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Pre‐ and postsynaptic neuromuscular junction abnormalities in musk myasthenia

Muscle & nerve, 2010-08, Vol.42 (2), p.283-288

2010

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Increased expression of rapsyn in muscles prevents acetylcholine receptor loss in experimental autoimmune myasthenia gravis

Brain (London, England : 1878), 2005-10, Vol.128 (10), p.2327-2337

2005

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Oxidant treatment causes a dose-dependent phenotype of apoptosis in cultured motoneurons

Journal of neuroscience research, 1998-12, Vol.54 (6), p.778-786

1998

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Schwartz-Jampel syndrome: I. Clinical, electromyographic, and histologic studies

Muscle & nerve, 1990-06, Vol.13 (6), p.516-527

1990

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Tubuloreticular Structures in Different Types of Myositis: Implications for Pathogenesis

Ultrastructural pathology, 2008-01, Vol.32 (4), p.123-126

2008

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Oxidant treatment causes a dose‐dependent phenotype of apoptosis in cultured motoneurons

Journal of neuroscience research, 1998-12, Vol.54 (6), p.778-786

1998

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Tubuloreticular Structures in Different Types of Myositis: Implications for Pathogenesis1

Ultrastructural pathology, 2008, Vol.32 (4), p.123-126

2008

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Rimmed vacuoles and the added value of SMI-31 staining in diagnosing sporadic inclusion body myositis

Neuromuscular disorders : NMD, 2001-07, Vol.11 (5), p.447-451

2001

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Collagen IV deposits do not prevent regrowing axons from penetrating the lesion site in spinal cord injury

Journal of neuroscience research, 2000-12, Vol.62 (5), p.686-691

2000

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Forbidden subgraphs that imply hamiltonian-connectedness

Journal of graph theory, 2002-06, Vol.40 (2), p.104-119

2002

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Pre- and postsynaptic localization of RC3/neurogranin in the adult rat spinal cord: An immunohistochemical study

Journal of neuroscience research, 2000-03, Vol.59 (6), p.750-759

2000

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Cycles through subsets with large degree sums

Discrete mathematics, 1997-06, Vol.171 (1), p.43-54

1997

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Clinically distinct codon 69 mutations in major myelin protein zero in demyelinating neuropathies

Annals of neurology, 1996-10, Vol.40 (4), p.672-675

1996

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Botulinum toxin-induced myopathy in the rat

Brain (London, England : 1878), 1995-04, Vol.118 (2), p.533-545

1995

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Degree sums for edges and cycle lengths in graphs

Journal of graph theory, 1997-08, Vol.25 (4), p.253-256

1997

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Elimination of superfluous neuromuscular junctions in rat calf muscles recovering from botulinum toxin-induced paralysis

Muscle & nerve, 1994-06, Vol.17 (6), p.623-631

1994

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Calcitonin gene-related peptide-like immunoreactivity, in botulinum toxin-paralysed rat muscles

Neuromuscular disorders : NMD, 1994-09, Vol.4 (5), p.489-496

1994

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Stored platelets release nucleotides as inhibitors of platelet function

Thrombosis and haemostasis, 1992-11, Vol.68 (5), p.595-599

1992

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Basic fibroblast growth factor immunoreactivity in the peripheral motor system of the rat

Acta neuropathologica, 1994-04, Vol.87 (4), p.405-410

1994

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Hamiltonian properties of graphs with large neighborhood unions

Discrete mathematics, 1991-12, Vol.96 (1), p.33-49

1991

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Deficiency of acetylcholine receptors in a case of end-plate acetylcholinesterase deficiency: A histochemical investigation

Muscle & nerve, 1992-01, Vol.15 (1), p.63-72

1992

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Pre- and postsynaptic neuromuscular junction abnormalities in musk myasthenia

Muscle & Nerve, 2010, Vol.42 (2), p.283-288

2010

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A Congenital Myasthenic Disorder with Paucity of Secondary Synaptic Clefts: Deficiency and Altered Distribution of Acetylcholine Receptorsa

Annals of the New York Academy of Sciences, 1987-08, Vol.505 (1), p.346-356

1987

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A Congenital Myasthenic Disorder with Paucity of Secondary Synaptic Clefts: Deficiency and Altered Distribution of Acetylcholine Receptors

Annals of the New York Academy of Sciences, 1987, Vol.505 (1), p.346-356

1987

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Immunofluorescence microscopy of a myopathy: α-Actinin is a major constituent of nemaline rods

Experimental cell research, 1980-01, Vol.127 (2), p.409-420

1980

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Pre‐ and postsynaptic neuromuscular junction abnormalities in musk myasthenia

Increased expression of rapsyn in muscles prevents acetylcholine receptor loss in experimental autoimmune myasthenia gravis

Oxidant treatment causes a dose-dependent phenotype of apoptosis in cultured motoneurons

Schwartz-Jampel syndrome: I. Clinical, electromyographic, and histologic studies

Tubuloreticular Structures in Different Types of Myositis: Implications for Pathogenesis

Oxidant treatment causes a dose‐dependent phenotype of apoptosis in cultured motoneurons

Tubuloreticular Structures in Different Types of Myositis: Implications for Pathogenesis1

Rimmed vacuoles and the added value of SMI-31 staining in diagnosing sporadic inclusion body myositis

Collagen IV deposits do not prevent regrowing axons from penetrating the lesion site in spinal cord injury

Forbidden subgraphs that imply hamiltonian-connectedness

Pre- and postsynaptic localization of RC3/neurogranin in the adult rat spinal cord: An immunohistochemical study

Cycles through subsets with large degree sums

Clinically distinct codon 69 mutations in major myelin protein zero in demyelinating neuropathies

Botulinum toxin-induced myopathy in the rat

Degree sums for edges and cycle lengths in graphs

Elimination of superfluous neuromuscular junctions in rat calf muscles recovering from botulinum toxin-induced paralysis

Calcitonin gene-related peptide-like immunoreactivity, in botulinum toxin-paralysed rat muscles

Stored platelets release nucleotides as inhibitors of platelet function

Basic fibroblast growth factor immunoreactivity in the peripheral motor system of the rat

Hamiltonian properties of graphs with large neighborhood unions

Deficiency of acetylcholine receptors in a case of end-plate acetylcholinesterase deficiency: A histochemical investigation

Pre- and postsynaptic neuromuscular junction abnormalities in musk myasthenia

A Congenital Myasthenic Disorder with Paucity of Secondary Synaptic Clefts: Deficiency and Altered Distribution of Acetylcholine Receptorsa

A Congenital Myasthenic Disorder with Paucity of Secondary Synaptic Clefts: Deficiency and Altered Distribution of Acetylcholine Receptors

Immunofluorescence microscopy of a myopathy: α-Actinin is a major constituent of nemaline rods

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