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Molecular mechanism of intramembrane proteolysis by γ-secretase

Journal of biochemistry (Tokyo), 2014-10, Vol.156 (4), p.195-201

Tomita, Taisuke

2014

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Secretase inhibitors and modulators for Alzheimer's disease treatment

Expert review of neurotherapeutics, 2009-05, Vol.9 (5), p.661-679

Tomita, Taisuke

2009

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Aberrant proteolytic processing and therapeutic strategies in Alzheimer disease

Advances in biological regulation, 2017-05, Vol.64, p.33-38

Tomita, Taisuke

2017

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Mouse Models of Alzheimer's Disease

Frontiers in molecular neuroscience, 2022-06, Vol.15, p.912995

2022

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Sequential conformational changes in transmembrane domains of presenilin 1 in Aβ42 downregulation

Journal of biochemistry (Tokyo), 2021-08, Vol.170 (2), p.215-227

2021

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Molecular mechanisms of the genetic risk factors in pathogenesis of Alzheimer disease

Frontiers in bioscience, 2017-01, Vol.22 (1), p.180-192, Article 4480

2017

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Genetic Risk Factors for Alzheimer Disease: Emerging Roles of Microglia in Disease Pathomechanisms

Advances in experimental medicine and biology, 2019-01, Vol.1118, p.83-116

2019

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Distinct Neurotoxic Effects of Extracellular Tau Species in Primary Neuronal-Glial Cultures

Molecular neurobiology, 2021-02, Vol.58 (2), p.658-667

2021

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Structural Biology of Presenilins and Signal Peptide Peptidases

The Journal of biological chemistry, 2013-05, Vol.288 (21), p.14673-14680

2013

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BORCS6 is involved in the enlargement of lung lamellar bodies in Lrrk2 knockout mice

Human molecular genetics, 2021-08, Vol.30 (17), p.1618-1631

2021

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AP180 N-Terminal Homology (ANTH) and Epsin N-Terminal Homology (ENTH) Domains: Physiological Functions and Involvement in Disease

Advances in experimental medicine and biology, 2019-01, Vol.1111, p.55-76

2019

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Pathogenic LRRK2 compromises the subcellular distribution of lysosomes in a Rab12‐RILPL1‐dependent manner

The FASEB journal, 2023-05, Vol.37 (5), p.e22930-n/a

2023

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Seeded assembly in vitro does not replicate the structures of α‐synuclein filaments from multiple system atrophy

FEBS open bio, 2021-04, Vol.11 (4), p.999-1013

2021

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CD2-associated protein (CD2AP) overexpression accelerates amyloid precursor protein (APP) transfer from early endosomes to the lysosomal degradation pathway

The Journal of biological chemistry, 2019-07, Vol.294 (28), p.10886-10899

2019

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Specific mutations in presenilin 1 cause conformational changes in γ-secretase to modulate amyloid β trimming

Journal of biochemistry (Tokyo), 2019-01, Vol.165 (1), p.37-46

2019

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Photo-oxygenation inhibits tau amyloid formation

Chemical communications (Cambridge, England), 2019-05, Vol.55 (44), p.6165-6168

2019

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Histidine 131 in presenilin 1 is the pH-sensitive residue that causes the increase in Aβ42 level in acidic pH

Journal of biochemistry (Tokyo), 2020-05, Vol.167 (5), p.463-471

2020

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Detection of amyloid plaques targeted by bifunctional USPIO in Alzheimer's disease transgenic mice using magnetic resonance microimaging

PloS one, 2013-02, Vol.8 (2), p.e57097

2013

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Protein trafficking and maturation regulate intramembrane proteolysis

Biochimica et biophysica acta, 2013-12, Vol.1828 (12), p.2855-2861

2013

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Specific Mutations in Aph1 Cause γ-Secretase Activation

International journal of molecular sciences, 2022-01, Vol.23 (1), p.507

2022

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Presenilin Is Essential for ApoE Secretion, a Novel Role of Presenilin Involved in Alzheimer's Disease Pathogenesis

The Journal of neuroscience, 2022-02, Vol.42 (8), p.1574-1586

2022

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Human tauopathy-derived tau strains determine the substrates recruited for templated amplification

Brain (London, England : 1878), 2021-09, Vol.144 (8), p.2333-2348

2021

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The Regulation of Rab GTPases by Phosphorylation

Biomolecules (Basel, Switzerland), 2021-09, Vol.11 (9), p.1340

2021

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Novel aspects of the phosphorylation and structure of pathological tau: implications for tauopathy biomarkers

FEBS open bio, 2024-02, Vol.14 (2), p.181-193

2024

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Dietary cis-9, trans-11-conjugated linoleic acid reduces amyloid β-protein accumulation and upregulates anti-inflammatory cytokines in an Alzheimer’s disease mouse model

Scientific reports, 2021-05, Vol.11 (1), p.9749-9749, Article 9749

2021

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Molecular mechanism of intramembrane proteolysis by γ-secretase

Secretase inhibitors and modulators for Alzheimer's disease treatment

Aberrant proteolytic processing and therapeutic strategies in Alzheimer disease

Mouse Models of Alzheimer's Disease

Sequential conformational changes in transmembrane domains of presenilin 1 in Aβ42 downregulation

Molecular mechanisms of the genetic risk factors in pathogenesis of Alzheimer disease

Genetic Risk Factors for Alzheimer Disease: Emerging Roles of Microglia in Disease Pathomechanisms

Distinct Neurotoxic Effects of Extracellular Tau Species in Primary Neuronal-Glial Cultures

Structural Biology of Presenilins and Signal Peptide Peptidases

BORCS6 is involved in the enlargement of lung lamellar bodies in Lrrk2 knockout mice

AP180 N-Terminal Homology (ANTH) and Epsin N-Terminal Homology (ENTH) Domains: Physiological Functions and Involvement in Disease

Pathogenic LRRK2 compromises the subcellular distribution of lysosomes in a Rab12‐RILPL1‐dependent manner

Seeded assembly in vitro does not replicate the structures of α‐synuclein filaments from multiple system atrophy

CD2-associated protein (CD2AP) overexpression accelerates amyloid precursor protein (APP) transfer from early endosomes to the lysosomal degradation pathway

Specific mutations in presenilin 1 cause conformational changes in γ-secretase to modulate amyloid β trimming

Photo-oxygenation inhibits tau amyloid formation

Histidine 131 in presenilin 1 is the pH-sensitive residue that causes the increase in Aβ42 level in acidic pH

Detection of amyloid plaques targeted by bifunctional USPIO in Alzheimer's disease transgenic mice using magnetic resonance microimaging

Protein trafficking and maturation regulate intramembrane proteolysis

Specific Mutations in Aph1 Cause γ-Secretase Activation

Presenilin Is Essential for ApoE Secretion, a Novel Role of Presenilin Involved in Alzheimer's Disease Pathogenesis

Human tauopathy-derived tau strains determine the substrates recruited for templated amplification

The Regulation of Rab GTPases by Phosphorylation

Novel aspects of the phosphorylation and structure of pathological tau: implications for tauopathy biomarkers

Dietary cis-9, trans-11-conjugated linoleic acid reduces amyloid β-protein accumulation and upregulates anti-inflammatory cytokines in an Alzheimer’s disease mouse model

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