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Hydrogen sulfide: an emerging gasotransmitter involved in the survival of pancreatic islets during diabetes therapy

Medical gas research, 2025-03, Vol.15 (1), p.136-138

Karunakaran, Udayakumar

2025

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A systematic review of oxidative stress and safety of antioxidants in diabetes: focus on islets and their defense

Diabetes and Metabolism Journal, 2013, 37(2), 136, pp.106-112

2013

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Pioglitazone-induced AMPK-Glutaminase-1 prevents high glucose-induced pancreatic β-cell dysfunction by glutathione antioxidant system

Redox biology, 2021-09, Vol.45, p.102029-102029, Article 102029

2021

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Ferroptosis Signaling in Pancreatic β-Cells: Novel Insights & Therapeutic Targeting

International journal of molecular sciences, 2022-11, Vol.23 (22), p.13679

2022

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Physiological effect and therapeutic application of alpha lipoic acid

Current medicinal chemistry, 2014-01, Vol.21 (32), p.3636-3645

2014

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Mitochondrial ALDH2 improves ß‐cell survival and function against doxorubicin‐induced apoptosis by targeting CK2 signaling

Journal of diabetes investigation, 2024-06, Vol.15 (6), p.684-692

2024

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Mitochondrial aldehyde dehydrogenase-2 coordinates the hydrogen sulfide - AMPK axis to attenuate high glucose-induced pancreatic β-cell dysfunction by glutathione antioxidant system

Redox biology, 2024-02, Vol.69, p.102994, Article 102994

2024

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Metformin prevents glucotoxicity by alleviating oxidative and ER stress–induced CD36 expression in pancreatic beta cells

Journal of diabetes and its complications, 2017-01, Vol.31 (1), p.21-30

2017

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CD36 Signal Transduction in Metabolic Diseases: Novel Insights and Therapeutic Targeting

Cells (Basel, Switzerland), 2021-07, Vol.10 (7), p.1833

2021

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Myricetin Protects Against High Glucose-Induced β-Cell Apoptosis by Attenuating Endoplasmic Reticulum Stress via Inactivation of Cyclin-Dependent Kinase 5

Diabetes and Metabolism Journal, 2019, 43(2), 172, pp.192-205

2019

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Rac1-NADPH oxidase signaling promotes CD36 activation under glucotoxic conditions in pancreatic beta cells

Redox biology, 2017-04, Vol.11, p.126-134

2017

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CD36 initiated signaling mediates ceramide-induced TXNIP expression in pancreatic beta-cells

Biochimica et biophysica acta. Molecular basis of disease, 2015-11, Vol.1852 (11), p.2414-2422

2015

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The Role of CD36 in Type 2 Diabetes Mellitus: β-Cell Dysfunction and Beyond

Diabetes and Metabolism Journal, 2020, 44(2), 178, pp.222-233

2020

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NADPH Oxidase (NOX) Targeting in Diabetes: A Special Emphasis on Pancreatic β-Cell Dysfunction

Cells (Basel, Switzerland), 2021-06, Vol.10 (7), p.1573

2021

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High glucose-induced PRDX3 acetylation contributes to glucotoxicity in pancreatic β-cells: Prevention by Teneligliptin

Free radical biology & medicine, 2020-11, Vol.160, p.618-629

2020

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c-Abl tyrosine kinase inhibition attenuate oxidative stress-induced pancreatic β-Cell dysfunction via glutathione antioxidant system

Translational research : the journal of laboratory and clinical medicine, 2022-11, Vol.249, p.74-87

2022

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Perfluorooctane sulfonate-induced oxidative stress contributes to pancreatic β-cell apoptosis by inhibiting cyclic adenosine monophosphate pathway: Prevention by pentoxifylline

Environmental pollution (1987), 2023-03, Vol.320, p.120959, Article 120959

2023

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Myricetin prevents thapsigargin-induced CDK5-P66Shc signalosome mediated pancreatic β-cell dysfunction

Free radical biology & medicine, 2019-09, Vol.141, p.59-66

2019

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CD36 dependent redoxosomes promotes ceramide-mediated pancreatic β-cell failure via p66Shc activation

Free radical biology & medicine, 2019-04, Vol.134, p.505-515

2019

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The Role of CD36 in Type 2 Diabetes Mellitus: β-Cell Dysfunction and Beyond

Diabetes & metabolism journal, 2020-04, Vol.44 (2), p.222

2020

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Non-receptor tyrosine kinase inhibitors enhances β-cell survival by suppressing the PKCδ signal transduction pathway in streptozotocin – induced β-cell apoptosis

Cellular signalling, 2015-06, Vol.27 (6), p.1066-1074

2015

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Reviews : A Systematic Review of Oxidative Stress and Safety of Antioxidants in Diabetes: Focus on Islets and Their Defense

Diabetes & metabolism journal, 2013-04, Vol.37 (2), p.106

2013

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Myricetin Protects Against High Glucose-Induced β-Cell Apoptosis by Attenuating Endoplasmic Reticulum Stress via Inactivation of Cyclin-Dependent Kinase 5

Diabetes & metabolism journal, 2019-04, Vol.43 (2), p.192

2019

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Guards and Culprits in the Endoplasmic Reticulum: Glucolipotoxicity and β-Cell Failure in Type II Diabetes

Journal of diabetes research, 2011, Vol.2012 (2012), p.1-9

2011

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Guards and Culprits in the Endoplasmic Reticulum : Glucolipotoxicity and β-Cell Failure in Type II Diabetes

Experimental diabetes research, 2012-01, Vol.2012 (2012), p.1-9

2012

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Hydrogen sulfide: an emerging gasotransmitter involved in the survival of pancreatic islets during diabetes therapy

A systematic review of oxidative stress and safety of antioxidants in diabetes: focus on islets and their defense

Pioglitazone-induced AMPK-Glutaminase-1 prevents high glucose-induced pancreatic β-cell dysfunction by glutathione antioxidant system

Ferroptosis Signaling in Pancreatic β-Cells: Novel Insights & Therapeutic Targeting

Physiological effect and therapeutic application of alpha lipoic acid

Mitochondrial ALDH2 improves ß‐cell survival and function against doxorubicin‐induced apoptosis by targeting CK2 signaling

Mitochondrial aldehyde dehydrogenase-2 coordinates the hydrogen sulfide - AMPK axis to attenuate high glucose-induced pancreatic β-cell dysfunction by glutathione antioxidant system

Metformin prevents glucotoxicity by alleviating oxidative and ER stress–induced CD36 expression in pancreatic beta cells

CD36 Signal Transduction in Metabolic Diseases: Novel Insights and Therapeutic Targeting

Myricetin Protects Against High Glucose-Induced β-Cell Apoptosis by Attenuating Endoplasmic Reticulum Stress via Inactivation of Cyclin-Dependent Kinase 5

Rac1-NADPH oxidase signaling promotes CD36 activation under glucotoxic conditions in pancreatic beta cells

CD36 initiated signaling mediates ceramide-induced TXNIP expression in pancreatic beta-cells

The Role of CD36 in Type 2 Diabetes Mellitus: β-Cell Dysfunction and Beyond

NADPH Oxidase (NOX) Targeting in Diabetes: A Special Emphasis on Pancreatic β-Cell Dysfunction

High glucose-induced PRDX3 acetylation contributes to glucotoxicity in pancreatic β-cells: Prevention by Teneligliptin

c-Abl tyrosine kinase inhibition attenuate oxidative stress-induced pancreatic β-Cell dysfunction via glutathione antioxidant system

Perfluorooctane sulfonate-induced oxidative stress contributes to pancreatic β-cell apoptosis by inhibiting cyclic adenosine monophosphate pathway: Prevention by pentoxifylline

Myricetin prevents thapsigargin-induced CDK5-P66Shc signalosome mediated pancreatic β-cell dysfunction

CD36 dependent redoxosomes promotes ceramide-mediated pancreatic β-cell failure via p66Shc activation

The Role of CD36 in Type 2 Diabetes Mellitus: β-Cell Dysfunction and Beyond

Non-receptor tyrosine kinase inhibitors enhances β-cell survival by suppressing the PKCδ signal transduction pathway in streptozotocin – induced β-cell apoptosis

Reviews : A Systematic Review of Oxidative Stress and Safety of Antioxidants in Diabetes: Focus on Islets and Their Defense

Myricetin Protects Against High Glucose-Induced β-Cell Apoptosis by Attenuating Endoplasmic Reticulum Stress via Inactivation of Cyclin-Dependent Kinase 5

Guards and Culprits in the Endoplasmic Reticulum: Glucolipotoxicity and β-Cell Failure in Type II Diabetes

Guards and Culprits in the Endoplasmic Reticulum : Glucolipotoxicity and β-Cell Failure in Type II Diabetes

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