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Targeting MYCN in Pediatric and Adult Cancers

Frontiers in oncology, 2021-02, Vol.10, p.623679-623679

2021

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Cancer Stem Cells and Neuroblastoma: Characteristics and Therapeutic Targeting Options

Frontiers in endocrinology (Lausanne), 2019-11, Vol.10, p.782-782

2019

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Combining selinexor with alisertib to target the p53 pathway in neuroblastoma

Neoplasia (New York, N.Y.), 2022-04, Vol.26, p.100776-100776, Article 100776

2022

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MYCN drives oncogenesis by cooperating with the histone methyltransferase G9a and the WDR5 adaptor to orchestrate global gene transcription

PLoS biology, 2024-03, Vol.22 (3), p.e3002240-e3002240

2024

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Lineage specific transcription factor waves reprogram neuroblastoma from self-renewal to differentiation

Nature communications, 2024-04, Vol.15 (1), p.3432-3432, Article 3432

2024

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Novel small molecule DMAMCL induces differentiation in rhabdomyosarcoma by downregulating of DLL1

Biomedicine & pharmacotherapy, 2024-05, Vol.174, p.116562-116562, Article 116562

2024

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CASZ1 induces skeletal muscle and rhabdomyosarcoma differentiation through a feed-forward loop with MYOD and MYOG

Nature communications, 2020-02, Vol.11 (1), p.911-16, Article 911

2020

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Epigenetic siRNA and Chemical Screens Identify SETD8 Inhibition as a Therapeutic Strategy for p53 Activation in High-Risk Neuroblastoma

Cancer cell, 2017-01, Vol.31 (1), p.50-63

2017

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EZH2 Mediates Epigenetic Silencing of Neuroblastoma Suppressor Genes CASZ1, CLU, RUNX3, and NGFR

Cancer research (Chicago, Ill.), 2012-01, Vol.72 (1), p.315-324

2012

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Loss of CASZ1 tumor suppressor linked to oncogenic subversion of neuroblastoma core regulatory circuitry

Cell death & disease, 2022-10, Vol.13 (10), p.871-12, Article 871

2022

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The anti-tumor growth effect of a novel agent DMAMCL in rhabdomyosarcoma in vitro and in vivo

Journal of experimental & clinical cancer research, 2019-03, Vol.38 (1), p.118-17, Article 118

2019

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Essential Role of the Zinc Finger Transcription Factor Casz1 for Mammalian Cardiac Morphogenesis and Development

The Journal of biological chemistry, 2014-10, Vol.289 (43), p.29801-29816

2014

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PI3K and MAPK pathways mediate the BDNF/TrkB-increased metastasis in neuroblastoma

Tumor biology, 2016-12, Vol.37 (12), p.16227-16236

2016

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Combination of Rapamycin and MK-2206 Induced Cell Death via Autophagy and Necroptosis in MYCN-Amplified Neuroblastoma Cell Lines

Frontiers in pharmacology, 2020-02, Vol.11, p.31-31

2020

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CASZ1b, the short isoform of CASZ1 gene, coexpresses with CASZ1a during neurogenesis and suppresses neuroblastoma cell growth

PloS one, 2011-04, Vol.6 (4), p.e18557-e18557

2011

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Histone Methyltransferases G9a/Ehmt2 and GLP/Ehmt1 Are Associated with Cell Viability and Poorer Prognosis in Neuroblastoma and Ewing Sarcoma

International journal of molecular sciences, 2023-10, Vol.24 (20), p.15242

2023

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Corrigendum: Downregulation of ATXN3 enhances the sensitivity to AKT inhibitors (Perifosine or MK-2206), but decreases the sensitivity to chemotherapeutic drugs (etoposide or cisplatin) in neuroblastoma cells

Frontiers in oncology, 2022-08, Vol.12, p.984514-984514

2022

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Downregulation of ATXN3 Enhances the Sensitivity to AKT Inhibitors (Perifosine or MK-2206), but Decreases the Sensitivity to Chemotherapeutic Drugs (Etoposide or Cisplatin) in Neuroblastoma Cells

Frontiers in oncology, 2021-07, Vol.11, p.686898-686898

2021

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Perifosine‐induced inhibition of akt attenuates brain‐derived neurotrophic factor/TrkB‐induced chemoresistance in neuroblastoma in vivo

Cancer, 2011-12, Vol.117 (23), p.5412-5422

2011

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Antitumor effects of the small molecule DMAMCL in neuroblastoma via suppressing aerobic glycolysis and targeting PFKL

Cancer cell international, 2021-11, Vol.21 (1), p.1-619, Article 619

2021

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Preclinical testing of chimeric antigen receptor T cells in neuroblastoma mouse models

STAR protocols, 2021-12, Vol.2 (4), p.100942-100942, Article 100942

2021

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Unraveling the Enigmatic Origin of Neuroblastoma

Cancer cell, 2020-11, Vol.38 (5), p.618-620

2020

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Neuroblastoma Cells Isolated from Bone Marrow Metastases Contain a Naturally Enriched Tumor-Initiating Cell

Cancer research (Chicago, Ill.), 2007-12, Vol.67 (23), p.11234-11243

2007

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Preclinical Therapeutic Efficacy of RAF/MEK/ERK and IGF1R/AKT/mTOR Inhibition in Neuroblastoma

Cancers, 2024-06, Vol.16 (13), p.2320

2024

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Identification of Casz1 as a Regulatory Protein Controlling T Helper Cell Differentiation, Inflammation, and Immunity

Frontiers in immunology, 2018-02, Vol.9, p.184-184

2018

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Targeting MYCN in Pediatric and Adult Cancers

Cancer Stem Cells and Neuroblastoma: Characteristics and Therapeutic Targeting Options

Combining selinexor with alisertib to target the p53 pathway in neuroblastoma

MYCN drives oncogenesis by cooperating with the histone methyltransferase G9a and the WDR5 adaptor to orchestrate global gene transcription

Lineage specific transcription factor waves reprogram neuroblastoma from self-renewal to differentiation

Novel small molecule DMAMCL induces differentiation in rhabdomyosarcoma by downregulating of DLL1

CASZ1 induces skeletal muscle and rhabdomyosarcoma differentiation through a feed-forward loop with MYOD and MYOG

Epigenetic siRNA and Chemical Screens Identify SETD8 Inhibition as a Therapeutic Strategy for p53 Activation in High-Risk Neuroblastoma

EZH2 Mediates Epigenetic Silencing of Neuroblastoma Suppressor Genes CASZ1, CLU, RUNX3, and NGFR

Loss of CASZ1 tumor suppressor linked to oncogenic subversion of neuroblastoma core regulatory circuitry

The anti-tumor growth effect of a novel agent DMAMCL in rhabdomyosarcoma in vitro and in vivo

Essential Role of the Zinc Finger Transcription Factor Casz1 for Mammalian Cardiac Morphogenesis and Development

PI3K and MAPK pathways mediate the BDNF/TrkB-increased metastasis in neuroblastoma

Combination of Rapamycin and MK-2206 Induced Cell Death via Autophagy and Necroptosis in MYCN-Amplified Neuroblastoma Cell Lines

CASZ1b, the short isoform of CASZ1 gene, coexpresses with CASZ1a during neurogenesis and suppresses neuroblastoma cell growth

Histone Methyltransferases G9a/Ehmt2 and GLP/Ehmt1 Are Associated with Cell Viability and Poorer Prognosis in Neuroblastoma and Ewing Sarcoma

Corrigendum: Downregulation of ATXN3 enhances the sensitivity to AKT inhibitors (Perifosine or MK-2206), but decreases the sensitivity to chemotherapeutic drugs (etoposide or cisplatin) in neuroblastoma cells

Downregulation of ATXN3 Enhances the Sensitivity to AKT Inhibitors (Perifosine or MK-2206), but Decreases the Sensitivity to Chemotherapeutic Drugs (Etoposide or Cisplatin) in Neuroblastoma Cells

Perifosine‐induced inhibition of akt attenuates brain‐derived neurotrophic factor/TrkB‐induced chemoresistance in neuroblastoma in vivo

Antitumor effects of the small molecule DMAMCL in neuroblastoma via suppressing aerobic glycolysis and targeting PFKL

Preclinical testing of chimeric antigen receptor T cells in neuroblastoma mouse models

Unraveling the Enigmatic Origin of Neuroblastoma

Neuroblastoma Cells Isolated from Bone Marrow Metastases Contain a Naturally Enriched Tumor-Initiating Cell

Preclinical Therapeutic Efficacy of RAF/MEK/ERK and IGF1R/AKT/mTOR Inhibition in Neuroblastoma

Identification of Casz1 as a Regulatory Protein Controlling T Helper Cell Differentiation, Inflammation, and Immunity

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