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Die Bedeutung der Selbstabwertung mit negativen Emotionen – Bericht aus dem Praxisalltag

Balint-Journal : Zeitschrift der Deutschen Balint-Gesellschaft, 2016-07, Vol.17 (2), p.41-47

Thierfelder, L

2016

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Improvement of fractional flow reserve and collateral flow by treatment with external counterpulsation (Art.Net.-2 Trial)

European journal of clinical investigation, 2009-10, Vol.39 (10), p.866-875

2009

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Genetische Aspekte der Arrhythmieentstehung

Clinical research in cardiology, 2000-04, Vol.89, p.III1-III5

Thierfelder, L

2000

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Preserved heart function after left ventricular pressure overload in adult mice subjected to neonatal cardiac hypoplasia

Journal of developmental origins of health and disease, 2018-02, Vol.9 (1), p.112-124

2018

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Mutations in the desmosomal protein plakophilin-2 are common in arrhythmogenic right ventricular cardiomyopathy

Nature genetics, 2004-11, Vol.36 (11), p.1162-1164

2004

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P448Dietary protein restriction throughout intrauterine development and postnatal life alters myocardial tissue composition but not left ventricular function in the adult mouse heart

Cardiovascular research, 2018-04, Vol.114 (suppl_1), p.S107-S107

2018

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Mutant Desmocollin-2 Causes Arrhythmogenic Right Ventricular Cardiomyopathy

American journal of human genetics, 2006-12, Vol.79 (6), p.1081-1088

2006

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Mutations of TTN , encoding the giant muscle filament titin, cause familial dilated cardiomyopathy

Nature genetics, 2002-02, Vol.30 (2), p.201-204

2002

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Prenatal mTORC1 Inhibition causes Fetal Growth Restriction and Impairs Heart Development

The Thoracic and cardiovascular surgeon, 2016, Vol.64 (S 02)

2016

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Cardiac magnetic field map topology quantified by Kullback-Leibler entropy identifies patients with hypertrophic cardiomyopathy

Chaos (Woodbury, N.Y.), 2007-03, Vol.17 (1), p.015118-015118-10

2007

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Preserved Heart Function after Left Ventricular Pressure Overload in Adult Mice Subjected to Neonatal Cardiac Hypoplasia

The Thoracic and cardiovascular surgeon, 2017, Vol.65 (S 01), p.S1-S110

2017

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The impact of implantable cardioverter-defibrillator therapy on survival in autosomal-dominant arrhythmogenic right ventricular cardiomyopathy (ARVD5)

Journal of the American College of Cardiology, 2005-02, Vol.45 (3), p.400-408

2005

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A Familial Hypertrophic Cardiomyopathy Locus Maps to Chromosome 15q2

Proceedings of the National Academy of Sciences - PNAS, 1993-07, Vol.90 (13), p.6270-6274

1993

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Familial Hypertrophic cardiomyopathy with Wolff-Parkinson-White syndrome maps to a locus on chromosome 7q3

The Journal of clinical investigation, 1995-09, Vol.96 (3), p.1216-1220

1995

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P313Embryonic cardiomyocytes can orchestrate various cell protective mechanisms to survive metabolic stress

Cardiovascular research, 2014-07, Vol.103 (suppl 1), p.S57-S57

2014

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Isolated noncompaction of the left ventricular myocardium in the adult is an autosomal dominant disorder in the majority of patients

American journal of medical genetics, 2003-06, Vol.119A (2), p.162-167

2003

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Sporadic hypertrophic cardiomyopathy due to de novo myosin mutations

The Journal of clinical investigation, 1992-11, Vol.90 (5), p.1666-1671

1992

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Mutation detection by cycle sequencing

Current Protocols in Human Genetics, 2001-05, Vol.Chapter 7 (1), p.Unit 7.7-Unit 7.7

Thierfelder, L

2001

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Novel gene locus for autosomal dominant left ventricular noncompaction maps to chromosome 11p15

Circulation (New York, N.Y.), 2004-06, Vol.109 (22), p.2720-2723

2004

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Identification of a novel frameshift mutation in the giant muscle filament titin in a large Australian family with dilated cardiomyopathy

Journal of molecular medicine (Berlin, Germany), 2006-06, Vol.84 (6), p.478-483

2006

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Mutations in the cardiac myosin binding protein-C gene on chromosome 11 cause familial hypertrophic cardiomyopathy

Nature genetics, 1995-12, Vol.11 (4), p.434-437

1995

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Neonatal Cardiac Hypoplasia Alters Postnatal Growth and Stress Response in the Murine Heart

The Thoracic and cardiovascular surgeon, 2015, Vol.63 (S 03)

2015

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α-tropomyosin and cardiac troponin T mutations cause familial hypertrophic cardiomyopathy: A disease of the sarcomere

Cell, 1994-06, Vol.77 (5), p.701-712

1994

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Mutations in the Genes for Cardiac Troponin T and α-Tropomyosin in Hypertrophic Cardiomyopathy

The New England journal of medicine, 1995-04, Vol.332 (16), p.1058-1065

1995

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COMMENT ON ‘DE ROO ET AL. (2019). ON-FARM TRIALS FOR DEVELOPMENT IMPACT? THE ORGANIZATION OF RESEARCH AND THE SCALING OF AGRICULTURAL TECHNOLOGIES’

Experimental agriculture, 2019-04, Vol.55 (2), p.185-194

2019

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Die Bedeutung der Selbstabwertung mit negativen Emotionen – Bericht aus dem Praxisalltag

Improvement of fractional flow reserve and collateral flow by treatment with external counterpulsation (Art.Net.-2 Trial)

Genetische Aspekte der Arrhythmieentstehung

Preserved heart function after left ventricular pressure overload in adult mice subjected to neonatal cardiac hypoplasia

Mutations in the desmosomal protein plakophilin-2 are common in arrhythmogenic right ventricular cardiomyopathy

P448Dietary protein restriction throughout intrauterine development and postnatal life alters myocardial tissue composition but not left ventricular function in the adult mouse heart

Mutant Desmocollin-2 Causes Arrhythmogenic Right Ventricular Cardiomyopathy

Mutations of TTN , encoding the giant muscle filament titin, cause familial dilated cardiomyopathy

Prenatal mTORC1 Inhibition causes Fetal Growth Restriction and Impairs Heart Development

Cardiac magnetic field map topology quantified by Kullback-Leibler entropy identifies patients with hypertrophic cardiomyopathy

Preserved Heart Function after Left Ventricular Pressure Overload in Adult Mice Subjected to Neonatal Cardiac Hypoplasia

The impact of implantable cardioverter-defibrillator therapy on survival in autosomal-dominant arrhythmogenic right ventricular cardiomyopathy (ARVD5)

A Familial Hypertrophic Cardiomyopathy Locus Maps to Chromosome 15q2

Familial Hypertrophic cardiomyopathy with Wolff-Parkinson-White syndrome maps to a locus on chromosome 7q3

P313Embryonic cardiomyocytes can orchestrate various cell protective mechanisms to survive metabolic stress

Isolated noncompaction of the left ventricular myocardium in the adult is an autosomal dominant disorder in the majority of patients

Sporadic hypertrophic cardiomyopathy due to de novo myosin mutations

Mutation detection by cycle sequencing

Novel gene locus for autosomal dominant left ventricular noncompaction maps to chromosome 11p15

Identification of a novel frameshift mutation in the giant muscle filament titin in a large Australian family with dilated cardiomyopathy

Mutations in the cardiac myosin binding protein-C gene on chromosome 11 cause familial hypertrophic cardiomyopathy

Neonatal Cardiac Hypoplasia Alters Postnatal Growth and Stress Response in the Murine Heart

α-tropomyosin and cardiac troponin T mutations cause familial hypertrophic cardiomyopathy: A disease of the sarcomere

Mutations in the Genes for Cardiac Troponin T and α-Tropomyosin in Hypertrophic Cardiomyopathy

COMMENT ON ‘DE ROO ET AL. (2019). ON-FARM TRIALS FOR DEVELOPMENT IMPACT? THE ORGANIZATION OF RESEARCH AND THE SCALING OF AGRICULTURAL TECHNOLOGIES’

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