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The prolyl isomerase Pin1 modulates development of CD8+ cDC in mice

PloS one, 2012-01, Vol.7 (1), p.e29808-e29808

2012

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Phosphorylation of RelA/p65 on Serine 536 Defines an IκBα-independent NF-κB Pathway

The Journal of biological chemistry, 2005-10, Vol.280 (41), p.34538-34547

2005

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Traf1 Induction and Protection from Tumor Necrosis Factor by Nuclear Factor-κB p65 Is Independent of Serine 536 Phosphorylation

Cancer research (Chicago, Ill.), 2007-12, Vol.67 (23), p.11218-11225

2007

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Phosphorylation of RelA/p65 on serine 536 defines an I{kappa}B{alpha}-independent NF-{kappa}B pathway

The Journal of biological chemistry, 2005-10, Vol.280 (41), p.34538-34547

2005

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Phosphorylation of RelA/p65 on Serine 536 Defines an IÎºBÎ±-independent NF-ÎºB Pathway

The Journal of biological chemistry, 2005-10, Vol.280 (41), p.34538

2005

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Roles for Pin1 in Modulating Cells of the Innate Immune System

Barberi, Theresa J

2011

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Traf1 induction and protection from tumor necrosis factor by nuclear factor-kappaB p65 is independent of serine 536 phosphorylation

Cancer research (Chicago, Ill.), 2007-12, Vol.67 (23), p.11218-11225

2007

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CCAAT/Enhancer binding protein β controls androgen-deprivation-induced senescence in prostate cancer cells

Oncogene, 2015-11, Vol.34 (48), p.5912-5922

2015

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Absence of myeloid Klf4 reduces prostate cancer growth with pro-atherosclerotic activation of tumor myeloid cells and infiltration of CD8 T cells

PloS one, 2018-01, Vol.13 (1), p.e0191188-e0191188

2018

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Abstract 4063: Adoptive transfer of NF-kB p50 knockout immature myeloid cells shows a trend towards slower glioblastoma tumor growth in an orthotopic mouse model

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.4063-4063

2023

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NF-κB p50-deficient immature myeloid cell (p50-IMC) adoptive transfer slows the growth of murine prostate and pancreatic ductal carcinoma

Journal for immunotherapy of cancer, 2020-01, Vol.8 (1), p.e000244

2020

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C/EBPβ regulates sensitivity to bortezomib in prostate cancer cells by inducing REDD1 and autophagosome-lysosome fusion

Cancer letters, 2016-05, Vol.375 (1), p.152-161

2016

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Absence of host NF-κB p50 induces murine glioblastoma tumor regression, increases survival, and decreases T-cell induction of tumor-associated macrophage M2 polarization

Cancer Immunology, Immunotherapy, 2018-10, Vol.67 (10), p.1491-1503

2018

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Absence of host NF-κΒ p50 induces murine glioblastoma tumor regression, increases survival, and decreases T cell induction of tumor-associated macrophage M2 polarization

Cancer immunology, immunotherapy : CII, 2018-07, Vol.67 (10), p.1491-1503

2018

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Abstract B68: NF-kB p50-deficient immature myeloid cell (p50-IMC) adoptive transfer slows the growth of murine prostate and pancreatic ductal carcinoma

Cancer immunology research, 2020-03, Vol.8 (3_Supplement), p.B68-B68

2020

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C/EBP[beta] regulates sensitivity to bortezomib in prostate cancer cells by inducing REDD1 and autophagosome-lysosome fusion

Cancer letters, 2016-05, Vol.375 (1), p.152

2016

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The prolyl isomerase Pin1 modulates development of CD8+ cDC in mice

Phosphorylation of RelA/p65 on Serine 536 Defines an IκBα-independent NF-κB Pathway

Traf1 Induction and Protection from Tumor Necrosis Factor by Nuclear Factor-κB p65 Is Independent of Serine 536 Phosphorylation

Phosphorylation of RelA/p65 on serine 536 defines an I{kappa}B{alpha}-independent NF-{kappa}B pathway

Phosphorylation of RelA/p65 on Serine 536 Defines an IÎºBÎ±-independent NF-ÎºB Pathway

Roles for Pin1 in Modulating Cells of the Innate Immune System

Traf1 induction and protection from tumor necrosis factor by nuclear factor-kappaB p65 is independent of serine 536 phosphorylation

CCAAT/Enhancer binding protein β controls androgen-deprivation-induced senescence in prostate cancer cells

Absence of myeloid Klf4 reduces prostate cancer growth with pro-atherosclerotic activation of tumor myeloid cells and infiltration of CD8 T cells

Abstract 4063: Adoptive transfer of NF-kB p50 knockout immature myeloid cells shows a trend towards slower glioblastoma tumor growth in an orthotopic mouse model

NF-κB p50-deficient immature myeloid cell (p50-IMC) adoptive transfer slows the growth of murine prostate and pancreatic ductal carcinoma

C/EBPβ regulates sensitivity to bortezomib in prostate cancer cells by inducing REDD1 and autophagosome-lysosome fusion

Absence of host NF-κB p50 induces murine glioblastoma tumor regression, increases survival, and decreases T-cell induction of tumor-associated macrophage M2 polarization

Absence of host NF-κΒ p50 induces murine glioblastoma tumor regression, increases survival, and decreases T cell induction of tumor-associated macrophage M2 polarization

Abstract B68: NF-kB p50-deficient immature myeloid cell (p50-IMC) adoptive transfer slows the growth of murine prostate and pancreatic ductal carcinoma

C/EBP[beta] regulates sensitivity to bortezomib in prostate cancer cells by inducing REDD1 and autophagosome-lysosome fusion

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