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Reversal of Chronic Molecular and Cellular Abnormalities Due to Heart Failure by Passive Mechanical Ventricular Containment

Circulation research, 2003-11, Vol.93 (11), p.1095-1101

2003

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Inhibition of mitochondrial permeability transition pores by cyclosporine A improves cytochrome C oxidase function and increases rate of ATP synthesis in failing cardiomyocytes

Heart failure reviews, 2005-12, Vol.10 (4), p.305-310

2005

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Abnormal Mitochondrial Respiration in Failed Human Myocardium

Journal of molecular and cellular cardiology, 2000-12, Vol.32 (12), p.2361-2367

2000

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Expression of Cytoskeletal, Linkage and Extracellular Proteins in Failing Dog Myocardium

Heart failure reviews, 2005-12, Vol.10 (4), p.297-303

2005

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Left ventricular histomorphometric findings in dogs with heart failure treated with the Acorn Cardiac Support Device

Heart failure reviews, 2005-06, Vol.10 (2), p.141-147

2005

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Cyclosporine A attenuates mitochondrial permeability transition and improves mitochondrial respiratory function in cardiomyocytes isolated from dogs with heart failure

Journal of molecular and cellular cardiology, 2007-01, Vol.42 (1), p.150-158

2007

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Chronic therapy with metoprolol CR/XL prevents apoptosis inducing factor and downregulates the proapoptotic protein Bak in cardiomyocytes of dogs with heart failure

Journal of the American College of Cardiology, 2002-03, Vol.39, p.165-165

2002

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Effects of long-term monotherapy with eplerenone, a novel aldosterone blocker, on progression of left ventricular dysfunction and remodeling in dogs with heart failure

Circulation (New York, N.Y.), 2002-12, Vol.106 (23), p.2967-2972

2002

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Selective matrix metalloproteinase inhibition attenuates progression of left ventricular dysfunction and remodeling in dogs with chronic heart failure

American journal of physiology. Heart and circulatory physiology, 2006-06, Vol.290 (6), p.H2522-H2527

2006

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Chronic therapy with metoprolol attenuates cardiomyocyte apoptosis in dogs with heart failure

Journal of the American College of Cardiology, 2000-11, Vol.36 (5), p.1698-1705

2000

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Effects of the AT 1 ‐receptor antagonist eprosartan on the progression of left ventricular dysfunction in dogs with heart failure

British journal of pharmacology, 2009-01, Vol.138 (2), p.301-309

2009

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Hypoxia, angiotensin-II, and norepinephrine mediated apoptosis is stimulus specific in canine failed cardiomyocytes: a role for p38 MAPK, Fas-L and cyclin D1

European journal of heart failure, 2003-03, Vol.5 (2), p.121-129

2003

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Expression of cytoskeletal, linkage and extracellular proteins in failing dog myocardium

Heart failure reviews, 2005-12, Vol.10 (4), p.297-303

2005

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Hypoxia, angiotensin‐II, and norepinephrine mediated apoptosis is stimulus specific in canine failed cardiomyocytes: a role for p38 MAPK, Fas‐L and cyclin D 1

European journal of heart failure, 2003-03, Vol.5 (2), p.121-129

2003

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1116-12 PG-530742, a novel matrix metalloproteinase inhibitor, improves left ventricular function and attenuates remodeling in dogs with chronic heart failure

Journal of the American College of Cardiology, 2004-03, Vol.43 (5), p.A22-A22

2004

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Hypoxia-induced cleavage of caspase-3 and DFF45/ICAD in human failed cardiomyocytes

American journal of physiology. Heart and circulatory physiology, 2002-09, Vol.283 (3), p.H990-H995

2002

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Effects of long-term therapy with bosentan on the progression of left ventricular dysfunction and remodeling in dogs with heart failure

Journal of the American College of Cardiology, 2000-01, Vol.35 (1), p.222-229

2000

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Mitochondrial membrane potential is decreased in failing cardiomyocytes and is associated with decreased cytochrome oxidase-dependent respiration

Journal of the American College of Cardiology, 2003-03, Vol.41 (6), p.155-155

2003

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P4-02 Cardiac mRNA gene expression of alpha-myosin heavy chain is decreased and beta-myosin heavy chain is increased in both ischemic and idiopathic dilated cardiomyopathy

International journal of cardiology, 2004, Vol.97, p.S54-S54

2004

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Hypoxia-induced cleavage of caspase-3 and DFF45/ICAD in human failed cardiomyocytes

American journal of physiology. Heart and circulatory physiology, 2002-09, Vol.52 (3), p.H990-H995

2002

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Mitochondrial respiration is depressed early in the course of evolving left ventricular dysfunction in dogs with heart failure

Journal of cardiac failure, 1999-09, Vol.5 (3), p.6-6

1999

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Effects of the AT1‐receptor antagonist eprosartan on the progression of left ventricular dysfunction in dogs with heart failure

British journal of pharmacology, 2003-01, Vol.138 (2), p.301-309

2003

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Hypoxia triggers increased expression of p38 MAPK in cardiomycytes isolated from patients with idiopathic dilated cardiomyopathy

European journal of heart failure, 2000-06, Vol.2 (S1), p.54-54

2000

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Effects of chronic neutral endopeptidase inhibition on the progression of left ventricular dysfunction and remodeling in dogs with moderate heart failure

Cardiovascular drugs and therapy, 2002-05, Vol.16 (3), p.209-214

2002

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Reversal of Chronic Molecular and Cellular Abnormalities Due to Heart Failure by Passive Mechanical Ventricular Containment

Inhibition of mitochondrial permeability transition pores by cyclosporine A improves cytochrome C oxidase function and increases rate of ATP synthesis in failing cardiomyocytes

Abnormal Mitochondrial Respiration in Failed Human Myocardium

Expression of Cytoskeletal, Linkage and Extracellular Proteins in Failing Dog Myocardium

Left ventricular histomorphometric findings in dogs with heart failure treated with the Acorn Cardiac Support Device

Cyclosporine A attenuates mitochondrial permeability transition and improves mitochondrial respiratory function in cardiomyocytes isolated from dogs with heart failure

Chronic therapy with metoprolol CR/XL prevents apoptosis inducing factor and downregulates the proapoptotic protein Bak in cardiomyocytes of dogs with heart failure

Effects of long-term monotherapy with eplerenone, a novel aldosterone blocker, on progression of left ventricular dysfunction and remodeling in dogs with heart failure

Selective matrix metalloproteinase inhibition attenuates progression of left ventricular dysfunction and remodeling in dogs with chronic heart failure

Chronic therapy with metoprolol attenuates cardiomyocyte apoptosis in dogs with heart failure

Effects of the AT 1 ‐receptor antagonist eprosartan on the progression of left ventricular dysfunction in dogs with heart failure

Hypoxia, angiotensin-II, and norepinephrine mediated apoptosis is stimulus specific in canine failed cardiomyocytes: a role for p38 MAPK, Fas-L and cyclin D1

Expression of cytoskeletal, linkage and extracellular proteins in failing dog myocardium

Hypoxia, angiotensin‐II, and norepinephrine mediated apoptosis is stimulus specific in canine failed cardiomyocytes: a role for p38 MAPK, Fas‐L and cyclin D 1

1116-12 PG-530742, a novel matrix metalloproteinase inhibitor, improves left ventricular function and attenuates remodeling in dogs with chronic heart failure

Hypoxia-induced cleavage of caspase-3 and DFF45/ICAD in human failed cardiomyocytes

Effects of long-term therapy with bosentan on the progression of left ventricular dysfunction and remodeling in dogs with heart failure

Mitochondrial membrane potential is decreased in failing cardiomyocytes and is associated with decreased cytochrome oxidase-dependent respiration

P4-02 Cardiac mRNA gene expression of alpha-myosin heavy chain is decreased and beta-myosin heavy chain is increased in both ischemic and idiopathic dilated cardiomyopathy

Hypoxia-induced cleavage of caspase-3 and DFF45/ICAD in human failed cardiomyocytes

Mitochondrial respiration is depressed early in the course of evolving left ventricular dysfunction in dogs with heart failure

Effects of the AT1‐receptor antagonist eprosartan on the progression of left ventricular dysfunction in dogs with heart failure

Hypoxia triggers increased expression of p38 MAPK in cardiomycytes isolated from patients with idiopathic dilated cardiomyopathy

Effects of chronic neutral endopeptidase inhibition on the progression of left ventricular dysfunction and remodeling in dogs with moderate heart failure

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