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Role of the IGF-I receptor in mutagenesis and tumor promotion

Journal of endocrinology, 1997-03, Vol.152 (3), p.339-344

1997

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Stimulation of tumor growth by recombinant human insulin-like growth factor-I (IGF-I) is dependent on the dose and the level of IGF-I receptor expression

Cancer research (Chicago, Ill.), 1998-07, Vol.58 (14), p.3021-3027

1998

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Role of the carboxyl-terminal domains of the insulin and insulin-like growth factor I receptors in receptor function

The Journal of biological chemistry, 1994-05, Vol.269 (19), p.13922-13928

1994

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The yin and the yang of the IGF system: immunological manifestations of GH resistance

European journal of endocrinology, 1997-01, Vol.136 (1), p.33-34

1997

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Insulin-like Growth Factor-I Inhibits the Stress-activated Protein Kinase/c-Jun N-terminal Kinase

The Journal of biological chemistry, 1998-10, Vol.273 (40), p.25961-25966

1998

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Tumorigenic and mitogenic capacities are reduced in transfected fibroblasts expressing mutant insulin-like growth factor (IGF)-I receptors. The role of tyrosine residues 1250, 1251, and 1316 in the carboxy-terminus of the IGF-I receptor

Endocrinology (Philadelphia), 1996-02, Vol.137 (2), p.410-417

1996

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Mitogen-activated protein kinase and phosphatidylinositol 3-kinase pathways are not sufficient for insulin-like growth factor I-induced mitogenesis and tumorigenesis

Endocrinology (Philadelphia), 1997-06, Vol.138 (6), p.2552-2558

1997

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Replacement of Tyrosine 1251 in the Carboxyl Terminus of the Insulin-like Growth Factor-I Receptor Disrupts the Actin Cytoskeleton and Inhibits Proliferation and Anchorage-independent Growth

The Journal of biological chemistry, 1998-07, Vol.273 (29), p.18411-18422

1998

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Tumorigenic and mitogenic capacities are reduced in transfected fibroblasts expressing mutant insulin-like growth factor (IGF)-I receptors. The role of tyrosine residues 1250, 1251, and 1316 in the carboxy-terminus of the IGF-I receptor

Endocrinology (Philadelphia), 1996-02, Vol.137 (2), p.410-417

1996

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Interaction in vitro of the product of the c-Crk-II proto-oncogene with the insulin-like growth factor I receptor

Biochemical journal, 1998-03, Vol.330 ( Pt 2) (2), p.923-932

1998

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In vivo regulation of CrkII and CrkL proto-oncogenes in the uterus by insulin-like growth factor-I. Differential effects on tyrosine phosphorylation and association with paxillin

The Journal of biological chemistry, 1997-10, Vol.272 (44), p.27660-27664

1997

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Differential Phosphorylation of pp120 by Insulin and Insulin-like Growth Factor-1 Receptors: Role for the C-Terminal Domain of the β-Subunit

Biochemistry (Easton), 1997-06, Vol.36 (22), p.6827-6834

1997

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Sphingosine 1-Phosphate Stimulates Tyrosine Phosphorylation of Crk

The Journal of biological chemistry, 1997-06, Vol.272 (26), p.16211-16215

1997

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The insulin-like growth factor-I receptor and apoptosis. Implications for the aging progress

Endocrine, 1997-08, Vol.7 (1), p.103-105

1997

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The Proto-oncogene Crk-II Enhances Apoptosis by a Ras-Dependent, Raf-1/MAP Kinase-Independent Pathway

Biochemical and biophysical research communications, 1997-05, Vol.234 (3), p.616-620

1997

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Mutation of a Conserved Amino Acid Residue (Tryptophan 1173) in the Tyrosine Kinase Domain of the IGF-I Receptor Abolishes Autophosphorylation but Does Not Eliminate Biologic Function (∗)

The Journal of biological chemistry, 1995-02, Vol.270 (6), p.2764-2769

1995

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Current methodology to assess bioequivalence of levothyroxine sodium products is inadequate

The AAPS journal, 2005-03, Vol.7 (1), p.E42-E46

Blakesley, Vicky A

2005

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The role of the insulin-like growth factors in cancer

The Israel Medical Association journal, 1999-09, Vol.1 (1), p.25-30

1999

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Signaling via the insulin-like growth factor-I receptor: Does it differ from insulin receptor signaling?

Cytokine & growth factor reviews, 1996-08, Vol.7 (2), p.153-159

1996

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Characterization of transferable plasmids from Shigella flexneri 2a that confer resistance to trimethoprim, streptomycin, and sulfonamides

Plasmid, 1986-03, Vol.15 (2), p.119-131

1986

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O-12: Roles of the tyrosine residues in the carboxyterminal domain of the IGF-I receptor

Experimental and clinical endocrinology & diabetes, 1996, Vol.104 (S 02), p.19-21

1996

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The proto-oncogene product c-Crk associates with insulin receptor substrate-1 and 4PS. Modulation by insulin growth factor-I (IGF) and enhanced IGF-I signaling

The Journal of biological chemistry, 1996-04, Vol.271 (16), p.9287-9290

1996

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The constitutively active mutant Galpha13 transforms mouse fibroblast cells deficient in insulin-like growth factor-I receptor

The Journal of biological chemistry, 1997-11, Vol.272 (47), p.29438

1997

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The constitutively active mutant G sub( alpha 13) transforms mouse fibroblast cells deficient in insulin-like growth factor-I receptor

The Journal of biological chemistry, 1997-11, Vol.272 (47), p.29438-29441

1997

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PUK9 A RETROSPECTIVE MATCHED COHORT STUDY OF THE BURDEN OF SECONDARY HYPERPARATHYROIDISM (SHPT) IN PREDIALYSIS PATIENTS WITHOUT VITAMIN D RECEPTOR (VDR) ACTIVATOR THERAPY

Value in health, 2007, Vol.10 (3), p.A104-A105

2007

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Role of the IGF-I receptor in mutagenesis and tumor promotion

Stimulation of tumor growth by recombinant human insulin-like growth factor-I (IGF-I) is dependent on the dose and the level of IGF-I receptor expression

Role of the carboxyl-terminal domains of the insulin and insulin-like growth factor I receptors in receptor function

The yin and the yang of the IGF system: immunological manifestations of GH resistance

Insulin-like Growth Factor-I Inhibits the Stress-activated Protein Kinase/c-Jun N-terminal Kinase

Tumorigenic and mitogenic capacities are reduced in transfected fibroblasts expressing mutant insulin-like growth factor (IGF)-I receptors. The role of tyrosine residues 1250, 1251, and 1316 in the carboxy-terminus of the IGF-I receptor

Mitogen-activated protein kinase and phosphatidylinositol 3-kinase pathways are not sufficient for insulin-like growth factor I-induced mitogenesis and tumorigenesis

Replacement of Tyrosine 1251 in the Carboxyl Terminus of the Insulin-like Growth Factor-I Receptor Disrupts the Actin Cytoskeleton and Inhibits Proliferation and Anchorage-independent Growth

Tumorigenic and mitogenic capacities are reduced in transfected fibroblasts expressing mutant insulin-like growth factor (IGF)-I receptors. The role of tyrosine residues 1250, 1251, and 1316 in the carboxy-terminus of the IGF-I receptor

Interaction in vitro of the product of the c-Crk-II proto-oncogene with the insulin-like growth factor I receptor

In vivo regulation of CrkII and CrkL proto-oncogenes in the uterus by insulin-like growth factor-I. Differential effects on tyrosine phosphorylation and association with paxillin

Differential Phosphorylation of pp120 by Insulin and Insulin-like Growth Factor-1 Receptors: Role for the C-Terminal Domain of the β-Subunit

Sphingosine 1-Phosphate Stimulates Tyrosine Phosphorylation of Crk

The insulin-like growth factor-I receptor and apoptosis. Implications for the aging progress

The Proto-oncogene Crk-II Enhances Apoptosis by a Ras-Dependent, Raf-1/MAP Kinase-Independent Pathway

Mutation of a Conserved Amino Acid Residue (Tryptophan 1173) in the Tyrosine Kinase Domain of the IGF-I Receptor Abolishes Autophosphorylation but Does Not Eliminate Biologic Function (∗)

Current methodology to assess bioequivalence of levothyroxine sodium products is inadequate

The role of the insulin-like growth factors in cancer

Signaling via the insulin-like growth factor-I receptor: Does it differ from insulin receptor signaling?

Characterization of transferable plasmids from Shigella flexneri 2a that confer resistance to trimethoprim, streptomycin, and sulfonamides

O-12: Roles of the tyrosine residues in the carboxyterminal domain of the IGF-I receptor

The proto-oncogene product c-Crk associates with insulin receptor substrate-1 and 4PS. Modulation by insulin growth factor-I (IGF) and enhanced IGF-I signaling

The constitutively active mutant Galpha13 transforms mouse fibroblast cells deficient in insulin-like growth factor-I receptor

The constitutively active mutant G sub( alpha 13) transforms mouse fibroblast cells deficient in insulin-like growth factor-I receptor

PUK9 A RETROSPECTIVE MATCHED COHORT STUDY OF THE BURDEN OF SECONDARY HYPERPARATHYROIDISM (SHPT) IN PREDIALYSIS PATIENTS WITHOUT VITAMIN D RECEPTOR (VDR) ACTIVATOR THERAPY

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