UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 62

Sortiert nach

Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (19), p.6813-6818

2008

Volltextzugriff (PDF)

Concentration Dependent Cu2+ Induced Aggregation and Dityrosine Formation of the Alzheimer's Disease Amyloid-β Peptide

Biochemistry (Easton), 2007-03, Vol.46 (10), p.2881-2891

2007

Volltextzugriff (PDF)

Dopamine promotes α‐synuclein aggregation into SDS‐resistant soluble oligomers via a distinct folding pathway

The FASEB journal, 2005-08, Vol.19 (10), p.1377-1379

2005

Volltextzugriff (PDF)

Amyloid-β Peptide Disruption of Lipid Membranes and the Effect of Metal Ions

Journal of molecular biology, 2006-02, Vol.356 (3), p.759-770

2006

Volltextzugriff (PDF)

Cu2+ Binding Modes of Recombinant α-Synuclein − Insights from EPR Spectroscopy

Journal of the American Chemical Society, 2008-06, Vol.130 (24), p.7766-7773

2008

Volltextzugriff (PDF)

Copper-mediated Amyloid-β Toxicity Is Associated with an Intermolecular Histidine Bridge

The Journal of biological chemistry, 2006-06, Vol.281 (22), p.15145-15154

2006

Volltextzugriff (PDF)

Stabilization of Neurotoxic Soluble β-Sheet-Rich Conformations of the Alzheimer's Disease Amyloid-β Peptide

Biophysical journal, 2008-04, Vol.94 (7), p.2752-2766

2008

Volltextzugriff (PDF)

Restored degradation of the Alzheimer's amyloid-β peptide by targeting amyloid formation

Journal of neurochemistry, 2009-03, Vol.108 (5), p.1198-1207

2009

Volltextzugriff (PDF)

Formation of a High Affinity Lipid-Binding Intermediate during the Early Aggregation Phase of α-Synuclein

Biochemistry (Easton), 2008-02, Vol.47 (5), p.1425-1434

2008

Volltextzugriff (PDF)

Thrombin, a Survival Factor for Cultured Myoblasts

The Journal of biological chemistry, 1999-04, Vol.274 (14), p.9169-9174

1999

Volltextzugriff (PDF)

Stereospecific interactions are necessary for Alzheimer disease amyloid-β toxicity

Neurobiology of aging, 2011-02, Vol.32 (2), p.235-248

2011

Volltextzugriff (PDF)

Anionic Phospholipid Interactions of the Prion Protein N Terminus Are Minimally Perturbing and Not Driven Solely by the Octapeptide Repeat Domain

The Journal of biological chemistry, 2010-10, Vol.285 (42), p.32282-32292

2010

Volltextzugriff (PDF)

Methylation of the Imidazole Side Chains of the Alzheimer Disease Amyloid-β Peptide Results in Abolition of Superoxide Dismutase-like Structures and Inhibition of Neurotoxicity

The Journal of biological chemistry, 2005-04, Vol.280 (14), p.13355-13363

2005

Volltextzugriff (PDF)

Solid-phase synthesis of homodimeric peptides: preparation of covalently-linked dimers of amyloid beta peptide

Chemical communications (Cambridge, England), 2009-01 (41), p.6228-6230

2009

Volltextzugriff (PDF)

domain level interaction network of amyloid precursor protein and Aβ of Alzheimer's disease

Proteomics (Weinheim), 2010-06, Vol.10 (12), p.2377-2395

2010

Volltextzugriff (PDF)

Histidine 14 modulates membrane binding and neurotoxicity of the Alzheimer's disease amyloid-beta peptide

Journal of Alzheimer's disease, 2010-01, Vol.19 (4), p.1387

2010

Volltextzugriff (PDF)

Copper Coordination by Familial Mutants of Parkinson’s Disease-Associated α-Synuclein

Applied magnetic resonance, 2009-12, Vol.36 (2-4), p.223-229

2009

Volltextzugriff (PDF)

Intrinsic fluorescence changes and rapid kinetics of proteinase deformation during serpin inhibition

FEBS letters, 2001-04, Vol.494 (1), p.30-33

2001

Volltextzugriff (PDF)

Amyloid-β Peptide (Aβ) Neurotoxicity Is Modulated by the Rate of Peptide Aggregation: Aβ Dimers and Trimers Correlate with Neurotoxicity

The Journal of neuroscience, 2008-11, Vol.28 (46), p.11950-11958

2008

Volltextzugriff (PDF)

The Caenorhabditis elegans Aβ1–42 Model of Alzheimer Disease Predominantly Expresses Aβ3–42

The Journal of biological chemistry, 2009-08, Vol.284 (34), p.22697-22702

2009

Volltextzugriff (PDF)

P2-293: Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T458-T458

2008

Volltextzugriff (PDF)

Prevention of Polymerization of M and Z alpha 1-Antitrypsin (alpha 1-AT) with Trimethylamine N-Oxide . Implications for the Treatment of alpha 1-AT Deficiency

American journal of respiratory cell and molecular biology, 2001-06, Vol.24 (6), p.727-732

2001

Volltextzugriff (PDF)

Prevention of polymerization of M and Z alpha(1)-antitrypsin (alpha(1)-AT) with trimethylamine N-oxide: Implications for the treatment of alpha(1)-AT deficiency

American journal of respiratory cell and molecular biology, 2001-06, Vol.24 (6), p.727

2001

Volltextzugriff (PDF)

The Caenorhabditis elegans AÎ²1â42 Model of Alzheimer Disease Predominantly Expresses AÎ²3â42

The Journal of biological chemistry, 2009-08, Vol.284 (34), p.22697

2009

Volltextzugriff (PDF)

Caenorhabditis elegans Aβ₁₋₄₂ Model of Alzheimer Disease Predominantly Expresses Aβ

The Journal of biological chemistry, 2009, Vol.284 (34), p.22697-22702

2009

Volltextzugriff (PDF)

1 – 25 von 62

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(60)
Mit Volltextzugriff
(59)

Publikationsform

Artikel
(62)

Sprache

Englisch
(62)
Japanisch
(3)

Erscheinungsjahr

n.n

Thema

Science & Technology
(27)
Life Sciences & Biomedicine
(23)
Humans
(16)
Biochemistry & Molecular Biology
(15)
Animals
(11)
Amyloid Beta-Peptides - Chemistry
(9)
Amyloid Beta-Peptides - Metabolism
(9)
Mice
(9)
Alzheimer's Disease
(8)
Alzheimer Disease - Metabolism
(7)
Cells, Cultured
(7)
Neurology
(7)
Protein Structure, Secondary
(7)
Circular Dichroism
(6)
Amino Acid Sequence
(5)
Amyloid Beta-Peptides - Toxicity
(5)
Binding Sites
(5)
Copper - Chemistry
(5)
Electron Spin Resonance Spectroscopy
(5)
Female
(5)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Concentration Dependent Cu2+ Induced Aggregation and Dityrosine Formation of the Alzheimer's Disease Amyloid-β Peptide

Dopamine promotes α‐synuclein aggregation into SDS‐resistant soluble oligomers via a distinct folding pathway

Amyloid-β Peptide Disruption of Lipid Membranes and the Effect of Metal Ions

Cu2+ Binding Modes of Recombinant α-Synuclein − Insights from EPR Spectroscopy

Copper-mediated Amyloid-β Toxicity Is Associated with an Intermolecular Histidine Bridge

Stabilization of Neurotoxic Soluble β-Sheet-Rich Conformations of the Alzheimer's Disease Amyloid-β Peptide

Restored degradation of the Alzheimer's amyloid-β peptide by targeting amyloid formation

Formation of a High Affinity Lipid-Binding Intermediate during the Early Aggregation Phase of α-Synuclein

Thrombin, a Survival Factor for Cultured Myoblasts

Stereospecific interactions are necessary for Alzheimer disease amyloid-β toxicity

Anionic Phospholipid Interactions of the Prion Protein N Terminus Are Minimally Perturbing and Not Driven Solely by the Octapeptide Repeat Domain

Methylation of the Imidazole Side Chains of the Alzheimer Disease Amyloid-β Peptide Results in Abolition of Superoxide Dismutase-like Structures and Inhibition of Neurotoxicity

Solid-phase synthesis of homodimeric peptides: preparation of covalently-linked dimers of amyloid beta peptide

domain level interaction network of amyloid precursor protein and Aβ of Alzheimer's disease

Histidine 14 modulates membrane binding and neurotoxicity of the Alzheimer's disease amyloid-beta peptide

Copper Coordination by Familial Mutants of Parkinson’s Disease-Associated α-Synuclein

Intrinsic fluorescence changes and rapid kinetics of proteinase deformation during serpin inhibition

Amyloid-β Peptide (Aβ) Neurotoxicity Is Modulated by the Rate of Peptide Aggregation: Aβ Dimers and Trimers Correlate with Neurotoxicity

The Caenorhabditis elegans Aβ1–42 Model of Alzheimer Disease Predominantly Expresses Aβ3–42

P2-293: Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Prevention of Polymerization of M and Z alpha 1-Antitrypsin (alpha 1-AT) with Trimethylamine N-Oxide . Implications for the Treatment of alpha 1-AT Deficiency

Prevention of polymerization of M and Z alpha(1)-antitrypsin (alpha(1)-AT) with trimethylamine N-oxide: Implications for the treatment of alpha(1)-AT deficiency

The Caenorhabditis elegans AÎ²1â42 Model of Alzheimer Disease Predominantly Expresses AÎ²3â42

Caenorhabditis elegans Aβ₁₋₄₂ Model of Alzheimer Disease Predominantly Expresses Aβ

Suchergebnisse filtern