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Sarcopenia-derived exosomal micro-RNA 16-5p disturbs cardio-repair via a pro-apoptotic mechanism in myocardial infarction in mice

Scientific reports, 2021-09, Vol.11 (1), p.19163-19163, Article 19163

2021

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Prostaglandin E2 mediates the late phase of ischemic preconditioning in the heart via its receptor subtype EP4

Heart and vessels, 2023-04, Vol.38 (4), p.606-613

2023

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Pericyte-specific deletion of Ninjurin-1 induces fragile vasa vasorum formation and enhances intimal hyperplasia of injured vasculature

American journal of physiology. Heart and circulatory physiology, 2021-06, Vol.320 (6), p.H2438-H2447

2021

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Controlled Delivery of Basic Fibroblast Growth Factor Promotes Human Cardiosphere-Derived Cell Engraftment to Enhance Cardiac Repair for Chronic Myocardial Infarction

Journal of the American College of Cardiology, 2008-12, Vol.52 (23), p.1858-1865

2008

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Malnutrition, renal dysfunction and left ventricular hypertrophy synergistically increase the long-term incidence of cardiovascular events

Hypertension research, 2016-09, Vol.39 (9), p.633-639

2016

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The antioxidant and DNA-repair enzyme apurinic/apyrimidinic endonuclease 1 limits the development of tubulointerstitial fibrosis partly by modulating the immune system

Scientific reports, 2019-05, Vol.9 (1), p.7823-7823, Article 7823

2019

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Pericyte-Specific Ninjurin1 Deletion Attenuates Vessel Maturation and Blood Flow Recovery in Hind Limb Ischemia

Arteriosclerosis, thrombosis, and vascular biology, 2018-10, Vol.38 (10), p.2358-2370

2018

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Apoptosis-Resistant Cardiac Progenitor Cells Modified With Apurinic/Apyrimidinic Endonuclease/Redox Factor 1 Gene Overexpression Regulate Cardiac Repair After Myocardial Infarction

Stem cells translational medicine, 2016-08, Vol.5 (8), p.1067-1078

2016

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Cardiac regeneration therapy: connections to cardiac physiology

American journal of physiology. Heart and circulatory physiology, 2011-12, Vol.301 (6), p.H2169-H2180

2011

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Capillary‐resident EphA7+ pericytes are multipotent cells with anti‐ischemic effects through capillary formation

Stem cells translational medicine, 2020-01, Vol.9 (1), p.120-130

2020

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Ninjurin1 Is a Novel Factor to Regulate Angiogenesis Through the Function of Pericytes

Circulation Journal, 2015/05/25, Vol.79(6), pp.1363-1371

2015

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First-in-human phase 1 trial of hemoglobin vesicles as artificial red blood cells developed for use as a transfusion alternative

Blood advances, 2022-11, Vol.6 (21), p.5711-5715

2022

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EphA7+ perivascular cells as myogenic and angiogenic precursors improving skeletal muscle regeneration in a muscular dystrophic mouse model

Stem cell research, 2020-08, Vol.47, p.101914, Article 101914

2020

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Adult Accessory Mitral Valve with Septal Hypertrophy

Internal Medicine, 2016/09/01, Vol.55(17), pp.2515-2515

2016

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MURC, a Muscle-Restricted Coiled-Coil Protein That Modulates the Rho/ROCK Pathway, Induces Cardiac Dysfunction and Conduction Disturbance

Molecular and Cellular Biology, 2008-05, Vol.28 (10), p.3424-3436

2008

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Immortalized multipotent pericytes derived from the vasa vasorum in the injured vasculature. A cellular tool for studies of vascular remodeling and regeneration

Laboratory investigation, 2014-12, Vol.94 (12), p.1340-1354

2014

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Association between microalbuminuria predicting in-stent restenosis after myocardial infarction and cellular senescence of endothelial progenitor cells

PloS one, 2015-04, Vol.10 (4), p.e0123733-e0123733

2015

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Prostacyclin Stimulated Integrin-Dependent Angiogenic Effects of Endothelial Progenitor Cells and Mediated Potent Circulation Recovery in Ischemic Hind Limb Model

Circulation Journal, 2013, Vol.77(4), pp.1053-1062

2013

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Nerve growth factor stimulates regeneration of perivascular nerve, and induces the maturation of microvessels around the injured artery

Biochemical and biophysical research communications, 2014-01, Vol.443 (1), p.150-155

2014

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Cell Therapy for Cardiovascular Regeneration

Annals of Vascular Diseases, 2013/06/25, Vol.6(2), pp.137-144

Takehara, Naofumi

2013

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Clonally amplified cardiac stem cells are regulated by Sca-1 signaling for efficient cardiovascular regeneration

Journal of cell science, 2007-05, Vol.120 (10), p.1791-1800

2007

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MURC, a muscle-restricted coiled-coil protein, is involved in the regulation of skeletal myogenesis

American Journal of Physiology: Cell Physiology, 2008-08, Vol.295 (2), p.C490-C498

2008

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Apurinic/apyrimidinic endonucelase 1 maintains adhesion of endothelial progenitor cells and reduces neointima formation

American journal of physiology. Heart and circulatory physiology, 2013-10, Vol.305 (8), p.H1158-H1167

2013

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Visit-to-visit variability and seasonal variation in blood pressure: Combination of Antihypertensive Therapy in the Elderly, Multicenter Investigation (CAMUI) Trial subanalysis

Clinical and experimental hypertension (1993), 2015-01, Vol.37 (5), p.411-419

2015

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Three cases of corticosteroid therapy triggering ventricular fibrillation in J-wave syndromes

Heart and vessels, 2014-11, Vol.29 (6), p.867-872

2014

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Sarcopenia-derived exosomal micro-RNA 16-5p disturbs cardio-repair via a pro-apoptotic mechanism in myocardial infarction in mice

Prostaglandin E2 mediates the late phase of ischemic preconditioning in the heart via its receptor subtype EP4

Pericyte-specific deletion of Ninjurin-1 induces fragile vasa vasorum formation and enhances intimal hyperplasia of injured vasculature

Controlled Delivery of Basic Fibroblast Growth Factor Promotes Human Cardiosphere-Derived Cell Engraftment to Enhance Cardiac Repair for Chronic Myocardial Infarction

Malnutrition, renal dysfunction and left ventricular hypertrophy synergistically increase the long-term incidence of cardiovascular events

The antioxidant and DNA-repair enzyme apurinic/apyrimidinic endonuclease 1 limits the development of tubulointerstitial fibrosis partly by modulating the immune system

Pericyte-Specific Ninjurin1 Deletion Attenuates Vessel Maturation and Blood Flow Recovery in Hind Limb Ischemia

Apoptosis-Resistant Cardiac Progenitor Cells Modified With Apurinic/Apyrimidinic Endonuclease/Redox Factor 1 Gene Overexpression Regulate Cardiac Repair After Myocardial Infarction

Cardiac regeneration therapy: connections to cardiac physiology

Capillary‐resident EphA7+ pericytes are multipotent cells with anti‐ischemic effects through capillary formation

Ninjurin1 Is a Novel Factor to Regulate Angiogenesis Through the Function of Pericytes

First-in-human phase 1 trial of hemoglobin vesicles as artificial red blood cells developed for use as a transfusion alternative

EphA7+ perivascular cells as myogenic and angiogenic precursors improving skeletal muscle regeneration in a muscular dystrophic mouse model

Adult Accessory Mitral Valve with Septal Hypertrophy

MURC, a Muscle-Restricted Coiled-Coil Protein That Modulates the Rho/ROCK Pathway, Induces Cardiac Dysfunction and Conduction Disturbance

Immortalized multipotent pericytes derived from the vasa vasorum in the injured vasculature. A cellular tool for studies of vascular remodeling and regeneration

Association between microalbuminuria predicting in-stent restenosis after myocardial infarction and cellular senescence of endothelial progenitor cells

Prostacyclin Stimulated Integrin-Dependent Angiogenic Effects of Endothelial Progenitor Cells and Mediated Potent Circulation Recovery in Ischemic Hind Limb Model

Nerve growth factor stimulates regeneration of perivascular nerve, and induces the maturation of microvessels around the injured artery

Cell Therapy for Cardiovascular Regeneration

Clonally amplified cardiac stem cells are regulated by Sca-1 signaling for efficient cardiovascular regeneration

MURC, a muscle-restricted coiled-coil protein, is involved in the regulation of skeletal myogenesis

Apurinic/apyrimidinic endonucelase 1 maintains adhesion of endothelial progenitor cells and reduces neointima formation

Visit-to-visit variability and seasonal variation in blood pressure: Combination of Antihypertensive Therapy in the Elderly, Multicenter Investigation (CAMUI) Trial subanalysis

Three cases of corticosteroid therapy triggering ventricular fibrillation in J-wave syndromes

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