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The Epstein-Barr virus encoded BART miRNAs potentiate tumor growth in vivo

PLoS pathogens, 2015-01, Vol.11 (1), p.e1004561

2015

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EBV microRNA BART 18-5p targets MAP3K2 to facilitate persistence in vivo by inhibiting viral replication in B cells

Proceedings of the National Academy of Sciences - PNAS, 2014-07, Vol.111 (30), p.11157-11162

2014

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The future of HIV testing in eastern and southern Africa: Broader scope, targeted services

PLoS medicine, 2023-03, Vol.20 (3), p.e1004182-e1004182

2023

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Impact on childhood mortality of interventions to improve drinking water, sanitation, and hygiene (WASH) to households: Systematic review and meta-analysis

PLoS medicine, 2023-04, Vol.20 (4), p.e1004215-e1004215

2023

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Neonatal and maternal adverse outcomes and exposure to nonsteroidal anti-inflammatory drugs during early pregnancy in South Korea: A nationwide cohort study

PLoS medicine, 2023-02, Vol.20 (2), p.e1004183-e1004183

2023

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Persistence of the Epstein–Barr Virus and the Origins of Associated Lymphomas

The New England journal of medicine, 2004-03, Vol.350 (13), p.1328-1337

2004

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A novel persistence associated EBV miRNA expression profile is disrupted in neoplasia

PLoS pathogens, 2011-08, Vol.7 (8), p.e1002193-e1002193

2011

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Health system assessment for access to care after injury in low- or middle-income countries: A mixed methods study from Northern Malawi

PLoS medicine, 2024-01, Vol.21 (1), p.e1004344-e1004344

2024

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Evaluation of a community health worker home visit intervention to improve child development in South Africa: A cluster-randomized controlled trial

PLoS medicine, 2023-04, Vol.20 (4), p.e1004222-e1004222

2023

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Functional delivery of viral miRNAs via exosomes

Proceedings of the National Academy of Sciences - PNAS, 2010-04, Vol.107 (14), p.6328-6333

2010

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Terminal Differentiation into Plasma Cells Initiates the Replicative Cycle of Epstein-Barr Virus In Vivo

Journal of Virology, 2005-01, Vol.79 (2), p.1296-1307

2005

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The pathogenesis of Epstein–Barr virus persistent infection

Current opinion in virology, 2013-06, Vol.3 (3), p.227-232

2013

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Epstein-Barr virus: exploiting the immune system

Nature reviews. Immunology, 2001-10, Vol.1 (1), p.75-82

Thorley-Lawson, David A

2001

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A multifactorial role for P. falciparum malaria in endemic Burkitt's lymphoma pathogenesis

PLoS pathogens, 2014-05, Vol.10 (5), p.e1004170

2014

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Comprehensive Profiling of Epstein-Barr Virus MicroRNAs in Nasopharyngeal Carcinoma

Journal of Virology, 2009-03, Vol.83 (5), p.2357-2367

2009

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The Intersection of Epstein-Barr Virus with the Germinal Center

Journal of Virology, 2009-04, Vol.83 (8), p.3968-3976

2009

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The Expression Pattern of Epstein-Barr Virus Latent Genes In Vivo Is Dependent upon the Differentiation Stage of the Infected B Cell

Immunity (Cambridge, Mass.), 2000-10, Vol.13 (4), p.497-506

2000

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EBV and Systemic Lupus Erythematosus: A New Perspective

The Journal of immunology (1950), 2005-06, Vol.174 (11), p.6599-6607

2005

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Persistence of Epstein-Barr Virus in Self-Reactive Memory B Cells

Journal of Virology, 2012-11, Vol.86 (22), p.12330-12340

2012

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Plasma Cell-Specific Transcription Factor XBP-1s Binds to and Transactivates the Epstein-Barr Virus BZLF1 Promoter

Journal of Virology, 2007-12, Vol.81 (24), p.13566-13577

2007

Volltextzugriff (PDF)

Germinal Center B Cells Latently Infected with Epstein-Barr Virus Proliferate Extensively but Do Not Increase in Number

Journal of Virology, 2010-01, Vol.84 (2), p.1158-1168

2010

Volltextzugriff (PDF)

Demonstration of the Burkitt's Lymphoma Epstein-Barr Virus Phenotype in Dividing Latently Infected Memory Cells in vivo

Proceedings of the National Academy of Sciences - PNAS, 2004-01, Vol.101 (1), p.239-244

2004

Volltextzugriff (PDF)

The cycle of EBV infection explains persistence, the sizes of the infected cell populations and which come under CTL regulation

PLoS pathogens, 2013-10, Vol.9 (10), p.e1003685-e1003685

2013

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Induction of platelet aggregation after a direct physical interaction with diesel exhaust particles

Journal of thrombosis and haemostasis, 2013-02, Vol.11 (2), p.325-334

2013

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The Epstein-Barr virus encoded BART miRNAs potentiate tumor growth in vivo

EBV microRNA BART 18-5p targets MAP3K2 to facilitate persistence in vivo by inhibiting viral replication in B cells

The future of HIV testing in eastern and southern Africa: Broader scope, targeted services

Impact on childhood mortality of interventions to improve drinking water, sanitation, and hygiene (WASH) to households: Systematic review and meta-analysis

Neonatal and maternal adverse outcomes and exposure to nonsteroidal anti-inflammatory drugs during early pregnancy in South Korea: A nationwide cohort study

Persistence of the Epstein–Barr Virus and the Origins of Associated Lymphomas

A novel persistence associated EBV miRNA expression profile is disrupted in neoplasia

Health system assessment for access to care after injury in low- or middle-income countries: A mixed methods study from Northern Malawi

Evaluation of a community health worker home visit intervention to improve child development in South Africa: A cluster-randomized controlled trial

Home working and social and mental wellbeing at different stages of the COVID-19 pandemic in the UK: Evidence from 7 longitudinal population surveys

Functional delivery of viral miRNAs via exosomes

Terminal Differentiation into Plasma Cells Initiates the Replicative Cycle of Epstein-Barr Virus In Vivo

The pathogenesis of Epstein–Barr virus persistent infection

Epstein-Barr virus: exploiting the immune system

A multifactorial role for P. falciparum malaria in endemic Burkitt's lymphoma pathogenesis

Comprehensive Profiling of Epstein-Barr Virus MicroRNAs in Nasopharyngeal Carcinoma

The Intersection of Epstein-Barr Virus with the Germinal Center

The Expression Pattern of Epstein-Barr Virus Latent Genes In Vivo Is Dependent upon the Differentiation Stage of the Infected B Cell

EBV and Systemic Lupus Erythematosus: A New Perspective

Persistence of Epstein-Barr Virus in Self-Reactive Memory B Cells

Plasma Cell-Specific Transcription Factor XBP-1s Binds to and Transactivates the Epstein-Barr Virus BZLF1 Promoter

Germinal Center B Cells Latently Infected with Epstein-Barr Virus Proliferate Extensively but Do Not Increase in Number

Demonstration of the Burkitt's Lymphoma Epstein-Barr Virus Phenotype in Dividing Latently Infected Memory Cells in vivo

The cycle of EBV infection explains persistence, the sizes of the infected cell populations and which come under CTL regulation

Induction of platelet aggregation after a direct physical interaction with diesel exhaust particles

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