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Smaug/SAMD4A restores translational activity of CUGBP1 and suppresses CUG-induced myopathy

PLoS genetics, 2013-04, Vol.9 (4), p.e1003445-e1003445

2013

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Overexpression of CUG Triplet Repeat-binding Protein, CUGBP1, in Mice Inhibits Myogenesis

The Journal of biological chemistry, 2004-03, Vol.279 (13), p.13129-13139

2004

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Ectopic expression of cyclin D3 corrects differentiation of DM1 myoblasts through activation of RNA CUG-binding protein, CUGBP1

Experimental cell research, 2008-07, Vol.314 (11), p.2266-2278

2008

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Disruption of Splicing Regulated by a CUG-Binding Protein in Myotonic Dystrophy

Science (American Association for the Advancement of Science), 1998-05, Vol.280 (5364), p.737-741

1998

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GSK3β mediates muscle pathology in myotonic dystrophy

The Journal of clinical investigation, 2012-12, Vol.122 (12), p.4461-4472

2012

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Expression of RNA CCUG Repeats Dysregulates Translation and Degradation of Proteins in Myotonic Dystrophy 2 Patients

The American journal of pathology, 2009-08, Vol.175 (2), p.748-762

2009

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Dysfunction of protein homeostasis in myotonic dystrophies

Histology and histopathology, 2013-09, Vol.28 (9), p.1089

2013

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RNA CUG Repeats Sequester CUGBP1 and Alter Protein Levels and Activity of CUGBP1

The Journal of biological chemistry, 2001-03, Vol.276 (11), p.7820-7826

2001

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Functional KCa1.1 channels are crucial for regulating the proliferation, migration and differentiation of human primary skeletal myoblasts

Cell death & disease, 2016-10, Vol.7 (10), p.e2426-e2426

2016

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Most expression and splicing changes in myotonic dystrophy type 1 and type 2 skeletal muscle are shared with other muscular dystrophies

Neuromuscular disorders : NMD, 2014-03, Vol.24 (3), p.227-240

2014

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RNA CUG-binding Protein 1 Increases Translation of 20-kDa Isoform of CCAAT/Enhancer-binding Protein β by Interacting with the α and β Subunits of Eukaryotic Initiation Translation Factor 2

The Journal of biological chemistry, 2005-05, Vol.280 (21), p.20549-20557

2005

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Age-specific CUGBP1-eIF2 Complex Increases Translation of CCAAT/Enhancer-binding Protein β in Old Liver

The Journal of biological chemistry, 2006-10, Vol.281 (43), p.32806-32819

2006

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CCUG repeats reduce the rate of global protein synthesis in myotonic dystrophy type 2

Reviews in the neurosciences, 2010, Vol.21 (1), p.19

2010

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Molecular Basis for Impaired Muscle Differentiation in Myotonic Dystrophy

Molecular and Cellular Biology, 2001-10, Vol.21 (20), p.6927-6938

2001

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Quantitative measurement of GLUT4 translocation to the plasma membrane by flow cytometry

Journal of visualized experiments, 2010-11 (45)

2010

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Calreticulin Interacts with C/EBPα and C/EBPβ mRNAs and Represses Translation of C/EBP Proteins

Molecular and Cellular Biology, 2002-10, Vol.22 (20), p.7242-7257

2002

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The molecular basis of Friedreich ataxia

Advances in experimental medicine and biology, 2002-01, Vol.516, p.99

Pandolfo, Massimo

2002

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Cardiac Elav-type RNA-binding Protein (ETR-3) Binds to RNA CUG Repeats Expanded in Myotonic Dystrophy

Human molecular genetics, 1999-01, Vol.8 (1), p.53-60

1999

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Altered Phosphorylation and Intracellular Distribution of a (CUG)n Triplet Repeat RNA-Binding Protein in Patients with Myotonic Dystrophy and in Myotonin Protein Kinase Knockout Mice

Proceedings of the National Academy of Sciences - PNAS, 1997-11, Vol.94 (24), p.13221-13226

1997

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Molecular mechanisms of TRS instability

Advances in experimental medicine and biology, 2002-01, Vol.516, p.1

2002

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Spinocerebellar ataxia type 10: a disease caused by a large ATTCT repeat expansion

Advances in experimental medicine and biology, 2002-01, Vol.516, p.79

2002

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Spinocerebellar ataxias caused by polyglutamine expansions

Advances in experimental medicine and biology, 2002-01, Vol.516, p.47

2002

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Myotonic dystrophy: discussion of molecular basis

Advances in experimental medicine and biology, 2002-01, Vol.516, p.27

2002

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Smaug/SAMD4A Restores Translational Activity of CUGBP1 and Suppresses CUG-Induced Myopathy: e1003445

PLoS genetics, 2013-04, Vol.9 (4)

2013

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GSK3[Beta] mediates muscle pathology in myotonic dystrophy

The Journal of clinical investigation, 2012-12, Vol.122 (12), p.4461

2012

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Smaug/SAMD4A restores translational activity of CUGBP1 and suppresses CUG-induced myopathy

Overexpression of CUG Triplet Repeat-binding Protein, CUGBP1, in Mice Inhibits Myogenesis

Ectopic expression of cyclin D3 corrects differentiation of DM1 myoblasts through activation of RNA CUG-binding protein, CUGBP1

Disruption of Splicing Regulated by a CUG-Binding Protein in Myotonic Dystrophy

GSK3β mediates muscle pathology in myotonic dystrophy

Expression of RNA CCUG Repeats Dysregulates Translation and Degradation of Proteins in Myotonic Dystrophy 2 Patients

Dysfunction of protein homeostasis in myotonic dystrophies

RNA CUG Repeats Sequester CUGBP1 and Alter Protein Levels and Activity of CUGBP1

Functional KCa1.1 channels are crucial for regulating the proliferation, migration and differentiation of human primary skeletal myoblasts

Most expression and splicing changes in myotonic dystrophy type 1 and type 2 skeletal muscle are shared with other muscular dystrophies

RNA CUG-binding Protein 1 Increases Translation of 20-kDa Isoform of CCAAT/Enhancer-binding Protein β by Interacting with the α and β Subunits of Eukaryotic Initiation Translation Factor 2

Age-specific CUGBP1-eIF2 Complex Increases Translation of CCAAT/Enhancer-binding Protein β in Old Liver

CCUG repeats reduce the rate of global protein synthesis in myotonic dystrophy type 2

Molecular Basis for Impaired Muscle Differentiation in Myotonic Dystrophy

Quantitative measurement of GLUT4 translocation to the plasma membrane by flow cytometry

Calreticulin Interacts with C/EBPα and C/EBPβ mRNAs and Represses Translation of C/EBP Proteins

The molecular basis of Friedreich ataxia

Cardiac Elav-type RNA-binding Protein (ETR-3) Binds to RNA CUG Repeats Expanded in Myotonic Dystrophy

Altered Phosphorylation and Intracellular Distribution of a (CUG)n Triplet Repeat RNA-Binding Protein in Patients with Myotonic Dystrophy and in Myotonin Protein Kinase Knockout Mice

Molecular mechanisms of TRS instability

Spinocerebellar ataxia type 10: a disease caused by a large ATTCT repeat expansion

Spinocerebellar ataxias caused by polyglutamine expansions

Myotonic dystrophy: discussion of molecular basis

Smaug/SAMD4A Restores Translational Activity of CUGBP1 and Suppresses CUG-Induced Myopathy: e1003445

GSK3[Beta] mediates muscle pathology in myotonic dystrophy

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