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Conditional Ablation of Macrophages Halts Progression of Crescentic Glomerulonephritis

The American journal of pathology, 2005-11, Vol.167 (5), p.1207-1219

2005

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The Role of Interleukin-4 and Interleukin-12 in the Progression of Atherosclerosis in Apolipoprotein E-Deficient Mice

The American journal of pathology, 2003-09, Vol.163 (3), p.1117-1125

2003

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Protease-Activated Receptor-2 Augments Experimental Crescentic Glomerulonephritis

The American journal of pathology, 2007-09, Vol.171 (3), p.800-808

2007

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Intrinsic renal cells are the major source of tumor necrosis factor contributing to renal injury in murine crescentic glomerulonephritis

Journal of the American Society of Nephrology, 2003-07, Vol.14 (7), p.1785-1793

2003

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T cells in crescentic glomerulonephritis

Journal of the American Society of Nephrology, 2006-05, Vol.17 (5), p.1253-1263

2006

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A pathogenetic role for mast cells in experimental crescentic glomerulonephritis

Journal of the American Society of Nephrology, 2006, Vol.17 (1), p.150-159

2006

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Plasminogen activator inhibitor-1 is a significant determinant of renal injury in experimental crescentic glomerulonephritis

Journal of the American Society of Nephrology, 2003-06, Vol.14 (6), p.1487-1495

2003

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Granulocyte macrophage colony-stimulating factor expression by both renal parenchymal and immune cells mediates murine crescentic glomerulonephritis

Journal of the American Society of Nephrology, 2005-09, Vol.16 (9), p.2646-2656

2005

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Interleukin-8 production by macrophages from atheromatous plaques

Arteriosclerosis, thrombosis, and vascular biology, 1996-08, Vol.16 (8), p.1007

1996

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Glomerular expression of CD80 and CD86 is required for leukocyte accumulation and injury in crescentic glomerulonephritis

Journal of the American Society of Nephrology, 2005-07, Vol.16 (7), p.2012-2022

2005

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The requirement for granulocyte-macrophage colony-stimulating factor and granulocyte colony-stimulating factor in leukocyte-mediated immune glomerular injury

Journal of the American Society of Nephrology, 2002-02, Vol.13 (2), p.350-358

2002

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Toll-like receptors: the interface between innate and adaptive immunity

Journal of the American Society of Nephrology, 2006-07, Vol.17 (7), p.1769-1771

Tipping, Peter G

2006

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Are podocytes passive or provocative in proteinuric glomerular pathology?

Journal of the American Society of Nephrology, 2008-04, Vol.19 (4), p.651-653

Tipping, Peter G

2008

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The cytoplasmic domain of tissue factor in macrophages augments cutaneous delayed-type hypersensitivity

Journal of leukocyte biology, 2008-04, Vol.83 (4), p.902-911

2008

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Cytokines in Glomerulonephritis

Seminars in nephrology, 2007-05, Vol.27 (3), p.275-285

2007

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Contributions of intrinsic renal cells to crescentic glomerulonephritis

Nephron. Experimental nephrology, 2005-01, Vol.101 (4), p.e173

2005

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Interleukin-10 inhibits macrophage-induced glomerular injury

Journal of the American Society of Nephrology, 2000-02, Vol.11 (2), p.262-269

2000

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Crescentic nephritis—is it in your genes?

Nephrology, dialysis, transplantation, 2008-10, Vol.23 (10), p.3065-3066

Tipping, Peter G.

2008

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Leukocytes in glomerular injury

Seminars in immunopathology, 2007-11, Vol.29 (4), p.355-374

2007

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Protease-activated receptor 1 mediates thrombin-dependent, cell-mediated renal inflammation in crescentic glomerulonephritis

The Journal of experimental medicine, 2000-02, Vol.191 (3), p.455-462

2000

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Tissue Factor and Factor VIIa Receptor/Ligand Interactions Induce Proinflammatory Effects in Macrophages

Blood, 1999-11, Vol.94 (10), p.3413-3420

1999

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The cytoplasmic domain of tissue factor restricts physiological albuminuria and pathological proteinuria associated with glomerulonephritis in mice

Nephron, 2010-01, Vol.116 (4), p.e72

2010

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IL-12p40 and IL-18 in crescentic glomerulonephritis : IL-12p40 is the key Th1-defining cytokine chain, whereas IL-18 promotes local inflammation and leukocyte recruitment

Journal of the American Society of Nephrology, 2005-07, Vol.16 (7), p.2023-2033

2005

Volltextzugriff (PDF)

Leukocyte-Derived Interleukin-1β Interacts with Renal Interleukin-1 Receptor I to Promote Renal Tumor Necrosis Factor and Glomerular Injury in Murine Crescentic Glomerulonephritis

The American journal of pathology, 2004-06, Vol.164 (6), p.1967-1977

2004

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CD80 and CD86 costimulatory molecules regulate crescentic glomerulonephritis by different mechanisms

Kidney international, 2005-08, Vol.68 (2), p.584-594

2005

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Conditional Ablation of Macrophages Halts Progression of Crescentic Glomerulonephritis

The Role of Interleukin-4 and Interleukin-12 in the Progression of Atherosclerosis in Apolipoprotein E-Deficient Mice

Protease-Activated Receptor-2 Augments Experimental Crescentic Glomerulonephritis

Intrinsic renal cells are the major source of tumor necrosis factor contributing to renal injury in murine crescentic glomerulonephritis

T cells in crescentic glomerulonephritis

A pathogenetic role for mast cells in experimental crescentic glomerulonephritis

Plasminogen activator inhibitor-1 is a significant determinant of renal injury in experimental crescentic glomerulonephritis

Granulocyte macrophage colony-stimulating factor expression by both renal parenchymal and immune cells mediates murine crescentic glomerulonephritis

Interleukin-8 production by macrophages from atheromatous plaques

Glomerular expression of CD80 and CD86 is required for leukocyte accumulation and injury in crescentic glomerulonephritis

The requirement for granulocyte-macrophage colony-stimulating factor and granulocyte colony-stimulating factor in leukocyte-mediated immune glomerular injury

Toll-like receptors: the interface between innate and adaptive immunity

Are podocytes passive or provocative in proteinuric glomerular pathology?

The cytoplasmic domain of tissue factor in macrophages augments cutaneous delayed-type hypersensitivity

Cytokines in Glomerulonephritis

Contributions of intrinsic renal cells to crescentic glomerulonephritis

Interleukin-10 inhibits macrophage-induced glomerular injury

Crescentic nephritis—is it in your genes?

Leukocytes in glomerular injury

Protease-activated receptor 1 mediates thrombin-dependent, cell-mediated renal inflammation in crescentic glomerulonephritis

Tissue Factor and Factor VIIa Receptor/Ligand Interactions Induce Proinflammatory Effects in Macrophages

The cytoplasmic domain of tissue factor restricts physiological albuminuria and pathological proteinuria associated with glomerulonephritis in mice

IL-12p40 and IL-18 in crescentic glomerulonephritis : IL-12p40 is the key Th1-defining cytokine chain, whereas IL-18 promotes local inflammation and leukocyte recruitment

Leukocyte-Derived Interleukin-1β Interacts with Renal Interleukin-1 Receptor I to Promote Renal Tumor Necrosis Factor and Glomerular Injury in Murine Crescentic Glomerulonephritis

CD80 and CD86 costimulatory molecules regulate crescentic glomerulonephritis by different mechanisms

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