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Deficiency of the ribosome biogenesis gene Sbds in hematopoietic stem and progenitor cells causes neutropenia in mice by attenuating lineage progression in myelocytes

Haematologica (Roma), 2015-10, Vol.100 (10), p.1285-1293

2015

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MicroRNA-155 induces AML in combination with the loss of C/EBPA in mice

Leukemia, 2016-11, Vol.30 (11), p.2238-2241

2016

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Mutations in the Gene for the Granulocyte Colony-Stimulating–Factor Receptor in Patients with Acute Myeloid Leukemia Preceded by Severe Congenital Neutropenia

The New England journal of medicine, 1995-08, Vol.333 (8), p.487-493

1995

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Regulation of granulopoiesis by transcription factors and cytokine signals

Leukemia, 2000-06, Vol.14 (6), p.973-990

2000

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Novel role of WD40 and SOCS box protein-2 in steady-state distribution of granulocyte colony-stimulating factor receptor and G-CSF-controlled proliferation and differentiation signaling

Oncogene, 2007-03, Vol.26 (14), p.1985-1994

2007

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Hematopoietic Growth Factors and Their Receptors in Acute Leukemia

Blood, 1993-01, Vol.81 (2), p.281-292

1993

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Hematopoietic stem cell transplantation in severe congenital neutropenia

Pediatric blood & cancer, 2011-03, Vol.56 (3), p.444-451

2011

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Defective internalization and sustained activation of truncated granulocyte colony-stimulating factor receptor found in severe congenital neutropenia/acute myeloid leukemia

Blood, 1999-01, Vol.93 (2), p.447-458

1999

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Multiple pathways contribute to the hyperproliferative responses from truncated granulocyte colony-stimulating factor receptors

Leukemia, 2006-12, Vol.20 (12), p.2111-2118

2006

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The Carboxyl Terminus of the Granulocyte Colony- Stimulating Factor Receptor, Truncated in Patients with Severe Congenital Neutropenia/Acute Myeloid Leukemia, Is Required for SH2-Containing Phosphatase-1 Suppression of Stat Activation

The Journal of immunology (1950), 2001-12, Vol.167 (11), p.6447-6452

2001

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Distinct cytoplasmic regions of the human granulocyte colony-stimulating factor receptor involved in induction of proliferation and maturation

Molecular and Cellular Biology, 1993-12, Vol.13 (12), p.7774-7781

1993

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Mutations in the granulocyte colony-stimulating factor receptor gene in patients with severe congenital neutropenia

Leukemia, 1997, Vol.11 (1), p.120-125

1997

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Perturbed granulopoiesis in mice with a targeted mutation in the granulocyte colony-stimulating factor receptor gene associated with severe chronic neutropenia

Blood, 1998-07, Vol.92 (1), p.32-39

1998

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C/EBPα and G-CSF receptor signals cooperate to induce the myeloperoxidase and neutrophil elastase genes

Leukemia, 2001-05, Vol.15 (5), p.779-786

2001

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Acute lymphoblastic leukemia in a patient with congenital neutropenia without G-CSF-R and ELA2 mutations

Leukemia, 2005-09, Vol.19 (9), p.1710-1711

2005

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Identification of a Nonsense Mutation in the Granulocyte-Colony-Stimulating Factor Receptor in Severe Congenital Neutropenia

Proceedings of the National Academy of Sciences - PNAS, 1994-05, Vol.91 (10), p.4480-4484

1994

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Severe congenital neutropenia and chronic neutrophilic leukemia: an intriguing molecular connection unveiled by oncogenic mutations in CSF3R

Haematologica (Roma), 2013-10, Vol.98 (10), p.1490-1492

2013

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Multiple Signals Mediate Proliferation, Differentiation, and Survival from the Granulocyte Colony-stimulating Factor Receptor in Myeloid 32D Cells

The Journal of biological chemistry, 1999-05, Vol.274 (21), p.14956-14962

1999

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Granulocyte colony-stimulating factor and its receptor in normal myeloid cell development, leukemia and related blood cell disorders

Frontiers in bioscience, 2007-01, Vol.12 (1), p.800

2007

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The SH2 domain-containing protein tyrosine phosphatase SHP-1 is induced by granulocyte colony-stimulating factor (G-CSF) and modulates signaling from the G-CSF receptor

Leukemia, 2000-07, Vol.14 (7), p.1284-1291

2000

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Autonomous Proliferation of Leukemic Cells in Vitro as a Determinant of Prognosis in Adult Acute Myeloid Leukemia

The New England journal of medicine, 1993-03, Vol.328 (9), p.614-619

1993

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Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene

The Journal of experimental medicine, 1999-02, Vol.189 (4), p.683-692

1999

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Large-Scale Identification of Disease Genes Involved in Acute Myeloid Leukemia

Journal of Virology, 2004-02, Vol.78 (4), p.1971-1980

2004

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Distinct activities of suppressor of cytokine signaling (SOCS) proteins and involvement of the SOCS box in controlling G-CSF signaling

Journal of leukocyte biology, 2004-07, Vol.76 (1), p.237-244

2004

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A Point Mutation in the Granulocyte Colony-Stimulating Factor Receptor (G-CSF-R) Gene in a Case of Acute Myeloid Leukemia Results in the Overexpression of a Novel G-CSF-R Isoform

Blood, 1995-02, Vol.85 (4), p.902-911

1995

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Deficiency of the ribosome biogenesis gene Sbds in hematopoietic stem and progenitor cells causes neutropenia in mice by attenuating lineage progression in myelocytes

MicroRNA-155 induces AML in combination with the loss of C/EBPA in mice

Mutations in the Gene for the Granulocyte Colony-Stimulating–Factor Receptor in Patients with Acute Myeloid Leukemia Preceded by Severe Congenital Neutropenia

Regulation of granulopoiesis by transcription factors and cytokine signals

Novel role of WD40 and SOCS box protein-2 in steady-state distribution of granulocyte colony-stimulating factor receptor and G-CSF-controlled proliferation and differentiation signaling

Hematopoietic Growth Factors and Their Receptors in Acute Leukemia

Hematopoietic stem cell transplantation in severe congenital neutropenia

Defective internalization and sustained activation of truncated granulocyte colony-stimulating factor receptor found in severe congenital neutropenia/acute myeloid leukemia

Multiple pathways contribute to the hyperproliferative responses from truncated granulocyte colony-stimulating factor receptors

The Carboxyl Terminus of the Granulocyte Colony- Stimulating Factor Receptor, Truncated in Patients with Severe Congenital Neutropenia/Acute Myeloid Leukemia, Is Required for SH2-Containing Phosphatase-1 Suppression of Stat Activation

Distinct cytoplasmic regions of the human granulocyte colony-stimulating factor receptor involved in induction of proliferation and maturation

Mutations in the granulocyte colony-stimulating factor receptor gene in patients with severe congenital neutropenia

Perturbed granulopoiesis in mice with a targeted mutation in the granulocyte colony-stimulating factor receptor gene associated with severe chronic neutropenia

C/EBPα and G-CSF receptor signals cooperate to induce the myeloperoxidase and neutrophil elastase genes

Acute lymphoblastic leukemia in a patient with congenital neutropenia without G-CSF-R and ELA2 mutations

Identification of a Nonsense Mutation in the Granulocyte-Colony-Stimulating Factor Receptor in Severe Congenital Neutropenia

Severe congenital neutropenia and chronic neutrophilic leukemia: an intriguing molecular connection unveiled by oncogenic mutations in CSF3R

Multiple Signals Mediate Proliferation, Differentiation, and Survival from the Granulocyte Colony-stimulating Factor Receptor in Myeloid 32D Cells

Granulocyte colony-stimulating factor and its receptor in normal myeloid cell development, leukemia and related blood cell disorders

The SH2 domain-containing protein tyrosine phosphatase SHP-1 is induced by granulocyte colony-stimulating factor (G-CSF) and modulates signaling from the G-CSF receptor

Autonomous Proliferation of Leukemic Cells in Vitro as a Determinant of Prognosis in Adult Acute Myeloid Leukemia

Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene

Large-Scale Identification of Disease Genes Involved in Acute Myeloid Leukemia

Distinct activities of suppressor of cytokine signaling (SOCS) proteins and involvement of the SOCS box in controlling G-CSF signaling

A Point Mutation in the Granulocyte Colony-Stimulating Factor Receptor (G-CSF-R) Gene in a Case of Acute Myeloid Leukemia Results in the Overexpression of a Novel G-CSF-R Isoform

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