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3,3′-Diindolylmethane (DIM) inhibits the growth and invasion of drug-resistant human cancer cells expressing EGFR mutants

Cancer letters, 2010-09, Vol.295 (1), p.59-68

2010

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Phosphorylation of dedicator of cytokinesis 1 (Dock180) at tyrosine residue Y722 by Src family kinases mediates EGFRvIII-driven glioblastoma tumorigenesis

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (8), p.3018-3023

2012

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TOB suppresses breast cancer tumorigenesis

International journal of cancer, 2009-10, Vol.125 (8), p.1805-1813

2009

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Dissecting the EGFR-PI3K-AKT pathway in oral cancer highlights the role of the EGFR variant III and its clinical relevance

Journal of biomedical science, 2013-06, Vol.20 (1), p.43-43

2013

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EGFR variant‐mediated invasion by enhanced CXCR4 expression through transcriptional and post‐translational mechanisms

International journal of cancer, 2010-04, Vol.126 (8), p.1850-1860

2010

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CXCR4 suppression attenuates EGFRvIII-mediated invasion and induces p38 MAPK-dependent protein trafficking and degradation of EGFRvIII in breast cancer cells

Cancer letters, 2011-07, Vol.306 (1), p.43-51

2011

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Hypophosphorylation of residue Y1045 leads to defective downregulation of EGFRvIII

Cancer biology & therapy, 2006-10, Vol.5 (10), p.1361-1368

2006

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Evidence of high incidence of EGFRvIII expression and coexpression with EGFR in human invasive breast cancer by laser capture microdissection and immunohistochemical analysis

International journal of cancer, 2002-03, Vol.98 (3), p.357-361

2002

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Co-expression of EGFRvIII with ErbB-2 enhances tumorigenesis: EGFRvIII mediated constitutively activated and sustained signaling pathways, whereas EGF-induced a transient effect on EGFR-mediated signaling pathways

Cancer biology & therapy, 2008-11, Vol.7 (11), p.1818-1828

2008

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Blockage of heregulin expression inhibits tumorigenicity and metastasis of breast cancer

Oncogene, 2003-02, Vol.22 (5), p.761-768

2003

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Epidermal growth factor receptor vIII enhances tumorigenicity in human breast cancer

Cancer research (Chicago, Ill.), 2000-06, Vol.60 (11), p.3081-3087

2000

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Production of biologically active IgG hinge-tag soluble epidermal growth factor receptors (ErbB)

Biotechnology letters, 2010-03, Vol.32 (3), p.361-366

2010

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Ribozyme-mediated down-regulation of ErbB-4 in estrogen receptor-positive breast cancer cells inhibits proliferation both in vitro and in vivo

Cancer research (Chicago, Ill.), 1999-10, Vol.59 (20), p.5315-5322

1999

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Suppression of EGFRvIII‐mediated proliferation and tumorigenesis of breast cancer cells by ribozyme

International journal of cancer, 2003-05, Vol.104 (6), p.716-721

2003

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Reciprocal Control of RNA-Binding and Aconitase Activity in the Regulation of the Iron-Responsive Element Binding Protein: Role of the Iron-Sulfur Cluster

Proceedings of the National Academy of Sciences - PNAS, 1992-08, Vol.89 (16), p.7536-7540

1992

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EGFRvIII‐induced estrogen‐independence, tamoxifen‐resistance phenotype correlates with PgR expression and modulation of apoptotic molecules in breast cancer

International journal of cancer, 2009-11, Vol.125 (9), p.2021-2028

2009

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AEE788: A dual family epidermal growth factor receptor/ErbB2 and vascular endothelial growth factor receptor tyrosine kinase inhibitor with antitumor and antiangiogenic activity

Cancer research (Chicago, Ill.), 2004-07, Vol.64 (14), p.4931-4941

2004

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Tumor progression and metastasis from genetic to microenvironmental determinants: a workshop of the tumor progression and metastasis NIH study section in honor of Dr. Martin L. Padarathsingh, May 31, 2006, Georgetown, Washington, DC

Cancer biology & therapy, 2006-12, Vol.5 (12), p.1588-1599

2006

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AEE788

Cancer research (Chicago, Ill.), 2004-07, Vol.64 (14), p.4931-4941

2004

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Cloning of the cDNA Encoding an RNA Regulatory Protein-The Human Iron- Responsive Element-Binding Protein

Proceedings of the National Academy of Sciences - PNAS, 1990-10, Vol.87 (20), p.7958-7962

1990

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Heregulin is sufficient for the promotion of tumorigenicity and metastasis of breast cancer cells in vivo

Molecular cancer research, 2003, Vol.1 (3), p.165-175

2003

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ErbB-4 ribozymes abolish neuregulin-induced mitogenesis

Cancer research (Chicago, Ill.), 1998-08, Vol.58 (15), p.3415-3422

1998

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Abstract 2390: EGFR variant III act as the major determinant factors in the EGFR/PI3K/AKT pathway for the survival outcome of oral cancer patients

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.2390-2390

2013

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Tumor progression and metastasis from genetic to microenvironmental determinants

Cancer biology & therapy, 2006-12, Vol.5 (12), p.1588-1599

2006

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Overexpression of KAI1 suppresses in vitro invasiveness and in vivo metastasis in breast cancer cells

Cancer research (Chicago, Ill.), 2001-07, Vol.61 (13), p.5284-5288

2001

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3,3′-Diindolylmethane (DIM) inhibits the growth and invasion of drug-resistant human cancer cells expressing EGFR mutants

Phosphorylation of dedicator of cytokinesis 1 (Dock180) at tyrosine residue Y722 by Src family kinases mediates EGFRvIII-driven glioblastoma tumorigenesis

TOB suppresses breast cancer tumorigenesis

Dissecting the EGFR-PI3K-AKT pathway in oral cancer highlights the role of the EGFR variant III and its clinical relevance

EGFR variant‐mediated invasion by enhanced CXCR4 expression through transcriptional and post‐translational mechanisms

CXCR4 suppression attenuates EGFRvIII-mediated invasion and induces p38 MAPK-dependent protein trafficking and degradation of EGFRvIII in breast cancer cells

Hypophosphorylation of residue Y1045 leads to defective downregulation of EGFRvIII

Evidence of high incidence of EGFRvIII expression and coexpression with EGFR in human invasive breast cancer by laser capture microdissection and immunohistochemical analysis

Co-expression of EGFRvIII with ErbB-2 enhances tumorigenesis: EGFRvIII mediated constitutively activated and sustained signaling pathways, whereas EGF-induced a transient effect on EGFR-mediated signaling pathways

Blockage of heregulin expression inhibits tumorigenicity and metastasis of breast cancer

Epidermal growth factor receptor vIII enhances tumorigenicity in human breast cancer

Production of biologically active IgG hinge-tag soluble epidermal growth factor receptors (ErbB)

Ribozyme-mediated down-regulation of ErbB-4 in estrogen receptor-positive breast cancer cells inhibits proliferation both in vitro and in vivo

Suppression of EGFRvIII‐mediated proliferation and tumorigenesis of breast cancer cells by ribozyme

Reciprocal Control of RNA-Binding and Aconitase Activity in the Regulation of the Iron-Responsive Element Binding Protein: Role of the Iron-Sulfur Cluster

EGFRvIII‐induced estrogen‐independence, tamoxifen‐resistance phenotype correlates with PgR expression and modulation of apoptotic molecules in breast cancer

AEE788: A dual family epidermal growth factor receptor/ErbB2 and vascular endothelial growth factor receptor tyrosine kinase inhibitor with antitumor and antiangiogenic activity

Tumor progression and metastasis from genetic to microenvironmental determinants: a workshop of the tumor progression and metastasis NIH study section in honor of Dr. Martin L. Padarathsingh, May 31, 2006, Georgetown, Washington, DC

AEE788

Cloning of the cDNA Encoding an RNA Regulatory Protein-The Human Iron- Responsive Element-Binding Protein

Heregulin is sufficient for the promotion of tumorigenicity and metastasis of breast cancer cells in vivo

ErbB-4 ribozymes abolish neuregulin-induced mitogenesis

Abstract 2390: EGFR variant III act as the major determinant factors in the EGFR/PI3K/AKT pathway for the survival outcome of oral cancer patients

Tumor progression and metastasis from genetic to microenvironmental determinants

Overexpression of KAI1 suppresses in vitro invasiveness and in vivo metastasis in breast cancer cells

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