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Pharmacologic concentrations of ascorbate are achieved by parenteral administration and exhibit antitumoral effects

Free radical biology & medicine, 2009-07, Vol.47 (1), p.32-40

2009

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Regulation of catalase expression in healthy and cancerous cells

Free radical biology & medicine, 2015-10, Vol.87, p.84-97

2015

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Role of AMPK activation in oxidative cell damage: Implications for alcohol-induced liver disease

Biochemical pharmacology, 2013-07, Vol.86 (2), p.200-209

2013

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Targeting the lactate transporter MCT1 in endothelial cells inhibits lactate-induced HIF-1 activation and tumor angiogenesis

PloS one, 2012-03, Vol.7 (3), p.e33418-e33418

2012

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Lactate activates HIF-1 in oxidative but not in Warburg-phenotype human tumor cells

PloS one, 2012-10, Vol.7 (10), p.e46571-e46571

2012

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Catalase overexpression in mammary cancer cells leads to a less aggressive phenotype and an altered response to chemotherapy

Biochemical pharmacology, 2011-11, Vol.82 (10), p.1384-1390

2011

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p38MAPK-regulated induction of p62 and NBR1 after photodynamic therapy promotes autophagic clearance of ubiquitin aggregates and reduces reactive oxygen species levels by supporting Nrf2–antioxidant signaling

Free radical biology & medicine, 2014-02, Vol.67, p.292-303

2014

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Restoring specific lactobacilli levels decreases inflammation and muscle atrophy markers in an acute leukemia mouse model

PloS one, 2012-06, Vol.7 (6), p.e37971-e37971

2012

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Arsenic Trioxide Treatment Decreases the Oxygen Consumption Rate of Tumor Cells and Radiosensitizes Solid Tumors

Cancer research (Chicago, Ill.), 2012-01, Vol.72 (2), p.482-490

2012

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Biological evaluation of donor-acceptor aminonaphthoquinones as antitumor agents

European journal of medicinal chemistry, 2010-12, Vol.45 (12), p.6052-6057

2010

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Use of a cocktail of spin traps for fingerprinting large range of free radicals in biological systems

PloS one, 2017-03, Vol.12 (3), p.e0172998

2017

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Comparison of methods for measuring oxygen consumption in tumor cells in vitro

Analytical biochemistry, 2010-01, Vol.396 (2), p.250-256

2010

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Hsp90 is cleaved by reactive oxygen species at a highly conserved N-terminal amino acid motif

PloS one, 2012-07, Vol.7 (7), p.e40795-e40795

2012

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Redox-active quinones and ascorbate: an innovative cancer therapy that exploits the vulnerability of cancer cells to oxidative stress

Anti-cancer agents in medicinal chemistry, 2011-02, Vol.11 (2), p.213-221

2011

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Hsp90 cleavage by an oxidative stress leads to its client proteins degradation and cancer cell death

Biochemical pharmacology, 2009-02, Vol.77 (3), p.375-383

2009

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Role of glycolysis inhibition and poly(ADP‐ribose) polymerase activation in necrotic‐like cell death caused by ascorbate/menadione‐induced oxidative stress in K562 human chronic myelogenous leukemic cells

International journal of cancer, 2007-03, Vol.120 (6), p.1192-1197

2007

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Biological evaluation of 3-acyl-2-arylamino-1,4-naphthoquinones as inhibitors of Hsp90 chaperoning function

Current topics in medicinal chemistry, 2012-10, Vol.12 (19), p.2094-2102

2012

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Botulinum toxin potentiates cancer radiotherapy and chemotherapy

Clinical cancer research, 2006-02, Vol.12 (4), p.1276-1283

2006

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p38(MAPK)-regulated induction of p62 and NBR1 after photodynamic therapy promotes autophagic clearance of ubiquitin aggregates and reduces reactive oxygen species levels by supporting Nrf2-antioxidant signaling

Free radical biology & medicine, 2014-02, Vol.67, p.292-303

2014

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Targeting lactate-fueled respiration selectively kills hypoxic tumor cells in mice

The Journal of clinical investigation, 2008-12, Vol.118 (12), p.3930-3942

2008

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Chromatin remodeling regulates catalase expression during cancer cells adaptation to chronic oxidative stress

Free radical biology & medicine, 2016-10, Vol.99, p.436-450

2016

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Overexpression of GRP94 in breast cancer cells resistant to oxidative stress promotes high levels of cancer cell proliferation and migration: Implications for tumor recurrence

Free radical biology & medicine, 2012-03, Vol.52 (6), p.993-1002

2012

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Catalase expression in MCF-7 breast cancer cells is mainly controlled by PI3K/Akt/mTor signaling pathway

Biochemical pharmacology, 2014-05, Vol.89 (2), p.217-223

2014

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Intracellular ATP levels determine cell death fate of cancer cells exposed to both standard and redox chemotherapeutic agents

Biochemical pharmacology, 2011-12, Vol.82 (11), p.1540-1548

2011

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AICAR induces Nrf2 activation by an AMPK-independent mechanism in hepatocarcinoma cells

Biochemical pharmacology, 2014-09, Vol.91 (2), p.168-180

2014

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Pharmacologic concentrations of ascorbate are achieved by parenteral administration and exhibit antitumoral effects

Regulation of catalase expression in healthy and cancerous cells

Role of AMPK activation in oxidative cell damage: Implications for alcohol-induced liver disease

Targeting the lactate transporter MCT1 in endothelial cells inhibits lactate-induced HIF-1 activation and tumor angiogenesis

Lactate activates HIF-1 in oxidative but not in Warburg-phenotype human tumor cells

Catalase overexpression in mammary cancer cells leads to a less aggressive phenotype and an altered response to chemotherapy

p38MAPK-regulated induction of p62 and NBR1 after photodynamic therapy promotes autophagic clearance of ubiquitin aggregates and reduces reactive oxygen species levels by supporting Nrf2–antioxidant signaling

Restoring specific lactobacilli levels decreases inflammation and muscle atrophy markers in an acute leukemia mouse model

Arsenic Trioxide Treatment Decreases the Oxygen Consumption Rate of Tumor Cells and Radiosensitizes Solid Tumors

Biological evaluation of donor-acceptor aminonaphthoquinones as antitumor agents

Use of a cocktail of spin traps for fingerprinting large range of free radicals in biological systems

Comparison of methods for measuring oxygen consumption in tumor cells in vitro

Hsp90 is cleaved by reactive oxygen species at a highly conserved N-terminal amino acid motif

Redox-active quinones and ascorbate: an innovative cancer therapy that exploits the vulnerability of cancer cells to oxidative stress

Hsp90 cleavage by an oxidative stress leads to its client proteins degradation and cancer cell death

Role of glycolysis inhibition and poly(ADP‐ribose) polymerase activation in necrotic‐like cell death caused by ascorbate/menadione‐induced oxidative stress in K562 human chronic myelogenous leukemic cells

Biological evaluation of 3-acyl-2-arylamino-1,4-naphthoquinones as inhibitors of Hsp90 chaperoning function

Botulinum toxin potentiates cancer radiotherapy and chemotherapy

p38(MAPK)-regulated induction of p62 and NBR1 after photodynamic therapy promotes autophagic clearance of ubiquitin aggregates and reduces reactive oxygen species levels by supporting Nrf2-antioxidant signaling

Targeting lactate-fueled respiration selectively kills hypoxic tumor cells in mice

Chromatin remodeling regulates catalase expression during cancer cells adaptation to chronic oxidative stress

Overexpression of GRP94 in breast cancer cells resistant to oxidative stress promotes high levels of cancer cell proliferation and migration: Implications for tumor recurrence

Catalase expression in MCF-7 breast cancer cells is mainly controlled by PI3K/Akt/mTor signaling pathway

Intracellular ATP levels determine cell death fate of cancer cells exposed to both standard and redox chemotherapeutic agents

AICAR induces Nrf2 activation by an AMPK-independent mechanism in hepatocarcinoma cells

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