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The blood-brain barrier is dysregulated in COVID-19 and serves as a CNS entry route for SARS-CoV-2

Stem cell reports, 2022-02, Vol.17 (2), p.307-320

2022

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Activity-Dependent Regulation of Distinct Transport and Cytoskeletal Remodeling Functions of the Dendritic Kinesin KIF21B

Neuron (Cambridge, Mass.), 2016-11, Vol.92 (4), p.857-872

2016

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Spastin depletion increases tubulin polyglutamylation and impairs kinesin-mediated neuronal transport, leading to working and associative memory deficits

PLoS biology, 2020-08, Vol.18 (8), p.e3000820-e3000820

2020

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Swimming against the Tide: Mobility of the Microtubule-Associated Protein Tau in Neurons

The Journal of neuroscience, 2007-09, Vol.27 (37), p.9916-9927

2007

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MARK/PAR1 Kinase Is a Regulator of Microtubule-Dependent Transport in Axons

The Journal of cell biology, 2004-10, Vol.167 (1), p.99-110

2004

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Neurobeachin and the Kinesin KIF21B Are Critical for Endocytic Recycling of NMDA Receptors and Regulate Social Behavior

Cell reports (Cambridge), 2018-05, Vol.23 (9), p.2705-2717

2018

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The kinesin Kif21b binds myosin Va and mediates changes in actin dynamics underlying homeostatic synaptic downscaling

Cell reports (Cambridge), 2023-07, Vol.42 (7), p.112743-112743, Article 112743

2023

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The Kinesin KIF21B Regulates Microtubule Dynamics and Is Essential for Neuronal Morphology, Synapse Function, and Learning and Memory

Cell reports (Cambridge), 2016-05, Vol.15 (5), p.968-977

2016

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The kinesin KIF21B participates in the cell surface delivery of γ2 subunit-containing GABAA receptors

European journal of cell biology, 2014-08, Vol.93 (8-9), p.338-346

2014

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Loss of Homeostatic Microglia Signature in Prion Diseases

Cells (Basel, Switzerland), 2022-09, Vol.11 (19), p.2948

2022

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Assessing and improving the validity of COVID-19 autopsy studies - A multicentre approach to establish essential standards for immunohistochemical and ultrastructural analyses

EBioMedicine, 2022-09, Vol.83, p.104193-104193, Article 104193

2022

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TRIM3 regulates the motility of the kinesin motor protein KIF21B

PloS one, 2013-09, Vol.8 (9), p.e75603-e75603

2013

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Muskelin regulates actin-dependent synaptic changes and intrinsic brain activity relevant to behavioral and cognitive processes

Communications biology, 2022-06, Vol.5 (1), p.589-589, Article 589

2022

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Aβ Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines

The Journal of neuroscience, 2010-09, Vol.30 (36), p.11938-11950

2010

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Missorting of Tau in Neurons Causes Degeneration of Synapses That Can Be Rescued by the Kinase MARK2/Par-1

The Journal of neuroscience, 2007-03, Vol.27 (11), p.2896-2907

2007

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TRPM4 cation channel mediates axonal and neuronal degeneration in experimental autoimmune encephalomyelitis and multiple sclerosis

Nature medicine, 2012-12, Vol.18 (12), p.1805-1811

2012

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Inefficient tissue immune response against MPXV in an immunocompromised mpox patient

Journal of medical virology, 2024-07, Vol.96 (7), p.e29811-n/a

2024

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Neuronal growth cone collapse triggers lateral extensions along trailing axons

Nature neuroscience, 1999-03, Vol.2 (3), p.254-259

1999

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A method for the generation of YAC transgenic mice by pronuclear microinjection

Nucleic acids research, 1993-10, Vol.21 (20), p.4783-4787

1993

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The Microtubule Severing Protein Katanin Regulates Proliferation of Neuronal Progenitors in Embryonic and Adult Neurogenesis

Scientific reports, 2019-11, Vol.9 (1), p.15940-12, Article 15940

2019

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Appearance of target-specific guidance information for regenerating axons after CNS lesions

Neuron (Cambridge, Mass.), 1993-11, Vol.11 (5), p.975-983

1993

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Aβ Oligomers Cause Localized Ca 2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines

The Journal of neuroscience, 2010-09, Vol.30 (36), p.11938-11950

2010

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Ab Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines

The Journal of neuroscience, 2010-09, Vol.30 (36), p.11938-11950

2010

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P2-135: Mechanisms of neuronal traffic inhibition by tau protein

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T410-T410

2008

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Inhibition of APP Trafficking by Tau Protein Does Not Increase the Generation of Amyloid‐β Peptides

Traffic (Copenhagen, Denmark), 2006-07, Vol.7 (7), p.873-888

2006

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The blood-brain barrier is dysregulated in COVID-19 and serves as a CNS entry route for SARS-CoV-2

Activity-Dependent Regulation of Distinct Transport and Cytoskeletal Remodeling Functions of the Dendritic Kinesin KIF21B

Spastin depletion increases tubulin polyglutamylation and impairs kinesin-mediated neuronal transport, leading to working and associative memory deficits

Swimming against the Tide: Mobility of the Microtubule-Associated Protein Tau in Neurons

MARK/PAR1 Kinase Is a Regulator of Microtubule-Dependent Transport in Axons

Neurobeachin and the Kinesin KIF21B Are Critical for Endocytic Recycling of NMDA Receptors and Regulate Social Behavior

The kinesin Kif21b binds myosin Va and mediates changes in actin dynamics underlying homeostatic synaptic downscaling

The Kinesin KIF21B Regulates Microtubule Dynamics and Is Essential for Neuronal Morphology, Synapse Function, and Learning and Memory

The kinesin KIF21B participates in the cell surface delivery of γ2 subunit-containing GABAA receptors

Loss of Homeostatic Microglia Signature in Prion Diseases

Assessing and improving the validity of COVID-19 autopsy studies - A multicentre approach to establish essential standards for immunohistochemical and ultrastructural analyses

TRIM3 regulates the motility of the kinesin motor protein KIF21B

Muskelin regulates actin-dependent synaptic changes and intrinsic brain activity relevant to behavioral and cognitive processes

Aβ Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines

Missorting of Tau in Neurons Causes Degeneration of Synapses That Can Be Rescued by the Kinase MARK2/Par-1

TRPM4 cation channel mediates axonal and neuronal degeneration in experimental autoimmune encephalomyelitis and multiple sclerosis

Inefficient tissue immune response against MPXV in an immunocompromised mpox patient

Neuronal growth cone collapse triggers lateral extensions along trailing axons

A method for the generation of YAC transgenic mice by pronuclear microinjection

The Microtubule Severing Protein Katanin Regulates Proliferation of Neuronal Progenitors in Embryonic and Adult Neurogenesis

Appearance of target-specific guidance information for regenerating axons after CNS lesions

Aβ Oligomers Cause Localized Ca 2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines

Ab Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines

P2-135: Mechanisms of neuronal traffic inhibition by tau protein

Inhibition of APP Trafficking by Tau Protein Does Not Increase the Generation of Amyloid‐β Peptides

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