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Bidirectional, non-necrotizing glomerular crescents are the critical pathology in X-linked Alport syndrome mouse model harboring nonsense mutation of human COL4A5

Scientific reports, 2020-11, Vol.10 (1), p.18891-18891, Article 18891

2020

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Transgenic mice overexpressing miR-137 in the brain show schizophrenia-associated behavioral deficits and transcriptome profiles

PloS one, 2019-07, Vol.14 (7), p.e0220389-e0220389

2019

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Suppression of the hypothalamic–pituitary–gonadal axis by TAK-385 (relugolix), a novel, investigational, orally active, small molecule gonadotropin-releasing hormone (GnRH) antagonist: Studies in human GnRH receptor knock-in mice

European journal of pharmacology, 2014-01, Vol.723, p.167-174

2014

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Establishment of X-linked Alport syndrome model mice with a Col4a5 R471X mutation

Biochemistry and biophysics reports, 2019-03, Vol.17, p.81-86

2019

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Development of a novel murine heart failure model overexpressing human renin and angiotensinogen

FEBS open bio, 2020-05, Vol.10 (5), p.718-725

2020

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Efficient gene-targeting in rat embryonic stem cells by CRISPR/Cas and generation of human kynurenine aminotransferase II (KAT II) knock-in rat

Transgenic research, 2015-12, Vol.24 (6), p.991-1001

2015

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Derivation of rat embryonic stem cells and generation of protease-activated receptor-2 knockout rats

Transgenic research, 2012-08, Vol.21 (4), p.743-755

2012

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Hypercholesterolemia and atherosclerosis in low density lipoprotein receptor mutant rats

Biochemical and biophysical research communications, 2012-02, Vol.418 (3), p.553-558

2012

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Human apolipoprotein B transgenic SHR/NDmcr-cp rats show exacerbated kidney dysfunction

Experimental Animals, 2015, Vol.64(3), pp.313-321

2015

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Osborne-Mendel rats simultaneously develop cardiac and renal dysfunction, left atrial thrombosis, peripheral artery occlusion, and ascending aortic dissection

Biochemical and biophysical research communications, 2017-04, Vol.485 (2), p.550-555

2017

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Generation of gene-targeted mice using embryonic stem cells derived from a transgenic mouse model of Alzheimer’s disease

Transgenic research, 2013-06, Vol.22 (3), p.537-547

2013

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Mutational replacements of conserved amino acid residues in the α subunit change the catalytic properties of Escherichia coli F1-ATPase

Archives of biochemistry and biophysics, 1989-02, Vol.268 (2), p.643-648

1989

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Escherichia coli H+-ATPase: Role of the δ subunit in binding F1 to the F0 sector

Archives of biochemistry and biophysics, 1992-02, Vol.292 (2), p.376-381

1992

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The glycine-rich sequence of the beta subunit of Escherichia coli H(+)-ATPase is important for activity

The Journal of biological chemistry, 1990-12, Vol.265 (34), p.21279-21284

1990

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Role of the amino terminal region of the ϵ subunit of Escherichia coli H +-ATPase (FoF 1)

Archives of biochemistry and biophysics, 1992, Vol.292 (1), p.87-94

1992

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Escherichia coli H +-ATPase: Role of the δ subunit in binding F 1 to the F 0 sector

Archives of biochemistry and biophysics, 1992, Vol.292 (2), p.376-381

1992

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Mutational replacements of conserved amino acid residues in the α subunit change the catalytic properties of Escherichia coli F 1-ATPase

Archives of biochemistry and biophysics, 1989, Vol.268 (2), p.643-648

1989

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Escherichia coli H+-ATPase: loss of the carboxyl terminal region of the γ subunit causes defective assembly of the F1 portion

Archives of biochemistry and biophysics, 1986-12, Vol.251 (2), p.458-464

1986

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ß-Subunit of Escherichia coli F 1-ATPase : An amino acid replacement within a conserved sequence (G-X-X-X-X-G-K-T/S) of nucleotide-binding proteins

FEBS letters, 1987-06, Vol.218 (2), p.222-226

1987

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ß‐Subunit of Escherichia coli F1‐ATPase

FEBS letters, 1987-06, Vol.218 (2), p.222-226

1987

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Escherichia coli H +-ATPase: Loss of the carboxyl terminal region of the γ subunit causes defective assembly of the F 1 portion

Archives of biochemistry and biophysics, 1986, Vol.251 (2), p.458-464

1986

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Humoral and cellular components in bronchoalveolar lavage fluid of atopic asthmatics

Nihon Kyōbu Shikkan Gakkai zasshi, 1988-12, Vol.26 (12), p.1257

1988

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Correlation between cellular components and infiltrating cells in the lung tissue of asthmatics

Okayama Igakkai zasshi, 1983-12, Vol.95 (11-12), p.1153-1158

1983

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Bidirectional, non-necrotizing glomerular crescents are the critical pathology in X-linked Alport syndrome mouse model harboring nonsense mutation of human COL4A5

Transgenic mice overexpressing miR-137 in the brain show schizophrenia-associated behavioral deficits and transcriptome profiles

Suppression of the hypothalamic–pituitary–gonadal axis by TAK-385 (relugolix), a novel, investigational, orally active, small molecule gonadotropin-releasing hormone (GnRH) antagonist: Studies in human GnRH receptor knock-in mice

Establishment of X-linked Alport syndrome model mice with a Col4a5 R471X mutation

Development of a novel murine heart failure model overexpressing human renin and angiotensinogen

Efficient gene-targeting in rat embryonic stem cells by CRISPR/Cas and generation of human kynurenine aminotransferase II (KAT II) knock-in rat

Derivation of rat embryonic stem cells and generation of protease-activated receptor-2 knockout rats

Hypercholesterolemia and atherosclerosis in low density lipoprotein receptor mutant rats

Human apolipoprotein B transgenic SHR/NDmcr-cp rats show exacerbated kidney dysfunction

Osborne-Mendel rats simultaneously develop cardiac and renal dysfunction, left atrial thrombosis, peripheral artery occlusion, and ascending aortic dissection

Generation of gene-targeted mice using embryonic stem cells derived from a transgenic mouse model of Alzheimer’s disease

Mutational replacements of conserved amino acid residues in the α subunit change the catalytic properties of Escherichia coli F1-ATPase

Escherichia coli H+-ATPase: Role of the δ subunit in binding F1 to the F0 sector

The glycine-rich sequence of the beta subunit of Escherichia coli H(+)-ATPase is important for activity

Role of the amino terminal region of the ϵ subunit of Escherichia coli H +-ATPase (FoF 1)

Escherichia coli H +-ATPase: Role of the δ subunit in binding F 1 to the F 0 sector

Mutational replacements of conserved amino acid residues in the α subunit change the catalytic properties of Escherichia coli F 1-ATPase

Escherichia coli H+-ATPase: loss of the carboxyl terminal region of the γ subunit causes defective assembly of the F1 portion

ß-Subunit of Escherichia coli F 1-ATPase : An amino acid replacement within a conserved sequence (G-X-X-X-X-G-K-T/S) of nucleotide-binding proteins

ß‐Subunit of Escherichia coli F1‐ATPase

Escherichia coli H +-ATPase: Loss of the carboxyl terminal region of the γ subunit causes defective assembly of the F 1 portion

Humoral and cellular components in bronchoalveolar lavage fluid of atopic asthmatics

Correlation between cellular components and infiltrating cells in the lung tissue of asthmatics

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