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Modulation of Osteoclast Differentiation and Function by the New Members of the Tumor Necrosis Factor Receptor and Ligand Families

Endocrine reviews, 1999-06, Vol.20 (3), p.345-357

1999

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Dental Pulp and Periodontal Ligament Cells Support Osteoclastic Differentiation

Journal of dental research, 2009-07, Vol.88 (7), p.609-614

2009

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Osteoclast Differentiation Factor is a Ligand for Osteoprotegerin/Osteoclastogenesis-Inhibitory Factor and is Identical to TRANCE/RANKL

Proceedings of the National Academy of Sciences - PNAS, 1998-03, Vol.95 (7), p.3597-3602

1998

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Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering

Oral diseases, 2015-07, Vol.21 (5), p.583-592

2015

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Transforming Growth Factor β Affects Osteoclast Differentiation via Direct and Indirect Actions

Journal of bone and mineral research, 2001-10, Vol.16 (10), p.1787-1794

2001

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Coordination of microtubules and the actin cytoskeleton is important in osteoclast function, but calcitonin disrupts sealing zones without affecting microtubule networks

Bone (New York, N.Y.), 2006-10, Vol.39 (4), p.684-693

2006

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Osteoblasts/stromal cells stimulate osteoclast activation through expression of osteoclast differentiation factor/RANKL but not macrophage colony-stimulating factor

Bone (New York, N.Y.), 1999-11, Vol.25 (5), p.517-523

1999

Volltextzugriff (PDF)

Non-canonical Wnt signals regulate cytoskeletal remodeling in osteoclasts

Cellular and molecular life sciences : CMLS, 2018-10, Vol.75 (20), p.3683-3692

2018

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Modulation of osteoclast differentiation by local factors

Bone (New York, N.Y.), 1995-08, Vol.17 (2 Suppl), p.87S-S91

1995

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A novel molecular mechanism modulating osteoclast differentiation and function

Bone (New York, N.Y.), 1999-07, Vol.25 (1), p.109-113

1999

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Soluble Interleukin-6 Receptor Triggers Osteoclast Formation by Interleukin 6

Proceedings of the National Academy of Sciences - PNAS, 1993-12, Vol.90 (24), p.11924-11928

1993

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RANKL/OPG ratio regulates odontoclastogenesis in damaged dental pulp

Scientific reports, 2021-02, Vol.11 (1), p.4575-4575, Article 4575

2021

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Macrophage colony-stimulating factor is indispensable for both proliferation and differentiation of osteoclast progenitors

The Journal of clinical investigation, 1993, Vol.91 (1), p.257-263

1993

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A possible mechanism of the specific action of bisphosphonates on osteoclasts: Tiludronate preferentially affects polarized osteoclasts having ruffled borders

Bone (New York, N.Y.), 1995-08, Vol.17 (2), p.137-144

1995

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The Role of the Plasminogen System in Bone Resorption In Vitro

Journal of bone and mineral research, 1999-06, Vol.14 (6), p.946-952

1999

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Action of RANKL and OPG for osteoclastogenesis

Critical reviews in eukaryotic gene expression, 2009, Vol.19 (1), p.61

2009

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Destruxins, cyclodepsipeptides, block the formation of actin rings and prominent clear zones and ruffled borders in osteoclasts

Bone (New York, N.Y.), 2003-09, Vol.33 (3), p.443-455

2003

Volltextzugriff (PDF)

Origin of Osteoclasts: Mature Monocytes and Macrophages are Capable of Differentiating Into Osteoclasts Under a Suitable Microenvironment Prepared by Bone Marrow-Derived Stromal Cells

Proceedings of the National Academy of Sciences - PNAS, 1990-09, Vol.87 (18), p.7260-7264

1990

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Investigation on Magnetic Fields From Field-Generating Layer in MAMR

IEEE transactions on magnetics, 2008-11, Vol.44 (11), p.3408-3411

2008

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Evaluation of Pharmaceuticals With a Novel 50‐Hour Animal Model of Bone Loss

Journal of bone and mineral research, 2009-07, Vol.24 (7), p.1194-1205

2009

Volltextzugriff (PDF)

Daily administration of eldecalcitol (ED-71), an active vitamin D analog, increases bone mineral density by suppressing RANKL expression in mouse trabecular bone

Journal of bone and mineral research, 2012-02, Vol.27 (2), p.461-473

2012

Volltextzugriff (PDF)

Lineage‐committed osteoclast precursors circulate in blood and settle down into bone

Journal of bone and mineral research, 2011-12, Vol.26 (12), p.2978-2990

2011

Volltextzugriff (PDF)

A New Member of Tumor Necrosis Factor Ligand Family, ODF/OPGL/TRANCE/RANKL, Regulates Osteoclast Differentiation and Function

Biochemical and biophysical research communications, 1999-03, Vol.256 (3), p.449-455

1999

Volltextzugriff (PDF)

Ror2 signaling enhances osteoclast formation in physiological and pathological conditions

Bone (New York, N.Y.), 2009-05, Vol.44, p.S50-S50

2009

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A critical role for interleukin-6 family-mediated Stat3 activation in osteoblast differentiation and bone formation

Bone (New York, N.Y.), 2006-09, Vol.39 (3), p.505-512

2006

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Modulation of Osteoclast Differentiation and Function by the New Members of the Tumor Necrosis Factor Receptor and Ligand Families

Dental Pulp and Periodontal Ligament Cells Support Osteoclastic Differentiation

Osteoclast Differentiation Factor is a Ligand for Osteoprotegerin/Osteoclastogenesis-Inhibitory Factor and is Identical to TRANCE/RANKL

Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering

Transforming Growth Factor β Affects Osteoclast Differentiation via Direct and Indirect Actions

Coordination of microtubules and the actin cytoskeleton is important in osteoclast function, but calcitonin disrupts sealing zones without affecting microtubule networks

Osteoblasts/stromal cells stimulate osteoclast activation through expression of osteoclast differentiation factor/RANKL but not macrophage colony-stimulating factor

Non-canonical Wnt signals regulate cytoskeletal remodeling in osteoclasts

Modulation of osteoclast differentiation by local factors

A novel molecular mechanism modulating osteoclast differentiation and function

Soluble Interleukin-6 Receptor Triggers Osteoclast Formation by Interleukin 6

RANKL/OPG ratio regulates odontoclastogenesis in damaged dental pulp

Macrophage colony-stimulating factor is indispensable for both proliferation and differentiation of osteoclast progenitors

A possible mechanism of the specific action of bisphosphonates on osteoclasts: Tiludronate preferentially affects polarized osteoclasts having ruffled borders

The Role of the Plasminogen System in Bone Resorption In Vitro

Action of RANKL and OPG for osteoclastogenesis

Destruxins, cyclodepsipeptides, block the formation of actin rings and prominent clear zones and ruffled borders in osteoclasts

Origin of Osteoclasts: Mature Monocytes and Macrophages are Capable of Differentiating Into Osteoclasts Under a Suitable Microenvironment Prepared by Bone Marrow-Derived Stromal Cells

Investigation on Magnetic Fields From Field-Generating Layer in MAMR

Evaluation of Pharmaceuticals With a Novel 50‐Hour Animal Model of Bone Loss

Daily administration of eldecalcitol (ED-71), an active vitamin D analog, increases bone mineral density by suppressing RANKL expression in mouse trabecular bone

Lineage‐committed osteoclast precursors circulate in blood and settle down into bone

A New Member of Tumor Necrosis Factor Ligand Family, ODF/OPGL/TRANCE/RANKL, Regulates Osteoclast Differentiation and Function

Ror2 signaling enhances osteoclast formation in physiological and pathological conditions

A critical role for interleukin-6 family-mediated Stat3 activation in osteoblast differentiation and bone formation

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