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Trisomy of human chromosome 21 enhances amyloid-β deposition independently of an extra copy of APP

Brain (London, England : 1878), 2018-08, Vol.141 (8), p.2457-2474

2018

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Genetic dissection of down syndrome-associated alterations in APP/amyloid-β biology using mouse models

Scientific reports, 2021-03, Vol.11 (1), p.5736-5736, Article 5736

2021

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Dissecting Alzheimer disease in Down syndrome using mouse models

Frontiers in behavioral neuroscience, 2015-10, Vol.9, p.268-268

2015

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Publisher Correction: Genetic dissection of down syndrome‑associated alterations in APP/amyloid‑β biology using mouse models

Scientific reports, 2021-07, Vol.11 (1), p.14966-14966, Article 14966

2021

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Gene expression dysregulation domains are not a specific feature of Down syndrome

Nature communications, 2019-06, Vol.10 (1), p.2489-2489, Article 2489

2019

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Comprehensive phenotypic analysis of the Dp1Tyb mouse strain reveals a broad range of Down syndrome-related phenotypes

Disease models & mechanisms, 2021-10, Vol.14 (10)

2021

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The integration site of the APP transgene in the J20 mouse model of Alzheimer's disease

Wellcome open research, 2017, Vol.2, p.84

2017

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The integration site of the APP transgene in the J20 mouse model of Alzheimer’s disease

Wellcome open research, 2018-10, Vol.2, p.84-84

2018

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Mouse models of aneuploidy to understand chromosome disorders

Mammalian genome, 2022-03, Vol.33 (1), p.157-168

2022

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Section on Cognitive Neuropsychology, Laboratory of Brain and Cognition, National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland 20892

The Journal of neuroscience, 2022-08, Vol.42 (33), p.6453

2022

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Modelling of the development and response to amyloid‐β accumulation in the context of trisomy21 in the rodent brain

Alzheimer's & dementia, 2023-12, Vol.19 (S12), p.n/a

2023

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Genetic Mapping of APP and Amyloid-β Biology Modulation by Trisomy 21

The Journal of neuroscience, 2022-08, Vol.42 (33), p.6453-6468

2022

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Genetic mapping of APP and amyloid‐β biology modulation by trisomy 21

Alzheimer's & dementia, 2023-06, Vol.19 (S1), p.n/a

2023

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Alzheimer's disease in Down syndrome : mapping modifiers of Alzheimer's pathology in novel mouse crosses

Tosh, Justin

2019

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Downregulated Wnt/β-catenin signalling in the Down syndrome hippocampus

Scientific reports, 2019-05, Vol.9 (1), p.7322, Article 7322

2019

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Using mouse models to understand Alzheimer's disease mechanisms in the context of trisomy of chromosome 21

Progress in brain research, 2020, Vol.251, p.181-208

2020

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TRISOMY 21 CAUSES A DEFICIT IN LYSOSOMAL CATHEPSINS AND ALTERS APP/Aβ PROCESSING, INDEPENDENTLY OF AN EXTRA COPY OF APP

Alzheimer's & dementia, 2017-07, Vol.13 (7), p.P661-P662

2017

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Structure activity relationship of novel antiviral nucleosides against Enterovirus A71

Bioorganic & medicinal chemistry letters, 2020-12, Vol.30 (23), p.127599-127599, Article 127599

2020

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Extremely Elevated Erythrocyte Sedimentation Rates: Associations With Patients' Diagnoses, Demographic Characteristics, and Comorbidities

Mayo Clinic proceedings, 2017-11, Vol.92 (11), p.1636-1643

2017

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A Phase IIa Controlled Human Malaria Infection and Immunogenicity Study of RTS,S/AS01E and RTS,S/AS01B Delayed Fractional Dose Regimens in Malaria-Naive Adults

The Journal of infectious diseases, 2020-10, Vol.222 (10), p.1681-1691

2020

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Extremely Elevated Erythrocyte Sedimentation Rates: Associations With Patients' Diagnoses, Demographic Characteristics, and Comorbidities

Mayo Clinic proceedings, 2017-11, Vol.92 (11), p.1636

2017

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Infectious Complications of CAR T-Cell Therapy in Non-Hodgkin Lymphoma

Biology of blood and marrow transplantation, 2020-03, Vol.26 (3), p.S275-S275

2020

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Trisomy of human chromosome 21 enhances amyloid-β deposition independently of an extra copy of APP

Genetic dissection of down syndrome-associated alterations in APP/amyloid-β biology using mouse models

Dissecting Alzheimer disease in Down syndrome using mouse models

Publisher Correction: Genetic dissection of down syndrome‑associated alterations in APP/amyloid‑β biology using mouse models

Gene expression dysregulation domains are not a specific feature of Down syndrome

Comprehensive phenotypic analysis of the Dp1Tyb mouse strain reveals a broad range of Down syndrome-related phenotypes

The integration site of the APP transgene in the J20 mouse model of Alzheimer's disease

The integration site of the APP transgene in the J20 mouse model of Alzheimer’s disease

Mouse models of aneuploidy to understand chromosome disorders

Section on Cognitive Neuropsychology, Laboratory of Brain and Cognition, National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland 20892

Modelling of the development and response to amyloid‐β accumulation in the context of trisomy21 in the rodent brain

Genetic Mapping of APP and Amyloid-β Biology Modulation by Trisomy 21

Genetic mapping of APP and amyloid‐β biology modulation by trisomy 21

Alzheimer's disease in Down syndrome : mapping modifiers of Alzheimer's pathology in novel mouse crosses

Downregulated Wnt/β-catenin signalling in the Down syndrome hippocampus

Using mouse models to understand Alzheimer's disease mechanisms in the context of trisomy of chromosome 21

TRISOMY 21 CAUSES A DEFICIT IN LYSOSOMAL CATHEPSINS AND ALTERS APP/Aβ PROCESSING, INDEPENDENTLY OF AN EXTRA COPY OF APP

Structure activity relationship of novel antiviral nucleosides against Enterovirus A71

Extremely Elevated Erythrocyte Sedimentation Rates: Associations With Patients' Diagnoses, Demographic Characteristics, and Comorbidities

A Phase IIa Controlled Human Malaria Infection and Immunogenicity Study of RTS,S/AS01E and RTS,S/AS01B Delayed Fractional Dose Regimens in Malaria-Naive Adults

Extremely Elevated Erythrocyte Sedimentation Rates: Associations With Patients' Diagnoses, Demographic Characteristics, and Comorbidities

Infectious Complications of CAR T-Cell Therapy in Non-Hodgkin Lymphoma

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