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E2 proteins of high risk human papillomaviruses down-modulate STING and IFN-κ transcription in keratinocytes

PloS one, 2014-03, Vol.9 (3), p.e91473

2014

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HPV16 E2 Is an Immediate Early Marker of Viral Infection, Preceding E7 Expression in Precursor Structures of Cervical Carcinoma

Cancer research (Chicago, Ill.), 2010-07, Vol.70 (13), p.5316-5325

2010

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Transcriptional regulation of the papillomavirus oncogenes by cellular and viral transcription factors in cervical carcinoma

Virology (New York, N.Y.), 2009-02, Vol.384 (2), p.375-379

Thierry, Françoise

2009

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The Human Papillomavirus Type 18 E2 Protein Is a Cell Cycle-Dependent Target of the SCFSkp2 Ubiquitin Ligase

Journal of Virology, 2010-01, Vol.84 (1), p.437-444

2010

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The CXCL12/CXCR4 Signaling Pathway: A New Susceptibility Factor in Human Papillomavirus Pathogenesis

PLoS pathogens, 2016-12, Vol.12 (12), p.e1006039

2016

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HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma

Oncotarget, 2015-10, Vol.6 (33), p.34979-34991

2015

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Localization of HPV-18 E2 at mitochondrial membranes induces ROS release and modulates host cell metabolism

PloS one, 2013-09, Vol.8 (9), p.e75625

2013

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Tumor suppressor or oncogene? A critical role of the human papillomavirus (HPV) E2 protein in cervical cancer progression

American journal of cancer research, 2011-01, Vol.1 (3), p.373-389

2011

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Abstract LB-268: Loss of HPV16 E2 protein expression and transcriptional modulation of cellular and viral genes correlate with progression to cervical carcinoma

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.LB-268-LB-268

Thierry, Francoise

2012

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Human papillomavirus proteins as prospective therapeutic targets

Microbial pathogenesis, 2013-05, Vol.58, p.55-65

2013

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Mapping of HPV transcripts in four human cervical lesions using RNAseq suggests quantitative rearrangements during carcinogenic progression

Virology (New York, N.Y.), 2014-08, Vol.462, p.14-24

2014

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Genomic organization of amplified MYC genes suggests distinct mechanisms of amplification in tumorigenesis

Cancer research (Chicago, Ill.), 2005-02, Vol.65 (4), p.1174-1179

2005

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Genome-wide analysis of high risk human papillomavirus E2 proteins in human primary keratinocytes

Genomics data, 2014-12, Vol.2 (C), p.147-149

2014

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A pivotal role for CXCL12 signaling in HPV-mediated transformation of keratinocytes: clues to understanding HPV-pathogenesis in WHIM syndrome

Cell host & microbe, 2010-12, Vol.8 (6), p.523-533

2010

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Nucleo-cytoplasmic Shuttling of High Risk Human Papillomavirus E2 Proteins Induces Apoptosis

The Journal of biological chemistry, 2005-10, Vol.280 (43), p.36088-36098

2005

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Transcriptional control of late differentiation in human keratinocytes by TAp63 and Notch

Experimental dermatology, 2015-10, Vol.24 (10), p.754-760

2015

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The human papillomavirus E6 oncogene represses a cell adhesion pathway and disrupts focal adhesion through degradation of TAp63β upon transformation

PLoS pathogens, 2011-09, Vol.7 (9), p.e1002256

2011

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A New E6/P63 Pathway, Together with a Strong E7/E2F Mitotic Pathway, Modulates the Transcriptome in Cervical Cancer Cells

Journal of Virology, 2007-09, Vol.81 (17), p.9368-9376

2007

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Transcription-independent triggering of the extrinsic pathway of apoptosis by human papillomavirus 18 E2 protein

Oncogene, 2003-01, Vol.22 (2), p.168-175

2003

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Monitoring Gender Data and Evaluating Differential Effects to Reduce Inequality

Advances in gender research, 2019, Vol.28, p.79-98

Anne-Françoise Thierry

2019

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A genomic approach reveals a novel mitotic pathway in papillomavirus carcinogenesis

Cancer research (Chicago, Ill.), 2004-02, Vol.64 (3), p.895-903

2004

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E2 Proteins of High Risk Human Papillomaviruses Down-Modulate STING and IFN-[kappa] Transcription in Keratinocytes

PloS one, 2014-03, Vol.9 (3), p.e91473

2014

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High-Risk But Not Low-Risk HPV E2 Proteins Bind to the APC Activators Cdh1 and Cdc20 and Cause Genomic Instability

Cell cycle (Georgetown, Tex.), 2005-11, Vol.4 (11), p.1608-1615

2005

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Frequent Alterations of the β-Catenin Protein in Cancer of the Uterine Cervix

Tumor biology, 2002-01, Vol.23 (1), p.45-53

2002

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Stability of the Human Papillomavirus Type 18 E2 Protein Is Regulated by a Proteasome Degradation Pathway through Its Amino-Terminal Transactivation Domain

Journal of Virology, 2001-08, Vol.75 (16), p.7244-7251

2001

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E2 proteins of high risk human papillomaviruses down-modulate STING and IFN-κ transcription in keratinocytes

HPV16 E2 Is an Immediate Early Marker of Viral Infection, Preceding E7 Expression in Precursor Structures of Cervical Carcinoma

Transcriptional regulation of the papillomavirus oncogenes by cellular and viral transcription factors in cervical carcinoma

The Human Papillomavirus Type 18 E2 Protein Is a Cell Cycle-Dependent Target of the SCFSkp2 Ubiquitin Ligase

The CXCL12/CXCR4 Signaling Pathway: A New Susceptibility Factor in Human Papillomavirus Pathogenesis

HPV16-E2 induces prophase arrest and activates the cellular DNA damage response in vitro and in precursor lesions of cervical carcinoma

Localization of HPV-18 E2 at mitochondrial membranes induces ROS release and modulates host cell metabolism

Tumor suppressor or oncogene? A critical role of the human papillomavirus (HPV) E2 protein in cervical cancer progression

Abstract LB-268: Loss of HPV16 E2 protein expression and transcriptional modulation of cellular and viral genes correlate with progression to cervical carcinoma

Human papillomavirus proteins as prospective therapeutic targets

Mapping of HPV transcripts in four human cervical lesions using RNAseq suggests quantitative rearrangements during carcinogenic progression

Genomic organization of amplified MYC genes suggests distinct mechanisms of amplification in tumorigenesis

Genome-wide analysis of high risk human papillomavirus E2 proteins in human primary keratinocytes

A pivotal role for CXCL12 signaling in HPV-mediated transformation of keratinocytes: clues to understanding HPV-pathogenesis in WHIM syndrome

Nucleo-cytoplasmic Shuttling of High Risk Human Papillomavirus E2 Proteins Induces Apoptosis

Transcriptional control of late differentiation in human keratinocytes by TAp63 and Notch

The human papillomavirus E6 oncogene represses a cell adhesion pathway and disrupts focal adhesion through degradation of TAp63β upon transformation

A New E6/P63 Pathway, Together with a Strong E7/E2F Mitotic Pathway, Modulates the Transcriptome in Cervical Cancer Cells

Transcription-independent triggering of the extrinsic pathway of apoptosis by human papillomavirus 18 E2 protein

Monitoring Gender Data and Evaluating Differential Effects to Reduce Inequality

A genomic approach reveals a novel mitotic pathway in papillomavirus carcinogenesis

E2 Proteins of High Risk Human Papillomaviruses Down-Modulate STING and IFN-[kappa] Transcription in Keratinocytes

High-Risk But Not Low-Risk HPV E2 Proteins Bind to the APC Activators Cdh1 and Cdc20 and Cause Genomic Instability

Frequent Alterations of the β-Catenin Protein in Cancer of the Uterine Cervix

Stability of the Human Papillomavirus Type 18 E2 Protein Is Regulated by a Proteasome Degradation Pathway through Its Amino-Terminal Transactivation Domain

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