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Passive immunization reduces behavioral and neuropathological deficits in an alpha-synuclein transgenic model of Lewy body disease

PloS one, 2011-04, Vol.6 (4), p.e19338-e19338

2011

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Widespread microRNA dysregulation in multiple system atrophy - disease-related alteration in miR-96

The European journal of neuroscience, 2014-03, Vol.39 (6), p.1026-1041

2014

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Fluoxetine rescues impaired hippocampal neurogenesis in a transgenic A53T synuclein mouse model

The European journal of neuroscience, 2012-01, Vol.35 (1), p.10-19

2012

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Autophagy in Dementias

Brain pathology (Zurich, Switzerland), 2012-01, Vol.22 (1), p.99-109

2012

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Cerebrolysin modulates pronerve growth factor/nerve growth factor ratio and ameliorates the cholinergic deficit in a transgenic model of Alzheimer's disease

Journal of neuroscience research, 2013-02, Vol.91 (2), p.167-177

2013

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A novel triple repeat mutant tau transgenic model that mimics aspects of pick's disease and fronto-temporal tauopathies

PloS one, 2015-03, Vol.10 (3), p.e0121570-e0121570

2015

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Differential effects of UCHL1 modulation on alpha-synuclein in PD-like models of alpha-synucleinopathy

PloS one, 2012-04, Vol.7 (4), p.e34713-e34713

2012

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Neuro-peptide treatment with Cerebrolysin improves the survival of neural stem cell grafts in an APP transgenic model of Alzheimer disease

Stem cell research, 2015-07, Vol.15 (1), p.54-67

2015

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Alterations in mGluR5 expression and signaling in Lewy body disease and in transgenic models of alpha-synucleinopathy--implications for excitotoxicity

PloS one, 2010-11, Vol.5 (11), p.e14020-e14020

2010

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Mitochondrial inhibitor 3-nitroproprionic acid enhances oxidative modification of alpha-synuclein in a transgenic mouse model of multiple system atrophy

Journal of neuroscience research, 2009-09, Vol.87 (12), p.2728-2739

2009

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Alzheimer's disease: recent advances and future perspectives

Journal of Alzheimer's disease, 2013-01, Vol.33 Suppl 1, p.S185

2013

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Multiple system atrophy: a clinical and neuropathological perspective

Trends in neurosciences (Regular ed.), 2011-11, Vol.34 (11), p.581-590

2011

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Cerebrolysin™ efficacy in a transgenic model of tauopathy: role in regulation of mitochondrial structure

BMC neuroscience, 2014-07, Vol.15 (1), p.90-90, Article 90

2014

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Antidepressants reduce neuroinflammatory responses and astroglial alpha-synuclein accumulation in a transgenic mouse model of multiple system atrophy

Glia, 2014-02, Vol.62 (2), p.317-337

2014

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Recent advances in the development of immunotherapies for tauopathies

Experimental neurology, 2011-08, Vol.230 (2), p.157-161

2011

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Corrigendum to “Fluoxetine ameliorates behavioral and neuropathological deficits in a transgenic model mouse of α-synucleinopathy” Exp Neurol. 2012 April; 234(2): 405–416

Experimental neurology, 2024-06, p.114873, Article 114873

2024

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Reducing C-terminal-truncated alpha-synuclein by immunotherapy attenuates neurodegeneration and propagation in Parkinson's disease-like models

The Journal of neuroscience, 2014-07, Vol.34 (28), p.9441-9454

2014

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Neurodegeneration in a transgenic mouse model of multiple system atrophy is associated with altered expression of oligodendroglial-derived neurotrophic factors

The Journal of neuroscience, 2010-05, Vol.30 (18), p.6236-6246

2010

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A comparison of the neuronal dysfunction caused by Drosophila tau and human tau in a Drosophila model of tauopathies

Invertebrate neuroscience, 2007-09, Vol.7 (3), p.165-171

2007

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Accumulation of oligomer-prone α-synuclein exacerbates synaptic and neuronal degeneration in vivo

Brain (London, England : 1878), 2014-05, Vol.137 (5), p.1496-1513

2014

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Fluoxetine ameliorates behavioral and neuropathological deficits in a transgenic model mouse of α-synucleinopathy

Experimental neurology, 2012-04, Vol.234 (2), p.405-416

2012

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Axonopathy in an α -Synuclein Transgenic Model of Lewy Body Disease Is Associated with Extensive Accumulation of C-Terminal–Truncated α -Synuclein

The American journal of pathology, 2013-03, Vol.182 (3), p.940-953

2013

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Progressive accumulation of amyloid-β oligomers in Alzheimer's disease and in amyloid precursor protein transgenic mice is accompanied by selective alterations in synaptic scaffold proteins

The FEBS journal, 2010-07, Vol.277 (14), p.3051-3067

2010

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Aβ exacerbates the neuronal dysfunction caused by human tau expression in a Drosophila model of Alzheimer's disease

Experimental neurology, 2010-06, Vol.223 (2), p.401-409

2010

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Lovastatin ameliorates α-synuclein accumulation and oxidation in transgenic mouse models of α-synucleinopathies

Experimental neurology, 2010-02, Vol.221 (2), p.267-274

2010

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Passive immunization reduces behavioral and neuropathological deficits in an alpha-synuclein transgenic model of Lewy body disease

Widespread microRNA dysregulation in multiple system atrophy - disease-related alteration in miR-96

Fluoxetine rescues impaired hippocampal neurogenesis in a transgenic A53T synuclein mouse model

Autophagy in Dementias

Cerebrolysin modulates pronerve growth factor/nerve growth factor ratio and ameliorates the cholinergic deficit in a transgenic model of Alzheimer's disease

A novel triple repeat mutant tau transgenic model that mimics aspects of pick's disease and fronto-temporal tauopathies

Differential effects of UCHL1 modulation on alpha-synuclein in PD-like models of alpha-synucleinopathy

Neuro-peptide treatment with Cerebrolysin improves the survival of neural stem cell grafts in an APP transgenic model of Alzheimer disease

Alterations in mGluR5 expression and signaling in Lewy body disease and in transgenic models of alpha-synucleinopathy--implications for excitotoxicity

Mitochondrial inhibitor 3-nitroproprionic acid enhances oxidative modification of alpha-synuclein in a transgenic mouse model of multiple system atrophy

Alzheimer's disease: recent advances and future perspectives

Multiple system atrophy: a clinical and neuropathological perspective

Cerebrolysin™ efficacy in a transgenic model of tauopathy: role in regulation of mitochondrial structure

Antidepressants reduce neuroinflammatory responses and astroglial alpha-synuclein accumulation in a transgenic mouse model of multiple system atrophy

Recent advances in the development of immunotherapies for tauopathies

Corrigendum to “Fluoxetine ameliorates behavioral and neuropathological deficits in a transgenic model mouse of α-synucleinopathy” Exp Neurol. 2012 April; 234(2): 405–416

Reducing C-terminal-truncated alpha-synuclein by immunotherapy attenuates neurodegeneration and propagation in Parkinson's disease-like models

Neurodegeneration in a transgenic mouse model of multiple system atrophy is associated with altered expression of oligodendroglial-derived neurotrophic factors

A comparison of the neuronal dysfunction caused by Drosophila tau and human tau in a Drosophila model of tauopathies

Accumulation of oligomer-prone α-synuclein exacerbates synaptic and neuronal degeneration in vivo

Fluoxetine ameliorates behavioral and neuropathological deficits in a transgenic model mouse of α-synucleinopathy

Axonopathy in an α -Synuclein Transgenic Model of Lewy Body Disease Is Associated with Extensive Accumulation of C-Terminal–Truncated α -Synuclein

Progressive accumulation of amyloid-β oligomers in Alzheimer's disease and in amyloid precursor protein transgenic mice is accompanied by selective alterations in synaptic scaffold proteins

Aβ exacerbates the neuronal dysfunction caused by human tau expression in a Drosophila model of Alzheimer's disease

Lovastatin ameliorates α-synuclein accumulation and oxidation in transgenic mouse models of α-synucleinopathies

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