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Defects in Galactose Metabolism and Glycoconjugate Biosynthesis in a UDP-Glucose Pyrophosphorylase-Deficient Cell Line Are Reversed by Adding Galactose to the Growth Medium

International journal of molecular sciences, 2020-03, Vol.21 (6), p.2028

2020

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Myc is required for activation of the ATM-dependent checkpoints in response to DNA damage

PloS one, 2010-01, Vol.5 (1), p.e8924

2010

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The Haemophilus ducreyi cytolethal distending toxin induces DNA double‐strand breaks and promotes ATM‐dependent activation of RhoA

Cellular microbiology, 2003-10, Vol.5 (10), p.695-707

2003

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Cellular internalization of cytolethal distending toxin: a new end to a known pathway

Cellular microbiology, 2005-07, Vol.7 (7), p.921-934

2005

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The Haemophilus ducreyi Cytolethal Distending Toxin Induces Cell Cycle Arrest and Apoptosis via the DNA Damage Checkpoint Pathways

The Journal of biological chemistry, 2001-02, Vol.276 (7), p.5296-5302

2001

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The Haemophilus ducreyi cytolethal distending toxin activates sensors of DNA damage and repair complexes in proliferating and non‐proliferating cells

Cellular microbiology, 2002-02, Vol.4 (2), p.87-99

2002

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Cellular Internalization of Cytolethal Distending Toxin from Haemophilus ducreyi

Infection and Immunity, 2000-12, Vol.68 (12), p.6903-6911

2000

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A Cellular Deficiency of Gangliosides Causes Hypersensitivity to Clostridium perfringens Phospholipase C

The Journal of biological chemistry, 2005-07, Vol.280 (29), p.26680-26689

2005

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R-Ras Glucosylation and Transient RhoA Activation Determine the Cytopathic Effect Produced by Toxin B Variants from Toxin A-negative Strains of Clostridium difficile

The Journal of biological chemistry, 2003-03, Vol.278 (10), p.7956-7963

2003

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A Cellular UDP-glucose Deficiency Causes Overexpression of Glucose/Oxygen-regulated Proteins Independent of the Endoplasmic Reticulum Stress Elements

The Journal of biological chemistry, 2004-05, Vol.279 (21), p.21724-21731

2004

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Divergent Roles for Ras and Rap in the Activation of p38 Mitogen-activated Protein Kinase by Interleukin-1

The Journal of biological chemistry, 2000-03, Vol.275 (11), p.7818-7825

2000

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Ras, Rap, and Rac Small GTP-binding Proteins Are Targets for Clostridium sordellii Lethal Toxin Glucosylation

The Journal of biological chemistry, 1996-04, Vol.271 (17), p.10217-10224

1996

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Effects of Clostridium perfringens phospholipase C in mammalian cells

Anaerobe, 2004-04, Vol.10 (2), p.115-123

2004

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Cellular UDP-Glucose Deficiency Caused by a Single Point Mutation in the UDP-Glucose Pyrophosphorylase Gene

The Journal of biological chemistry, 1997-09, Vol.272 (38), p.23784-23791

1997

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The projection structure of Perfringolysin O ( Clostridium perfringens θ-toxin)

FEBS letters, 1993-03, Vol.319 (1), p.125-127

1993

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Cytolethal distending toxins

Reviews of physiology, biochemistry and pharmacology, 2005-01, Vol.152, p.111

2005

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Is there a risk of cancer development after Campylobacter infection?

Scandinavian journal of gastroenterology, 2010-08, Vol.45 (7-8), p.893-897

2010

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A novel mode of translocation for cytolethal distending toxin

Biochimica et biophysica acta, 2009-03, Vol.1793 (3), p.489-495

2009

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UDP-Glucose Deficiency in a Mutant Cell Line Protects against Glucosyltransferase Toxins from Clostridium difficile and Clostridium sordellii

The Journal of biological chemistry, 1996-03, Vol.271 (12), p.6925-6932

1996

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UDP-glucose Deficiency Causes Hypersensitivity to the Cytotoxic Effect of Clostridium perfringens Phospholipase C

The Journal of biological chemistry, 1998-09, Vol.273 (38), p.24433-24438

1998

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The cytolethal distending toxins induce DNA damage and cell cycle arrest

Toxicon (Oxford), 2001-11, Vol.39 (11), p.1729-1736

2001

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Defects in galactose metabolism and glycoconjugate biosynthesis in UDP‐glucose pyrophosphorylase‐deficient fibroblasts are reversed by supplementing the cell growth medium with galactose

The FASEB journal, 2012-04, Vol.26 (S1)

2012

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GTPases of the Rho Subfamily Are Required for Brucella abortus Internalization in Nonprofessional Phagocytes

The Journal of biological chemistry, 2001-11, Vol.276 (48), p.44435-44443

2001

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Large clostridial cytotoxins — a family of glycosyltransferases modifying small GTP-binding proteins

Trends in microbiology (Regular ed.), 1996-10, Vol.4 (10), p.375-382

1996

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The cytolethal distending toxin from the chancroid bacterium Haemophilus ducreyi induces cell-cycle arrest in the G2 phase

The Journal of clinical investigation, 1999-01, Vol.103 (1), p.107-115

1999

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Defects in Galactose Metabolism and Glycoconjugate Biosynthesis in a UDP-Glucose Pyrophosphorylase-Deficient Cell Line Are Reversed by Adding Galactose to the Growth Medium

Myc is required for activation of the ATM-dependent checkpoints in response to DNA damage

The Haemophilus ducreyi cytolethal distending toxin induces DNA double‐strand breaks and promotes ATM‐dependent activation of RhoA

Cellular internalization of cytolethal distending toxin: a new end to a known pathway

The Haemophilus ducreyi Cytolethal Distending Toxin Induces Cell Cycle Arrest and Apoptosis via the DNA Damage Checkpoint Pathways

The Haemophilus ducreyi cytolethal distending toxin activates sensors of DNA damage and repair complexes in proliferating and non‐proliferating cells

Cellular Internalization of Cytolethal Distending Toxin from Haemophilus ducreyi

A Cellular Deficiency of Gangliosides Causes Hypersensitivity to Clostridium perfringens Phospholipase C

R-Ras Glucosylation and Transient RhoA Activation Determine the Cytopathic Effect Produced by Toxin B Variants from Toxin A-negative Strains of Clostridium difficile

A Cellular UDP-glucose Deficiency Causes Overexpression of Glucose/Oxygen-regulated Proteins Independent of the Endoplasmic Reticulum Stress Elements

Divergent Roles for Ras and Rap in the Activation of p38 Mitogen-activated Protein Kinase by Interleukin-1

Ras, Rap, and Rac Small GTP-binding Proteins Are Targets for Clostridium sordellii Lethal Toxin Glucosylation

Effects of Clostridium perfringens phospholipase C in mammalian cells

Cellular UDP-Glucose Deficiency Caused by a Single Point Mutation in the UDP-Glucose Pyrophosphorylase Gene

The projection structure of Perfringolysin O ( Clostridium perfringens θ-toxin)

Cytolethal distending toxins

Is there a risk of cancer development after Campylobacter infection?

A novel mode of translocation for cytolethal distending toxin

UDP-Glucose Deficiency in a Mutant Cell Line Protects against Glucosyltransferase Toxins from Clostridium difficile and Clostridium sordellii

UDP-glucose Deficiency Causes Hypersensitivity to the Cytotoxic Effect of Clostridium perfringens Phospholipase C

The cytolethal distending toxins induce DNA damage and cell cycle arrest

Defects in galactose metabolism and glycoconjugate biosynthesis in UDP‐glucose pyrophosphorylase‐deficient fibroblasts are reversed by supplementing the cell growth medium with galactose

GTPases of the Rho Subfamily Are Required for Brucella abortus Internalization in Nonprofessional Phagocytes

Large clostridial cytotoxins — a family of glycosyltransferases modifying small GTP-binding proteins

The cytolethal distending toxin from the chancroid bacterium Haemophilus ducreyi induces cell-cycle arrest in the G2 phase

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