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Quercetin treatment increases H2O2 removal by restoration of endogenous antioxidant activity and blocks isoproterenol-induced cardiac hypertrophy

Naunyn-Schmiedeberg's archives of pharmacology, 2021-02, Vol.394 (2), p.217-226

2021

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Calorie restriction changes lipidomic profiles and maintains mitochondrial function and redox balance during isoproterenol-induced cardiac hypertrophy

Journal of physiology and biochemistry, 2022-02, Vol.78 (1), p.283-294

2022

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Diazoxide Modulates Cardiac Hypertrophy by Targeting H2O2 Generation and Mitochondrial Superoxide Dismutase Activity

Current molecular pharmacology, 2020-01, Vol.13 (1), p.76-83

2020

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Mitochondria and Cardiac Hypertrophy

Advances in experimental medicine and biology, 2017, Vol.982, p.203-226

2017

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Insights into Mitochondrial Protection in Ischemic/pharmacological Preconditioning Through a Modified Glibenclamide Lacking the Sulphonylurea Portion

Biochimica et biophysica acta. Bioenergetics, 2024-09, Vol.1865, p.149422, Article 149422

2024

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Calorie restriction anti-hypertrophic effects are associated with improved mitochondrial content, blockage of Ca2+-induced mitochondrial damage, and lower reverse electron transport-mediated oxidative stress

Free radical research, 2024-04, Vol.58 (4), p.293-310

2024

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Calorie restriction anti-hypertrophic effects are associated with improved mitochondrial content, blockage of Ca 2+ -induced mitochondrial damage, and lower reverse electron transport-mediated oxidative stress

Free radical research, 2024-04, Vol.58 (4), p.293-310

2024

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Mitochondrial ATP-sensitive Potassium Channel Opening Inhibits Isoproterenol-induced Cardiac Hypertrophy by Preventing Oxidative Damage

Journal of cardiovascular pharmacology, 2015-04, Vol.65 (4), p.393-397

2015

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O‐GlcNAc signaling regulates cardiac hypertrophy via NFAT activation

The FASEB journal, 2010-04, Vol.24 (S1), p.978.18-978.18

2010

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Competitive interaction between ATP and GTP regulates mitochondrial ATP-sensitive potassium channels

Chemico-biological interactions, 2023-08, Vol.381, p.110560-110560, Article 110560

2023

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Association between Epstein-Barr virus (EBV) and cervical carcinoma: A meta-analysis

Gynecologic oncology, 2018-02, Vol.148 (2), p.317-328

2018

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Calorie restriction anti-hypertrophic effects are associated with improved mitochondrial content, blockage of Ca 2+ -induced mitochondrial damage, and lower reverse electron transport-mediated oxidative stress

Free radical research, 2024-04, p.1

2024

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Effects of vitamin D (VD3) supplementation on the brain mitochondrial function of male rats, in the 6-OHDA-induced model of Parkinson's disease

Neurochemistry international, 2022-03, Vol.154, p.105280-105280, Article 105280

2022

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Preface: Epstein–Barr Virus and Oncogenesis

Critical reviews in oncogenesis, 2019, Vol.24 (4), p.v-vii

2019

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Pharmacological and molecular docking studies reveal that glibenclamide competitively inhibits diazoxide-induced mitochondrial ATP-sensitive potassium channel activation and pharmacological preconditioning

European journal of pharmacology, 2021-10, Vol.908, p.174379, Article 174379

2021

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Association between Epstein-Barr Virus and Oral Carcinoma: A Systematic Review with Meta-Analysis

Critical reviews in oncogenesis, 2019, Vol.24 (4), p.349-368

2019

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Quercetin treatment increases H 2 O 2 removal by restoration of endogenous antioxidant activity and blocks isoproterenol-induced cardiac hypertrophy

Naunyn-Schmiedeberg's archives of pharmacology, 2021-02, Vol.394 (2), p.217

2021

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Quercetin treatment increases H2O2 removal by restoration of endogenous antioxidant activity and blocks isoproterenol-induced cardiac hypertrophy

Calorie restriction changes lipidomic profiles and maintains mitochondrial function and redox balance during isoproterenol-induced cardiac hypertrophy

Diazoxide Modulates Cardiac Hypertrophy by Targeting H2O2 Generation and Mitochondrial Superoxide Dismutase Activity

Mitochondria and Cardiac Hypertrophy

Insights into Mitochondrial Protection in Ischemic/pharmacological Preconditioning Through a Modified Glibenclamide Lacking the Sulphonylurea Portion

Calorie restriction anti-hypertrophic effects are associated with improved mitochondrial content, blockage of Ca2+-induced mitochondrial damage, and lower reverse electron transport-mediated oxidative stress

Calorie restriction anti-hypertrophic effects are associated with improved mitochondrial content, blockage of Ca 2+ -induced mitochondrial damage, and lower reverse electron transport-mediated oxidative stress

Mitochondrial ATP-sensitive Potassium Channel Opening Inhibits Isoproterenol-induced Cardiac Hypertrophy by Preventing Oxidative Damage

O‐GlcNAc signaling regulates cardiac hypertrophy via NFAT activation

Competitive interaction between ATP and GTP regulates mitochondrial ATP-sensitive potassium channels

Association between Epstein-Barr virus (EBV) and cervical carcinoma: A meta-analysis

Calorie restriction anti-hypertrophic effects are associated with improved mitochondrial content, blockage of Ca 2+ -induced mitochondrial damage, and lower reverse electron transport-mediated oxidative stress

Effects of vitamin D (VD3) supplementation on the brain mitochondrial function of male rats, in the 6-OHDA-induced model of Parkinson's disease

Preface: Epstein–Barr Virus and Oncogenesis

Pharmacological and molecular docking studies reveal that glibenclamide competitively inhibits diazoxide-induced mitochondrial ATP-sensitive potassium channel activation and pharmacological preconditioning

Association between Epstein-Barr Virus and Oral Carcinoma: A Systematic Review with Meta-Analysis

Quercetin treatment increases H 2 O 2 removal by restoration of endogenous antioxidant activity and blocks isoproterenol-induced cardiac hypertrophy

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