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Staphylococcal phages and pathogenicity islands drive plasmid evolution

Nature communications, 2021-10, Vol.12 (1), p.5845-5845, Article 5845

2021

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Interactions between horizontally acquired genes create a fitness cost in Pseudomonas aeruginosa

Nature communications, 2015-04, Vol.6 (1), p.6845-6845, Article 6845

2015

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Mistranslation can enhance fitness through purging of deleterious mutations

Nature communications, 2017-05, Vol.8 (1), p.15410-15410, Article 15410

2017

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Integrative analysis of fitness and metabolic effects of plasmids in Pseudomonas aeruginosa PAO1

The ISME Journal, 2018-12, Vol.12 (12), p.3014-3024

2018

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Fitness is strongly influenced by rare mutations of large effect in a microbial mutation accumulation experiment

Genetics (Austin), 2014-07, Vol.197 (3), p.981-990

2014

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Epistatic interactions between ancestral genotype and beneficial mutations shape evolvability in Pseudomonas aeruginosa

Evolution, 2016-07, Vol.70 (7), p.1659-1666

2016

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The Genomic Basis of Evolutionary Innovation in Pseudomonas aeruginosa

PLoS genetics, 2016-05, Vol.12 (5), p.e1006005

2016

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Natural selection drives the accumulation of amino acid tandem repeats in human proteins

Genome research, 2010-06, Vol.20 (6), p.745-754

2010

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Emergence of novel domains in proteins

BMC evolutionary biology, 2013-02, Vol.13 (1), p.47, Article 47

2013

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Structure and age jointly influence rates of protein evolution

PLoS computational biology, 2012-05, Vol.8 (5), p.e1002542-e1002542

2012

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Origin of Primate Orphan Genes: A Comparative Genomics Approach

Molecular biology and evolution, 2009-03, Vol.26 (3), p.603-612

2009

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Role of Low-Complexity Sequences in the Formation of Novel Protein Coding Sequences

Molecular biology and evolution, 2012-03, Vol.29 (3), p.883-886

2012

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Genetic dominance governs the evolution and spread of mobile genetic elements in bacteria

Proceedings of the National Academy of Sciences - PNAS, 2020-07, Vol.117 (27), p.15755-15762

2020

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New insights on Pseudoalteromonas haloplanktis TAC125 genome organization and benchmarks of genome assembly applications using next and third generation sequencing technologies

Scientific reports, 2019-11, Vol.9 (1), p.16444-14, Article 16444

2019

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Lineage-specific variation in intensity of natural selection in mammals

Molecular biology and evolution, 2011-01, Vol.28 (1), p.383-398

2011

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A limit on the evolutionary rescue of an Antarctic bacterium from rising temperatures

Science advances, 2022-07, Vol.8 (28), p.eabk3511

2022

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The genomic basis of adaptation to the fitness cost of rifampicin resistance in Pseudomonas aeruginosa

Proceedings of the Royal Society. B, Biological sciences, 2016-01, Vol.283 (1822), p.20152452

2016

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Epistatic interactions between ancestral genotype and beneficial mutations shape evolvability in Pseudomonas aeruginosa: BRIEF COMMUNICATION

Evolution, 2016-07, Vol.70 (7), p.1659-1666

2016

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Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a blaVIM-1 amplification causing high-level carbapenem resistance

Journal of antimicrobial chemotherapy, 2015-11, Vol.70 (11), p.3000-3003

2015

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The genomic basis of adaptation to the fitness cost of rifampicin resistance in Pseudomonas aeruginosa

Proceedings of the Royal Society. B, Biological sciences, 2016-01, Vol.283 (1822), p.1-9

2016

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Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a bla VIM-1 amplification causing high-level carbapenem resistance

Journal of antimicrobial chemotherapy, 2015-11, Vol.70 (11), p.3000-3003

2015

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Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a bla sub(VIM-1) amplification causing high-level carbapenem resistance

Journal of antimicrobial chemotherapy, 2015-11, Vol.70 (11), p.3000-3003

2015

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Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a bla^sub VIM-1^ amplification causing high-level carbapenem resistance

Journal of antimicrobial chemotherapy, 2015-11, Vol.70 (11), p.3000

2015

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Here’s to the Losers: Evolvable Residents Accelerate the Evolution of High-Fitness Invaders

The American naturalist, 2015-07, Vol.186 (1), p.41-49

2015

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Sequence shortening in the rodent ancestor

Genome research, 2012-03, Vol.22 (3), p.478-485

2012

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Staphylococcal phages and pathogenicity islands drive plasmid evolution

Interactions between horizontally acquired genes create a fitness cost in Pseudomonas aeruginosa

Mistranslation can enhance fitness through purging of deleterious mutations

Integrative analysis of fitness and metabolic effects of plasmids in Pseudomonas aeruginosa PAO1

Fitness is strongly influenced by rare mutations of large effect in a microbial mutation accumulation experiment

Epistatic interactions between ancestral genotype and beneficial mutations shape evolvability in Pseudomonas aeruginosa

The Genomic Basis of Evolutionary Innovation in Pseudomonas aeruginosa

Natural selection drives the accumulation of amino acid tandem repeats in human proteins

Emergence of novel domains in proteins

Structure and age jointly influence rates of protein evolution

Origin of Primate Orphan Genes: A Comparative Genomics Approach

Role of Low-Complexity Sequences in the Formation of Novel Protein Coding Sequences

Genetic dominance governs the evolution and spread of mobile genetic elements in bacteria

New insights on Pseudoalteromonas haloplanktis TAC125 genome organization and benchmarks of genome assembly applications using next and third generation sequencing technologies

Lineage-specific variation in intensity of natural selection in mammals

A limit on the evolutionary rescue of an Antarctic bacterium from rising temperatures

The genomic basis of adaptation to the fitness cost of rifampicin resistance in Pseudomonas aeruginosa

Epistatic interactions between ancestral genotype and beneficial mutations shape evolvability in Pseudomonas aeruginosa: BRIEF COMMUNICATION

Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a blaVIM-1 amplification causing high-level carbapenem resistance

The genomic basis of adaptation to the fitness cost of rifampicin resistance in Pseudomonas aeruginosa

Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a bla VIM-1 amplification causing high-level carbapenem resistance

Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a bla sub(VIM-1) amplification causing high-level carbapenem resistance

Sequencing of plasmids pAMBL1 and pAMBL2 from Pseudomonas aeruginosa reveals a bla^sub VIM-1^ amplification causing high-level carbapenem resistance

Here’s to the Losers: Evolvable Residents Accelerate the Evolution of High-Fitness Invaders

Sequence shortening in the rodent ancestor

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