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A phospho-switch controls RNF43-mediated degradation of Wnt receptors to suppress tumorigenesis

Nature communications, 2020-09, Vol.11 (1), p.1-16, Article 4586

2020

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The 3′ Pol II pausing at replication-dependent histone genes is regulated by Mediator through Cajal bodies’ association with histone locus bodies

Nature communications, 2022-05, Vol.13 (1), p.2905-2905, Article 2905

2022

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The role of Mediator and Little Elongation Complex in transcription termination

Nature communications, 2020-02, Vol.11 (1), p.1063-1063, Article 1063

2020

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Spatial regulation of VEGF receptor endocytosis in angiogenesis

Nature cell biology, 2013-03, Vol.15 (3), p.249-260

2013

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PAR3 restricts the expansion of neural precursor cells by regulating hedgehog signaling

Development (Cambridge), 2022-11, Vol.149 (21)

2022

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GABAB receptor signaling in the caudate putamen is involved in binge-like consumption during a high fat diet in mice

Scientific reports, 2021-09, Vol.11 (1), p.19296-19296, Article 19296

2021

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Phosphorylation and dephosphorylation of Ser852 and Ser889 control the clustering, localization and function of PAR3

Journal of cell science, 2020-11, Vol.133 (22)

2020

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Biallelic Mutations in Nuclear Pore Complex Subunit NUP107 Cause Early-Childhood-Onset Steroid-Resistant Nephrotic Syndrome

American journal of human genetics, 2015-10, Vol.97 (4), p.555-566

2015

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Tumor Type-Dependent Function of the Par3 Polarity Protein in Skin Tumorigenesis

Cancer cell, 2012-09, Vol.22 (3), p.389-403

2012

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aPKC controls endothelial growth by modulating c-Myc via FoxO1 DNA-binding ability

Nature communications, 2018-12, Vol.9 (1), p.5357-13, Article 5357

2018

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Atypical protein kinase C isoforms differentially regulate directional keratinocyte migration during wound healing

Journal of dermatological science, 2019-02, Vol.93 (2), p.101-108

2019

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ASPP2 Regulates Epithelial Cell Polarity through the PAR Complex

Current biology, 2010-08, Vol.20 (15), p.1408-1414

2010

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Inflammation in VTA Caused by HFD Induces Activation of Dopaminergic Neurons Accompanied by Binge-like Eating

Nutrients, 2022-09, Vol.14 (18), p.3835

2022

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Dietary sodium chloride attenuates increased β-cell mass to cause glucose intolerance in mice under a high-fat diet

PloS one, 2021-03, Vol.16 (3), p.e0248065-e0248065

2021

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aPKCλ maintains the integrity of the glomerular slit diaphragm through trafficking of nephrin to the cell surface

Journal of biochemistry (Tokyo), 2014-08, Vol.156 (2), p.115-128

2014

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Atypical Protein Kinase C Is Involved in the Evolutionarily Conserved PAR Protein Complex and Plays a Critical Role in Establishing Epithelia-Specific Junctional Structures

The Journal of cell biology, 2001-03, Vol.152 (6), p.1183-1196

2001

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Loss of aPKCλ in differentiated neurons disrupts the polarity complex but does not induce obvious neuronal loss or disorientation in mouse brains

PloS one, 2013-12, Vol.8 (12), p.e84036

2013

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PAR3 is essential for cyst-mediated epicardial development by establishing apical cortical domains

Development (Cambridge), 2006-04, Vol.133 (7), p.1389-1398

2006

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Regulated protein–protein interaction between aPKC and PAR‐3 plays an essential role in the polarization of epithelial cells

Genes to cells : devoted to molecular & cellular mechanisms, 2002-11, Vol.7 (11), p.1161-1171

2002

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aPKC restricts the basolateral determinant PtdIns(3,4,5)P3 to the basal region

Biochemical and biophysical research communications, 2008-04, Vol.368 (2), p.249-255

2008

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PAR‐6 regulates aPKC activity in a novel way and mediates cell‐cell contact‐induced formation of the epithelial junctional complex

Genes to cells : devoted to molecular & cellular mechanisms, 2001-08, Vol.6 (8), p.721-731

2001

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Intrarenal suppression of angiotensin II type 1 receptor binding molecule in angiotensin II-infused mice

American journal of physiology. Renal physiology, 2010-11, Vol.299 (5), p.F991-F1003

2010

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Self-association of PAR-3-mediated by the Conserved N-terminal Domain Contributes to the Development of Epithelial Tight Junctions

The Journal of biological chemistry, 2003-08, Vol.278 (33), p.31240-31250

2003

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Muscle Develops a Specific Form of Small Heat Shock Protein Complex Composed of MKBP/HSPB2 and HSPB3 during Myogenic Differentiation

The Journal of biological chemistry, 2000-01, Vol.275 (2), p.1095-1104

2000

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Involvement of ASIP/PAR-3 in the promotion of epithelial tight junction formation

Journal of cell science, 2002-06, Vol.115 (Pt 12), p.2485-2495

2002

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A phospho-switch controls RNF43-mediated degradation of Wnt receptors to suppress tumorigenesis

The 3′ Pol II pausing at replication-dependent histone genes is regulated by Mediator through Cajal bodies’ association with histone locus bodies

The role of Mediator and Little Elongation Complex in transcription termination

Spatial regulation of VEGF receptor endocytosis in angiogenesis

PAR3 restricts the expansion of neural precursor cells by regulating hedgehog signaling

GABAB receptor signaling in the caudate putamen is involved in binge-like consumption during a high fat diet in mice

Phosphorylation and dephosphorylation of Ser852 and Ser889 control the clustering, localization and function of PAR3

Biallelic Mutations in Nuclear Pore Complex Subunit NUP107 Cause Early-Childhood-Onset Steroid-Resistant Nephrotic Syndrome

Tumor Type-Dependent Function of the Par3 Polarity Protein in Skin Tumorigenesis

aPKC controls endothelial growth by modulating c-Myc via FoxO1 DNA-binding ability

Atypical protein kinase C isoforms differentially regulate directional keratinocyte migration during wound healing

ASPP2 Regulates Epithelial Cell Polarity through the PAR Complex

Inflammation in VTA Caused by HFD Induces Activation of Dopaminergic Neurons Accompanied by Binge-like Eating

Dietary sodium chloride attenuates increased β-cell mass to cause glucose intolerance in mice under a high-fat diet

aPKCλ maintains the integrity of the glomerular slit diaphragm through trafficking of nephrin to the cell surface

Atypical Protein Kinase C Is Involved in the Evolutionarily Conserved PAR Protein Complex and Plays a Critical Role in Establishing Epithelia-Specific Junctional Structures

Loss of aPKCλ in differentiated neurons disrupts the polarity complex but does not induce obvious neuronal loss or disorientation in mouse brains

PAR3 is essential for cyst-mediated epicardial development by establishing apical cortical domains

Regulated protein–protein interaction between aPKC and PAR‐3 plays an essential role in the polarization of epithelial cells

aPKC restricts the basolateral determinant PtdIns(3,4,5)P3 to the basal region

PAR‐6 regulates aPKC activity in a novel way and mediates cell‐cell contact‐induced formation of the epithelial junctional complex

Intrarenal suppression of angiotensin II type 1 receptor binding molecule in angiotensin II-infused mice

Self-association of PAR-3-mediated by the Conserved N-terminal Domain Contributes to the Development of Epithelial Tight Junctions

Muscle Develops a Specific Form of Small Heat Shock Protein Complex Composed of MKBP/HSPB2 and HSPB3 during Myogenic Differentiation

Involvement of ASIP/PAR-3 in the promotion of epithelial tight junction formation

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