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Raising the ‘Good’ Oxidants for Immune Protection

Frontiers in immunology, 2021-06, Vol.12, p.698042-698042

2021

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Defensive Mutualism Rescues NADPH Oxidase Inactivation in Gut Infection

Cell host & microbe, 2016-05, Vol.19 (5), p.651-663

2016

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Pharmacological characterization of the seven human NOX isoforms and their inhibitors

Redox biology, 2019-09, Vol.26, p.101272-101272, Article 101272

2019

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Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair

Redox biology, 2015-08, Vol.5, p.275-289

2015

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Isoform-selective NADPH oxidase inhibitor panel for pharmacological target validation

Free radical biology & medicine, 2020-02, Vol.148, p.60-69

2020

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Hydrogen peroxide production by lactobacilli promotes epithelial restitution during colitis

Redox biology, 2018-06, Vol.16, p.11-20

2018

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Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

Cellular signalling, 2006, Vol.18 (1), p.69-82

2006

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NADPH oxidases and ROS signaling in the gastrointestinal tract

Mucosal immunology, 2018-07, Vol.11 (4), p.1011-1023

2018

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Silencing of DUOX NADPH Oxidases by Promoter Hypermethylation in Lung Cancer

Cancer research (Chicago, Ill.), 2008-02, Vol.68 (4), p.1037-1045

2008

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House dust mite‐treated PAR2 over‐expressor mouse: A novel model of atopic dermatitis

Experimental dermatology, 2019-11, Vol.28 (11), p.1298-1308

2019

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Mucosal Reactive Oxygen Species Decrease Virulence by Disrupting Campylobacter jejuni Phosphotyrosine Signaling

Cell host & microbe, 2012-07, Vol.12 (1), p.47-59

2012

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VEO-IBD NOX1 variant highlights a structural region essential for NOX/DUOX catalytic activity

Redox biology, 2023-11, Vol.67, p.102905-102905, Article 102905

2023

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RhoA determines disease progression by controlling neutrophil motility and restricting hyperresponsiveness

Blood, 2014-06, Vol.123 (23), p.3635-3645

2014

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Structural Insights into Nox4 and Nox2: Motifs Involved in Function and Cellular Localization

Molecular and Cellular Biology, 2010-02, Vol.30 (4), p.961-975

2010

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Mouse Macrophages Completely Lacking Rho Subfamily GTPases (RhoA, RhoB, and RhoC) Have Severe Lamellipodial Retraction Defects, but Robust Chemotactic Navigation and Altered Motility

The Journal of biological chemistry, 2014-10, Vol.289 (44), p.30772-30784

2014

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Case report: Neonatal-onset inflammatory bowel disease due to novel compound heterozygous mutations in DUOX2

Frontiers in genetics, 2023-11, Vol.14, p.1276697

2023

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Ubiquitin C-terminal hydrolase-L1 increases cancer cell invasion by modulating hydrogen peroxide generated via NADPH oxidase 4

Oncotarget, 2015-06, Vol.6 (18), p.16287-16303

2015

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Roles of the Rac1 and Rac3 GTPases in human tumor cell invasion

Oncogene, 2005-11, Vol.24 (53), p.7821-7829

2005

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Heterodimerization controls localization of Duox-DuoxA NADPH oxidases in airway cells

Journal of cell science, 2009-04, Vol.122 (8), p.1238-1247

2009

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Oxidants in Physiological Processes

Handbook of experimental pharmacology, 2021, Vol.264, p.27-47

2021

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Defects in Nicotinamide-adenine Dinucleotide Phosphate Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease

Cellular and molecular gastroenterology and hepatology, 2015-09, Vol.1 (5), p.489-502

2015

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NADPH oxidase 4 is protective and not fibrogenic in intestinal inflammation

Redox biology, 2020-10, Vol.37, p.101752-101752, Article 101752

2020

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Impaired function of the Tie-2 receptor contributes to vascular leakage and lethality in anthrax

Proceedings of the National Academy of Sciences - PNAS, 2012-06, Vol.109 (25), p.10024-10029

2012

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Lung epithelial injury by B. anthracis lethal toxin is caused by MKK-dependent loss of cytoskeletal integrity

PloS one, 2009-03, Vol.4 (3), p.e4755-e4755

2009

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Regulation of Nox1 Activity via Protein Kinase A-mediated Phosphorylation of NoxA1 and 14-3-3 Binding

The Journal of biological chemistry, 2007-11, Vol.282 (48), p.34787-34800

2007

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Raising the ‘Good’ Oxidants for Immune Protection

Defensive Mutualism Rescues NADPH Oxidase Inactivation in Gut Infection

Pharmacological characterization of the seven human NOX isoforms and their inhibitors

Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair

Isoform-selective NADPH oxidase inhibitor panel for pharmacological target validation

Hydrogen peroxide production by lactobacilli promotes epithelial restitution during colitis

Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

NADPH oxidases and ROS signaling in the gastrointestinal tract

Silencing of DUOX NADPH Oxidases by Promoter Hypermethylation in Lung Cancer

House dust mite‐treated PAR2 over‐expressor mouse: A novel model of atopic dermatitis

Mucosal Reactive Oxygen Species Decrease Virulence by Disrupting Campylobacter jejuni Phosphotyrosine Signaling

VEO-IBD NOX1 variant highlights a structural region essential for NOX/DUOX catalytic activity

RhoA determines disease progression by controlling neutrophil motility and restricting hyperresponsiveness

Structural Insights into Nox4 and Nox2: Motifs Involved in Function and Cellular Localization

Mouse Macrophages Completely Lacking Rho Subfamily GTPases (RhoA, RhoB, and RhoC) Have Severe Lamellipodial Retraction Defects, but Robust Chemotactic Navigation and Altered Motility

Case report: Neonatal-onset inflammatory bowel disease due to novel compound heterozygous mutations in DUOX2

Ubiquitin C-terminal hydrolase-L1 increases cancer cell invasion by modulating hydrogen peroxide generated via NADPH oxidase 4

Roles of the Rac1 and Rac3 GTPases in human tumor cell invasion

Heterodimerization controls localization of Duox-DuoxA NADPH oxidases in airway cells

Oxidants in Physiological Processes

Defects in Nicotinamide-adenine Dinucleotide Phosphate Oxidase Genes NOX1 and DUOX2 in Very Early Onset Inflammatory Bowel Disease

NADPH oxidase 4 is protective and not fibrogenic in intestinal inflammation

Impaired function of the Tie-2 receptor contributes to vascular leakage and lethality in anthrax

Lung epithelial injury by B. anthracis lethal toxin is caused by MKK-dependent loss of cytoskeletal integrity

Regulation of Nox1 Activity via Protein Kinase A-mediated Phosphorylation of NoxA1 and 14-3-3 Binding

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