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Maternal imprinting at the H19-Igf2 locus maintains adult haematopoietic stem cell quiescence

Nature (London), 2013-08, Vol.500 (7462), p.345-349

2013

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Tissue-specific Grb10/Ddc insulator drives allelic architecture for cardiac development

Molecular cell, 2022-10, Vol.82 (19), p.3613-3631.e7

2022

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CTCF binding sites promote transcription initiation and prevent DNA methylation on the maternal allele at the imprinted H19/Igf2 locus

Human molecular genetics, 2006-10, Vol.15 (19), p.2945-2954

2006

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Dysregulated H19/Igf2 expression disrupts cardiac-placental axis during development of Silver-Russell syndrome-like mouse models

eLife, 2022-11, Vol.11

2022

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Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)

Science (American Association for the Advancement of Science), 2001-06, Vol.292 (5522), p.1728-1731

2001

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Tissue-specific and mosaic imprinting defects underlie opposite congenital growth disorders in mice

PLoS genetics, 2018-02, Vol.14 (2), p.e1007243

2018

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Derivation and investigation of the first human cell-based model of Beckwith-Wiedemann syndrome

Epigenetics, 2021-12, Vol.16 (12), p.1295-1305

2021

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H19 is not hypomethylated or upregulated with age or sex in the aortic valves of mice

Physiological reports, 2019-10, Vol.7 (19), p.e14244-n/a

2019

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A YY1 Bridge for X Inactivation

Cell, 2011-07, Vol.146 (1), p.11-13

2011

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Two sides of the Dlk1-Dio3 story in imprinting

Developmental cell, 2021-11, Vol.56 (22), p.3035-3037

2021

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The Transcriptional Status but Not the Imprinting Control Region Determines Allele-Specific Histone Modifications at the Imprinted H19 Locus

Molecular and Cellular Biology, 2008-01, Vol.28 (1), p.71-82

2008

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Developmental Profile of H19 Differentially Methylated Domain (DMD) Deletion Alleles Reveals Multiple Roles of the DMD in Regulating Allelic Expression and DNA Methylation at the Imprinted H19/Igf2 Locus

Molecular and Cellular Biology, 2006-02, Vol.26 (4), p.1245-1258

2006

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Tissue-specific insulator function at H19/Igf2 revealed by deletions at the imprinting control region

Human molecular genetics, 2014-12, Vol.23 (23), p.6246-6259

2014

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Humanized H19/Igf2 locus reveals diverged imprinting mechanism between mouse and human and reflects Silver–Russell syndrome phenotypes

Proceedings of the National Academy of Sciences - PNAS, 2016-09, Vol.113 (39), p.10938-10943

2016

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Analysis of Sequence Upstream of the Endogenous H19 Gene Reveals Elements Both Essential and Dispensable for Imprinting

Molecular and Cellular Biology, 2002-04, Vol.22 (8), p.2450-2462

2002

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Deletion of the H19 differentially methylated domain results in loss of imprinted expression of H19 and Igf2

Genes & development, 1998-12, Vol.12 (23), p.3693-3702

1998

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Nonrandom X chromosome inactivation is influenced by multiple regions on the murine X chromosome

Genetics (Austin), 2012-11, Vol.192 (3), p.1095-1107

2012

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Three-dimensional conformation at the H19/Igf2 locus supports a model of enhancer tracking

Human molecular genetics, 2008-10, Vol.17 (19), p.3021-3029

2008

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SnapShot: Imprinted Gene Clusters

Cell, 2007-09, Vol.130 (5), p.958.e1-958.e2

2007

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Novel cis-regulatory function in ICR-mediated imprinted repression of H19

Developmental biology, 2011-07, Vol.355 (2), p.349-357

2011

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X-tra! X-tra! News from the Mouse X Chromosome

Developmental biology, 2006-10, Vol.298 (2), p.344-353

2006

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Akt1/PKBα Is Required for Normal Growth but Dispensable for Maintenance of Glucose Homeostasis in Mice

The Journal of biological chemistry, 2001-10, Vol.276 (42), p.38349-38352

2001

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An N-Ethyl-N-Nitrosourea Mutagenesis Screen for Epigenetic Mutations in the Mouse

Genetics (Austin), 2003-08, Vol.164 (4), p.1481-1494

2003

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Mothers Setting Boundaries

Science (American Association for the Advancement of Science), 2000-06, Vol.288 (5474), p.2145-2146

2000

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Abstract 1936: Epigenetic challenges in derivation of the first cell-based model of Beckwith-Wiedemann Syndrome

Cancer research (Chicago, Ill.), 2017, Vol.77 (13_Supplement), p.1936-1936

2017

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Maternal imprinting at the H19-Igf2 locus maintains adult haematopoietic stem cell quiescence

Tissue-specific Grb10/Ddc insulator drives allelic architecture for cardiac development

CTCF binding sites promote transcription initiation and prevent DNA methylation on the maternal allele at the imprinted H19/Igf2 locus

Dysregulated H19/Igf2 expression disrupts cardiac-placental axis during development of Silver-Russell syndrome-like mouse models

Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)

Tissue-specific and mosaic imprinting defects underlie opposite congenital growth disorders in mice

Derivation and investigation of the first human cell-based model of Beckwith-Wiedemann syndrome

H19 is not hypomethylated or upregulated with age or sex in the aortic valves of mice

A YY1 Bridge for X Inactivation

Two sides of the Dlk1-Dio3 story in imprinting

The Transcriptional Status but Not the Imprinting Control Region Determines Allele-Specific Histone Modifications at the Imprinted H19 Locus

Developmental Profile of H19 Differentially Methylated Domain (DMD) Deletion Alleles Reveals Multiple Roles of the DMD in Regulating Allelic Expression and DNA Methylation at the Imprinted H19/Igf2 Locus

Tissue-specific insulator function at H19/Igf2 revealed by deletions at the imprinting control region

Humanized H19/Igf2 locus reveals diverged imprinting mechanism between mouse and human and reflects Silver–Russell syndrome phenotypes

Analysis of Sequence Upstream of the Endogenous H19 Gene Reveals Elements Both Essential and Dispensable for Imprinting

Deletion of the H19 differentially methylated domain results in loss of imprinted expression of H19 and Igf2

Nonrandom X chromosome inactivation is influenced by multiple regions on the murine X chromosome

Three-dimensional conformation at the H19/Igf2 locus supports a model of enhancer tracking

SnapShot: Imprinted Gene Clusters

Novel cis-regulatory function in ICR-mediated imprinted repression of H19

X-tra! X-tra! News from the Mouse X Chromosome

Akt1/PKBα Is Required for Normal Growth but Dispensable for Maintenance of Glucose Homeostasis in Mice

An N-Ethyl-N-Nitrosourea Mutagenesis Screen for Epigenetic Mutations in the Mouse

Mothers Setting Boundaries

Abstract 1936: Epigenetic challenges in derivation of the first cell-based model of Beckwith-Wiedemann Syndrome

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