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Toxic AGEs (TAGE) theory: a new concept for preventing the development of diseases related to lifestyle

Diabetology and metabolic syndrome, 2020-11, Vol.12 (1), p.105-12, Article 105

Takeuchi, Masayoshi

2020

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Serum Levels of Toxic AGEs (TAGE) May Be a Promising Novel Biomarker for the Onset/Progression of Lifestyle-Related Diseases

Diagnostics (Basel), 2016-06, Vol.6 (2), p.23

Takeuchi, Masayoshi

2016

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Intracellular toxic advanced glycation end-products in cardiomyocytes may cause cardiovascular disease

Scientific reports, 2019-02, Vol.9 (1), p.2121, Article 2121

2019

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Accumulation of Toxic Advanced Glycation End-Products Induces Cytotoxicity and Inflammation in Hepatocyte-Like Cells Differentiated from Human Induced Pluripotent Stem Cells

Biological and Pharmaceutical Bulletin, 2021/10/01, Vol.44(10), pp.1399-1402

2021

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Intracellular Toxic Advanced Glycation End-Products Promote the Production of Reactive Oxygen Species in HepG2 Cells

International journal of molecular sciences, 2020-07, Vol.21 (14), p.4861

2020

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Suppression of Hepatic Stellate Cell Death by Toxic Advanced Glycation End-Products

Biological and Pharmaceutical Bulletin, 2021/01/01, Vol.44(1), pp.112-117

2021

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Possible involvement of advanced glycation end-products (AGEs) in the pathogenesis of Alzheimer's disease

Current pharmaceutical design, 2008-04, Vol.14 (10), p.973

2008

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Intracellular Toxic Advanced Glycation End-Products in 1.4E7 Cell Line Induce Death with Reduction of Microtubule-Associated Protein 1 Light Chain 3 and p62

Nutrients, 2022-01, Vol.14 (2), p.332

2022

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Effect of Anagliptin on Vascular Injury in the Femoral Artery of Type 2 Diabetic Rats

Biological and Pharmaceutical Bulletin, 2024/01/19, Vol.47(1), pp.204-212

2024

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Glucagon-Like Peptide-1 Receptor Agonist Inhibits Asymmetric Dimethylarginine Generation in the Kidney of Streptozotocin-Induced Diabetic Rats by Blocking Advanced Glycation End Product–Induced Protein Arginine Methyltranferase-1 Expression

The American journal of pathology, 2013, Vol.182 (1), p.132-141

2013

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RasGRP2 inhibits glyceraldehyde-derived toxic advanced glycation end-products from inducing permeability in vascular endothelial cells

Scientific reports, 2021-02, Vol.11 (1), p.2959-10, Article 2959

2021

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Impact of intracellular toxic advanced glycation end-products (TAGE) on murine myoblast cell death

Diabetology and metabolic syndrome, 2020-06, Vol.12 (1), p.54-8, Article 54

2020

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Effect of Collagen Tripeptide on Atherosclerosis in Healthy Humans

Journal of Atherosclerosis and Thrombosis, 2017/05/01, Vol.24(5), pp.530-538

2017

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Glyceraldehyde caused Alzheimer's disease-like alterations in diagnostic marker levels in SH-SY5Y human neuroblastoma cells

Scientific reports, 2015-08, Vol.5 (1), p.13313, Article 13313

2015

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Glucagon-like peptide-1 (GLP-1) inhibits advanced glycation end product (AGE)-induced up-regulation of VCAM-1 mRNA levels in endothelial cells by suppressing AGE receptor (RAGE) expression

Biochemical and biophysical research communications, 2010-01, Vol.391 (3), p.1405-1408

2010

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Atorvastatin decreases serum levels of advanced glycation endproducts (AGEs) in nonalcoholic steatohepatitis (NASH) patients with dyslipidemia: clinical usefulness of AGEs as a biomarker for the attenuation of NASH

Journal of gastroenterology, 2010-07, Vol.45 (7), p.750-757

2010

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Generation of glyceraldehyde-derived advanced glycation end-products in pancreatic cancer cells and the potential of tumor promotion

World journal of gastroenterology : WJG, 2017-07, Vol.23 (27), p.4910-4919

2017

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Pyridoxamine and Aminoguanidine Attenuate the Abnormal Aggregation of β-Tubulin and Suppression of Neurite Outgrowth by Glyceraldehyde-Derived Toxic Advanced Glycation End-Products

Frontiers in pharmacology, 2022-06, Vol.13, p.921611-921611

2022

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Glycer-AGEs-RAGE signaling enhances the angiogenic potential of hepatocellular carcinoma by upregulating VEGF expression

World journal of gastroenterology : WJG, 2012-04, Vol.18 (15), p.1781-1788

2012

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Impact of intracellular glyceraldehyde-derived advanced glycation end-products on human hepatocyte cell death

Scientific reports, 2017-10, Vol.7 (1), p.14282-11, Article 14282

2017

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Immunological evidence for in vivo production of novel advanced glycation end-products from 1,5-anhydro-D-fructose, a glycogen metabolite

Scientific reports, 2019-07, Vol.9 (1), p.10194-9, Article 10194

2019

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Intracellular Toxic Advanced Glycation End-Products May Induce Cell Death and Suppress Cardiac Fibroblasts

Metabolites, 2022-07, Vol.12 (7), p.615

2022

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Involvement of Intracellular TAGE and the TAGE-RAGE-ROS Axis in the Onset and Progression of NAFLD/NASH

Antioxidants, 2023-03, Vol.12 (3), p.748

2023

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Sodium-glucose cotransporter 2-mediated oxidative stress augments advanced glycation end products-induced tubular cell apoptosis

Diabetes/metabolism research and reviews, 2013-07, Vol.29 (5), p.406-412

2013

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The Association between Accumulation of Toxic Advanced Glycation End-Products and Cytotoxic Effect in MC3T3-E1 Cells

Nutrients, 2022-02, Vol.14 (5), p.990

2022

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Toxic AGEs (TAGE) theory: a new concept for preventing the development of diseases related to lifestyle

Serum Levels of Toxic AGEs (TAGE) May Be a Promising Novel Biomarker for the Onset/Progression of Lifestyle-Related Diseases

Intracellular toxic advanced glycation end-products in cardiomyocytes may cause cardiovascular disease

Accumulation of Toxic Advanced Glycation End-Products Induces Cytotoxicity and Inflammation in Hepatocyte-Like Cells Differentiated from Human Induced Pluripotent Stem Cells

Intracellular Toxic Advanced Glycation End-Products Promote the Production of Reactive Oxygen Species in HepG2 Cells

Suppression of Hepatic Stellate Cell Death by Toxic Advanced Glycation End-Products

Possible involvement of advanced glycation end-products (AGEs) in the pathogenesis of Alzheimer's disease

Intracellular Toxic Advanced Glycation End-Products in 1.4E7 Cell Line Induce Death with Reduction of Microtubule-Associated Protein 1 Light Chain 3 and p62

Effect of Anagliptin on Vascular Injury in the Femoral Artery of Type 2 Diabetic Rats

Glucagon-Like Peptide-1 Receptor Agonist Inhibits Asymmetric Dimethylarginine Generation in the Kidney of Streptozotocin-Induced Diabetic Rats by Blocking Advanced Glycation End Product–Induced Protein Arginine Methyltranferase-1 Expression

RasGRP2 inhibits glyceraldehyde-derived toxic advanced glycation end-products from inducing permeability in vascular endothelial cells

Impact of intracellular toxic advanced glycation end-products (TAGE) on murine myoblast cell death

Effect of Collagen Tripeptide on Atherosclerosis in Healthy Humans

Glyceraldehyde caused Alzheimer's disease-like alterations in diagnostic marker levels in SH-SY5Y human neuroblastoma cells

Glucagon-like peptide-1 (GLP-1) inhibits advanced glycation end product (AGE)-induced up-regulation of VCAM-1 mRNA levels in endothelial cells by suppressing AGE receptor (RAGE) expression

Atorvastatin decreases serum levels of advanced glycation endproducts (AGEs) in nonalcoholic steatohepatitis (NASH) patients with dyslipidemia: clinical usefulness of AGEs as a biomarker for the attenuation of NASH

Generation of glyceraldehyde-derived advanced glycation end-products in pancreatic cancer cells and the potential of tumor promotion

Pyridoxamine and Aminoguanidine Attenuate the Abnormal Aggregation of β-Tubulin and Suppression of Neurite Outgrowth by Glyceraldehyde-Derived Toxic Advanced Glycation End-Products

Glycer-AGEs-RAGE signaling enhances the angiogenic potential of hepatocellular carcinoma by upregulating VEGF expression

Impact of intracellular glyceraldehyde-derived advanced glycation end-products on human hepatocyte cell death

Immunological evidence for in vivo production of novel advanced glycation end-products from 1,5-anhydro-D-fructose, a glycogen metabolite

Intracellular Toxic Advanced Glycation End-Products May Induce Cell Death and Suppress Cardiac Fibroblasts

Involvement of Intracellular TAGE and the TAGE-RAGE-ROS Axis in the Onset and Progression of NAFLD/NASH

Sodium-glucose cotransporter 2-mediated oxidative stress augments advanced glycation end products-induced tubular cell apoptosis

The Association between Accumulation of Toxic Advanced Glycation End-Products and Cytotoxic Effect in MC3T3-E1 Cells

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