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Opioid-induced hyperalgesia: Are thalamic T-type calcium channels treatment targets?

The Journal of clinical investigation, 2022-12, Vol.132 (24), p.1-2

Todorovic, Slobodan M

2022

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General Anesthesia Causes Long-term Impairment of Mitochondrial Morphogenesis and Synaptic Transmission in Developing Rat Brain

Anesthesiology (Philadelphia), 2011-11, Vol.115 (5), p.992-1002

2011

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Is Diabetic Nerve Pain Caused by Dysregulated Ion Channels in Sensory Neurons?

Diabetes (New York, N.Y.), 2015-12, Vol.64 (12), p.3987-3989

Todorovic, Slobodan M

2015

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Thalamic T-Type Calcium Channels as Targets for Hypnotics and General Anesthetics

International journal of molecular sciences, 2022-02, Vol.23 (4), p.2349

2022

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Neonatal ketamine exposure impairs infrapyramidal bundle pruning and causes lasting increase in excitatory synaptic transmission in hippocampal CA3 neurons

Neurobiology of disease, 2022-12, Vol.175, p.105923-105923, Article 105923

2022

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Neonatal general anesthesia causes lasting alterations in excitatory and inhibitory synaptic transmission in the ventrobasal thalamus of adolescent female rats

Neurobiology of disease, 2019-07, Vol.127, p.472-481

2019

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The Role of Neuroactive Steroids in Analgesia and Anesthesia: An Interesting Comeback?

Biomolecules (Basel, Switzerland), 2023-11, Vol.13 (11), p.1654

2023

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General Anesthesia and the Young Brain: The Importance of Novel Strategies with Alternate Mechanisms of Action

International journal of molecular sciences, 2022-02, Vol.23 (3), p.1889

2022

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Painful diabetic neuropathy leads to functional CaV3.2 expression and spontaneous activity in skin nociceptors of mice

Experimental neurology, 2021-12, Vol.346, p.113838-113838, Article 113838

2021

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CaV3.2 T-type calcium channels in peripheral sensory neurons are important for mibefradil-induced reversal of hyperalgesia and allodynia in rats with painful diabetic neuropathy

PloS one, 2014-04, Vol.9 (4), p.e91467-e91467

2014

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Neurosteroids in Pain Management: A New Perspective on an Old Player

Frontiers in pharmacology, 2018-10, Vol.9, p.1127-1127

2018

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Alpha lipoic acid attenuates evoked and spontaneous pain following surgical skin incision in rats

Channels (Austin, Tex.), 2021-01, Vol.15 (1), p.398-407

2021

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Glycosylation of CaV3.2 Channels Contributes to the Hyperalgesia in Peripheral Neuropathy of Type 1 Diabetes

Frontiers in cellular neuroscience, 2020-12, Vol.14, p.605312-605312

2020

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Hyperexcitability of rat thalamocortical networks after exposure to general anesthesia during brain development

The Journal of neuroscience, 2015-01, Vol.35 (4), p.1481-1492

2015

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Global genetic deletion of CaV3.3 channels facilitates anaesthetic induction and enhances isoflurane-sparing effects of T-type calcium channel blockers

Scientific reports, 2020-12, Vol.10 (1), p.21510-21510, Article 21510

2020

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Redox and trace metal regulation of ion channels in the pain pathway

Biochemical journal, 2015-09, Vol.470 (3), p.275-280

2015

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Further Evidence that Inhibition of Neuronal Voltage-Gated Calcium Channels Contributes to the Hypnotic Effect of Neurosteroid Analogue, 3β-OH

Frontiers in pharmacology, 2022-05, Vol.13, p.850658-850658

2022

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The Mechanisms of Plasticity of Nociceptive Ion Channels in Painful Diabetic Neuropathy

Frontiers in pain research (Lausanne, Switzerland), 2022-03, Vol.3, p.869735

2022

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Selective T-Type Calcium Channel Blockade Alleviates Hyperalgesia in ob/ob Mice

Diabetes (New York, N.Y.), 2009-11, Vol.58 (11), p.2656-2665

2009

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Neonatal Isoflurane Does Not Affect Sleep Architecture and Minimally Alters Neuronal Beta Oscillations in Adolescent Rats

Frontiers in behavioral neuroscience, 2021-11, Vol.15, p.703859

2021

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CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy

Genes & development, 2015-07, Vol.29 (14), p.1535-1551

2015

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Pharmacological Antagonism of T-Type Calcium Channels Constrains Rebound Burst Firing in Two Distinct Subpopulations of GABA Neurons in the Rat Ventral Tegmental Area: Implications for α-Lipoic Acid

Frontiers in pharmacology, 2019-11, Vol.10, p.1402-1402

2019

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Neonatal Ketamine Alters High-Frequency Oscillations and Synaptic Plasticity in the Subiculum But Does not Affect Sleep Macrostructure in Adolescent Rats

Frontiers in systems neuroscience, 2020-05, Vol.14, p.26

2020

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T‐type voltage‐gated calcium channels as targets for the development of novel pain therapies

British journal of pharmacology, 2011-06, Vol.163 (3), p.484-495

2011

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Preemptive Analgesic Effect of Intrathecal Applications of Neuroactive Steroids in a Rodent Model of Post-Surgical Pain: Evidence for the Role of T-Type Calcium Channels

Cells (Basel, Switzerland), 2020-12, Vol.9 (12), p.2674

2020

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Opioid-induced hyperalgesia: Are thalamic T-type calcium channels treatment targets?

General Anesthesia Causes Long-term Impairment of Mitochondrial Morphogenesis and Synaptic Transmission in Developing Rat Brain

Is Diabetic Nerve Pain Caused by Dysregulated Ion Channels in Sensory Neurons?

Thalamic T-Type Calcium Channels as Targets for Hypnotics and General Anesthetics

Neonatal ketamine exposure impairs infrapyramidal bundle pruning and causes lasting increase in excitatory synaptic transmission in hippocampal CA3 neurons

Neonatal general anesthesia causes lasting alterations in excitatory and inhibitory synaptic transmission in the ventrobasal thalamus of adolescent female rats

The Role of Neuroactive Steroids in Analgesia and Anesthesia: An Interesting Comeback?

General Anesthesia and the Young Brain: The Importance of Novel Strategies with Alternate Mechanisms of Action

Painful diabetic neuropathy leads to functional CaV3.2 expression and spontaneous activity in skin nociceptors of mice

CaV3.2 T-type calcium channels in peripheral sensory neurons are important for mibefradil-induced reversal of hyperalgesia and allodynia in rats with painful diabetic neuropathy

Neurosteroids in Pain Management: A New Perspective on an Old Player

Alpha lipoic acid attenuates evoked and spontaneous pain following surgical skin incision in rats

Glycosylation of CaV3.2 Channels Contributes to the Hyperalgesia in Peripheral Neuropathy of Type 1 Diabetes

Hyperexcitability of rat thalamocortical networks after exposure to general anesthesia during brain development

Global genetic deletion of CaV3.3 channels facilitates anaesthetic induction and enhances isoflurane-sparing effects of T-type calcium channel blockers

Redox and trace metal regulation of ion channels in the pain pathway

Further Evidence that Inhibition of Neuronal Voltage-Gated Calcium Channels Contributes to the Hypnotic Effect of Neurosteroid Analogue, 3β-OH

The Mechanisms of Plasticity of Nociceptive Ion Channels in Painful Diabetic Neuropathy

Selective T-Type Calcium Channel Blockade Alleviates Hyperalgesia in ob/ob Mice

Neonatal Isoflurane Does Not Affect Sleep Architecture and Minimally Alters Neuronal Beta Oscillations in Adolescent Rats

CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy

Pharmacological Antagonism of T-Type Calcium Channels Constrains Rebound Burst Firing in Two Distinct Subpopulations of GABA Neurons in the Rat Ventral Tegmental Area: Implications for α-Lipoic Acid

Neonatal Ketamine Alters High-Frequency Oscillations and Synaptic Plasticity in the Subiculum But Does not Affect Sleep Macrostructure in Adolescent Rats

T‐type voltage‐gated calcium channels as targets for the development of novel pain therapies

Preemptive Analgesic Effect of Intrathecal Applications of Neuroactive Steroids in a Rodent Model of Post-Surgical Pain: Evidence for the Role of T-Type Calcium Channels

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