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Eukaryotic translational termination efficiency is influenced by the 3' nucleotides within the ribosomal mRNA channel

Nucleic acids research, 2018-02, Vol.46 (4), p.1927-1944

2018

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A pilot study on the immune cell proteome of long COVID patients shows changes to physiological pathways similar to those in myalgic encephalomyelitis/chronic fatigue syndrome

Scientific reports, 2023-12, Vol.13 (1), p.22068-22068, Article 22068

2023

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Role of mitochondria, oxidative stress and the response to antioxidants in myalgic encephalomyelitis/chronic fatigue syndrome: A possible approach to SARS-CoV-2 ‘long-haulers’?

Chronic diseases and translational medicine, 2021-03, Vol.7 (1), p.14-26

2021

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Primordial soup or vinaigrette: did the RNA world evolve at acidic pH?

Biology direct, 2012-01, Vol.7 (1), p.4-4

2012

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A SWATH-MS analysis of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome peripheral blood mononuclear cell proteomes reveals mitochondrial dysfunction

Journal of translational medicine, 2020-09, Vol.18 (1), p.365-18, Article 365

2020

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Secreted Amyloid Precursor Protein Alpha (sAPPα Regulates the Cellular Proteome and Secretome of Mouse Primary Astrocytes

International journal of molecular sciences, 2023-04, Vol.24 (8)

2023

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Secreted Amyloid Precursor Protein Alpha (sAPPα) Regulates the Cellular Proteome and Secretome of Mouse Primary Astrocytes

International journal of molecular sciences, 2023-04, Vol.24 (8), p.7165

2023

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Dynamic Epigenetic Changes during a Relapse and Recovery Cycle in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome

International journal of molecular sciences, 2022-10, Vol.23 (19), p.11852

2022

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A New Perspective on the Maillard Reaction and the Origin of Life

Journal of molecular evolution, 2021-12, Vol.89 (9-10), p.594-597

2021

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Towards a Better Understanding of the Complexities of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome and Long COVID

International journal of molecular sciences, 2023-03, Vol.24 (6), p.5124

2023

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A compromised paraventricular nucleus within a dysfunctional hypothalamus: A novel neuroinflammatory paradigm for ME/CFS

International journal of immunopathology and pharmacology, 2018-03, Vol.32

2018

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Molecular Mechanisms of Neuroinflammation in ME/CFS and Long COVID to Sustain Disease and Promote Relapses

Frontiers in neurology, 2022-05, Vol.13, p.877772-877772

2022

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Temporal profiling of gene networks associated with the late phase of long-term potentiation in vivo

PloS one, 2012-07, Vol.7 (7), p.e40538-e40538

2012

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Roles of amyloid precursor protein and its fragments in regulating neural activity, plasticity and memory

Progress in neurobiology, 2003-05, Vol.70 (1), p.1-32

2003

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Amyloid-Beta1-42 -Induced Increase in GABAergic Tonic Conductance in Mouse Hippocampal CA1 Pyramidal Cells

Molecules (Basel, Switzerland), 2020-02, Vol.25 (3), p.693

2020

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Secreted Amyloid Precursor Protein Alpha, a Neuroprotective Protein in the Brain Has Widespread Effects on the Transcriptome and Proteome of Human Inducible Pluripotent Stem Cell-Derived Glutamatergic Neurons Related to Memory Mechanisms

Frontiers in neuroscience, 2022-05, Vol.16, p.858524-858524

2022

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Beta-Amyloid (Aβ 1-42 ) Increases the Expression of NKCC1 in the Mouse Hippocampus

Molecules (Basel, Switzerland), 2022-04, Vol.27 (8), p.2440

2022

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‘Stop’ in protein synthesis is modulated with exquisite subtlety by an extended RNA translation signal

Biochemical Society transactions, 2018-12, Vol.46 (6), p.1615-1625

2018

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Glutamate Receptor Trafficking and Protein Synthesis Mediate the Facilitation of LTP by Secreted Amyloid Precursor Protein-Alpha

The Journal of neuroscience, 2019-04, Vol.39 (17), p.3188-3203

2019

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The Acute Effects of Amyloid-Beta1–42 on Glutamatergic Receptor and Transporter Expression in the Mouse Hippocampus

Frontiers in neuroscience, 2020-01, Vol.13, p.1427-1427

2020

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The highly conserved codon following the slippery sequence supports -1 frameshift efficiency at the HIV-1 frameshift site

PloS one, 2015-03, Vol.10 (3), p.e0122176-e0122176

2015

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Treatment of beta amyloid 1–42 (Aβ1–42)-induced basal forebrain cholinergic damage by a non-classical estrogen signaling activator in vivo

Scientific reports, 2016-02, Vol.6 (1), p.21101, Article 21101

2016

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Secreted amyloid precursor protein-alpha can restore novel object location memory and hippocampal LTP in aged rats

Neurobiology of learning and memory, 2017-02, Vol.138, p.291-299

2017

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Amyloid‐beta1–42 induced glutamatergic receptor and transporter expression changes in the mouse hippocampus

Journal of neurochemistry, 2020-10, Vol.155 (1), p.62-80

2020

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The transition from noncoded to coded protein synthesis: did coding mRNAs arise from stability-enhancing binding partners to tRNA?

Biology direct, 2010-04, Vol.5 (1), p.16-16

2010

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Eukaryotic translational termination efficiency is influenced by the 3' nucleotides within the ribosomal mRNA channel

A pilot study on the immune cell proteome of long COVID patients shows changes to physiological pathways similar to those in myalgic encephalomyelitis/chronic fatigue syndrome

Role of mitochondria, oxidative stress and the response to antioxidants in myalgic encephalomyelitis/chronic fatigue syndrome: A possible approach to SARS-CoV-2 ‘long-haulers’?

Primordial soup or vinaigrette: did the RNA world evolve at acidic pH?

A SWATH-MS analysis of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome peripheral blood mononuclear cell proteomes reveals mitochondrial dysfunction

Secreted Amyloid Precursor Protein Alpha (sAPPα Regulates the Cellular Proteome and Secretome of Mouse Primary Astrocytes

Secreted Amyloid Precursor Protein Alpha (sAPPα) Regulates the Cellular Proteome and Secretome of Mouse Primary Astrocytes

Dynamic Epigenetic Changes during a Relapse and Recovery Cycle in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome

A New Perspective on the Maillard Reaction and the Origin of Life

Towards a Better Understanding of the Complexities of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome and Long COVID

A compromised paraventricular nucleus within a dysfunctional hypothalamus: A novel neuroinflammatory paradigm for ME/CFS

Molecular Mechanisms of Neuroinflammation in ME/CFS and Long COVID to Sustain Disease and Promote Relapses

Temporal profiling of gene networks associated with the late phase of long-term potentiation in vivo

Roles of amyloid precursor protein and its fragments in regulating neural activity, plasticity and memory

Amyloid-Beta1-42 -Induced Increase in GABAergic Tonic Conductance in Mouse Hippocampal CA1 Pyramidal Cells

Secreted Amyloid Precursor Protein Alpha, a Neuroprotective Protein in the Brain Has Widespread Effects on the Transcriptome and Proteome of Human Inducible Pluripotent Stem Cell-Derived Glutamatergic Neurons Related to Memory Mechanisms

Beta-Amyloid (Aβ 1-42 ) Increases the Expression of NKCC1 in the Mouse Hippocampus

‘Stop’ in protein synthesis is modulated with exquisite subtlety by an extended RNA translation signal

Glutamate Receptor Trafficking and Protein Synthesis Mediate the Facilitation of LTP by Secreted Amyloid Precursor Protein-Alpha

The Acute Effects of Amyloid-Beta1–42 on Glutamatergic Receptor and Transporter Expression in the Mouse Hippocampus

The highly conserved codon following the slippery sequence supports -1 frameshift efficiency at the HIV-1 frameshift site

Treatment of beta amyloid 1–42 (Aβ1–42)-induced basal forebrain cholinergic damage by a non-classical estrogen signaling activator in vivo

Secreted amyloid precursor protein-alpha can restore novel object location memory and hippocampal LTP in aged rats

Amyloid‐beta1–42 induced glutamatergic receptor and transporter expression changes in the mouse hippocampus

The transition from noncoded to coded protein synthesis: did coding mRNAs arise from stability-enhancing binding partners to tRNA?

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