology, 2007-06, Vol.9 (6), p.698-706

2007

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Mechanism of resistance to cisplatin in a human ovarian‐carcinoma cell line selected for resistance to doxorubicin: Possible role of p53

International journal of cancer, 1997-07, Vol.72 (1), p.155-159

1997

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p 73: A chiaroscuro gene in cancer

European journal of cancer (1990), 2007, Vol.43 (9), p.1361-1372

2007

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Variolin B and its derivate deoxy-variolin B: New marine natural compounds with cyclin-dependent kinase inhibitor activity

European journal of cancer (1990), 2005-10, Vol.41 (15), p.2366-2377

2005

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p73 competes with p53 and attenuates its response in a human ovarian cancer cell line

Nucleic acids research, 2000-01, Vol.28 (2), p.513

2000

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Human papillomavirus type 16 E6‐enhanced susceptibility to apoptosis induced by TNF in A2780 human ovarian cancer cell line

International journal of cancer, 2002-02, Vol.97 (6), p.732-739

2002

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p53 status does not affect sensitivity of human ovarian cancer cell lines to paclitaxel

Cancer research (Chicago, Ill.), 1997-03, Vol.57 (5), p.870-874

1997

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Characterization of the 5'flanking region of the human Chk1 gene: identification of E2F1 functional sites

Cell cycle (Georgetown, Tex.), 2003-11, Vol.2 (6), p.604-609

2003

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Inhibition of cancer cell growth and c-Myc transcriptional activity by a c-Myc helix 1-type peptide fused to an internalization sequence

Cancer research (Chicago, Ill.), 1998-08, Vol.58 (16), p.3654-3659

1998

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Decreased cytotoxic effects of doxorubicin in a human ovarian cancer‐cell line expressing wild‐type p53 and WAF1/CIP1 genes

International journal of cancer, 1995-05, Vol.61 (3), p.397-401

1995

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Decreased cytotoxic effects of doxorubicin in a human ovarian cancer‐cell line expressing wild‐type p53 and WAF 1/ CIP 1 genes

International journal of cancer, 1995-05, Vol.61 (3), p.397-401

1995

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DNA damage induces transcriptional activation of p73 by removing C-EBPalpha repression on E2F1

Nucleic acids research, 2003-11, Vol.31 (22), p.6624

2003

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Changes in Cyclins and Cyclin-Dependent Kinases Induced by DNA Damaging Agents in a Human Ovarian Cancer Cell Line Expressing Mutated or Wild-Type P53

Experimental cell research, 1996-09, Vol.227 (2), p.380-385

1996

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Genetic alterations in ovarian cancer cells that might account for sensitivity to chemotherapy in patients

International Review of Cytology, 2002, Vol.219, p.157-198

2002

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Evaluation of the Combined Effect of p53 Codon 72 Polymorphism and Hotspot Mutations in Response to Anticancer Drugs

Characterization of the 5'flanking Region of the Human chk1 Gene: Identification of E2F1 Functional Sites

p73 overexpression increases VEGF and reduces thrombospondin-1 production: implications for tumor angiogenesis

Cisplatinum and Taxol Induce Different Patterns of p53 Phosphorylation

Inactivation of p53 in a Human Ovarian Cancer Cell Line Increases the Sensitivity to Paclitaxel by Inducing G2/M Arrest and Apoptosis

Cancer-derived p53 mutants suppress p53-target gene expression--potential mechanism for gain of function of mutant p53

Cooperation between p53 and hMLH1 in a Human Colocarcinoma Cell Line in Response to DNA Damage

Introduction of wild-type p53 in a human ovarian cancer cell line not expressing endogenous p53

p73: A chiaroscuro gene in cancer

p73 supports cellular growth through c-Jun-dependent AP-1 transactivation

Mechanism of resistance to cisplatin in a human ovarian‐carcinoma cell line selected for resistance to doxorubicin: Possible role of p53

p 73: A chiaroscuro gene in cancer

Variolin B and its derivate deoxy-variolin B: New marine natural compounds with cyclin-dependent kinase inhibitor activity

p73 competes with p53 and attenuates its response in a human ovarian cancer cell line

Human papillomavirus type 16 E6‐enhanced susceptibility to apoptosis induced by TNF in A2780 human ovarian cancer cell line

p53 status does not affect sensitivity of human ovarian cancer cell lines to paclitaxel

Characterization of the 5'flanking region of the human Chk1 gene: identification of E2F1 functional sites

Inhibition of cancer cell growth and c-Myc transcriptional activity by a c-Myc helix 1-type peptide fused to an internalization sequence

Decreased cytotoxic effects of doxorubicin in a human ovarian cancer‐cell line expressing wild‐type p53 and WAF1/CIP1 genes

Decreased cytotoxic effects of doxorubicin in a human ovarian cancer‐cell line expressing wild‐type p53 and WAF 1/ CIP 1 genes

DNA damage induces transcriptional activation of p73 by removing C-EBPalpha repression on E2F1

Changes in Cyclins and Cyclin-Dependent Kinases Induced by DNA Damaging Agents in a Human Ovarian Cancer Cell Line Expressing Mutated or Wild-Type P53

Genetic alterations in ovarian cancer cells that might account for sensitivity to chemotherapy in patients

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