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Ranolazine Improves Cardiac Diastolic Dysfunction Through Modulation of Myofilament Calcium Sensitivity

Circulation research, 2012-03, Vol.110 (6), p.841-850

2012

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P21-activated kinase in inflammatory and cardiovascular disease

Cellular signalling, 2014-09, Vol.26 (9), p.2060-2069

2014

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Pak1 as a novel therapeutic target for antihypertrophic treatment in the heart

Circulation (New York, N.Y.), 2011-12, Vol.124 (24), p.2702-2715

2011

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The β-arrestin-biased ligand TRV120023 inhibits angiotensin II-induced cardiac hypertrophy while preserving enhanced myofilament response to calcium

American journal of physiology. Heart and circulatory physiology, 2013-09, Vol.305 (6), p.H856-H866

2013

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Tetrahydrobiopterin improves diastolic dysfunction by reversing changes in myofilament properties

Journal of molecular and cellular cardiology, 2013-03, Vol.56, p.44-54

2013

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Ablation of p21-activated kinase-1 in mice promotes isoproterenol-induced cardiac hypertrophy in association with activation of Erk1/2 and inhibition of protein phosphatase 2A

Journal of molecular and cellular cardiology, 2011-12, Vol.51 (6), p.988-996

2011

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Src Homology 2 Domain-containing Phosphatase 2 (Shp2) Is a Component of the A-kinase-anchoring Protein (AKAP)-Lbc Complex and Is Inhibited by Protein Kinase A (PKA) under Pathological Hypertrophic Conditions in the Heart

The Journal of biological chemistry, 2012-11, Vol.287 (48), p.40535-40546

2012

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The C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy

Journal of molecular and cellular cardiology, 2014-01, Vol.66, p.27-40

2014

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Metabolic efficiency promotes protection from pressure overload in hearts expressing slow skeletal troponin I

Circulation. Heart failure, 2015-01, Vol.8 (1), p.119-127

2015

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p21-activated kinase improves cardiac contractility during ischemia-reperfusion concomitant with changes in troponin-T and myosin light chain 2 phosphorylation

American journal of physiology. Heart and circulatory physiology, 2012-01, Vol.302 (1), p.H224-H230

2012

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Post-translational modifications of myofilament proteins involved in length-dependent prolongation of relaxation in rabbit right ventricular myocardium

Archives of biochemistry and biophysics, 2013-07, Vol.535 (1), p.22-29

2013

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Limiting sarcolemmal Na+ entry during resuscitation from ventricular fibrillation prevents excess mitochondrial Ca2+ accumulation and attenuates myocardial injury

Journal of applied physiology (1985), 2007-07, Vol.103 (1), p.55-65

2007

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Cholesterol regulation of PIP2: why cell type is so important

Frontiers in physiology, 2013-01, Vol.3

2013

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Tetrahydrobiopterin Improves Diastolic Heart Failure by Increasing Myofilament Calcium Sensitivity

Journal of cardiac failure, 2012-08, Vol.18 (8), p.S7-S7

2012

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A‐kinase anchoring protein (AKAP)‐Lbc coordinates protein kinase A (PKA) phosphorylation and inhibition of Src homology 2 domain‐containing phosphatase 2 (Shp2)

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Inhibition of Angiotensin II-Induced Ventricular Hypertrophy and Fibrosis by Modulation of Pak1 Activity

Biophysical journal, 2012-01, Vol.102 (3), p.556a-556a

2012

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The [Beta]-arrestin-biased ligand TRV120023 inhibits angiotensin II-induced cardiac hypertrophy while preserving enhanced myofilament response to calcium

American journal of physiology. Heart and circulatory physiology, 2013-09, Vol.305 (6), p.H856

2013

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Post-Translational Modifications of Myofilament Proteins Involved in Length-Dependent Prolongation of Relaxation in Rabbit Right Ventricular Myocardium

Biophysical journal, 2012-01, Vol.102 (3), p.614a-614a

2012

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Abstract 8840: P21-Activated Kinase Improves Myocardial Performance through Modulation of Troponin-T and Myosin Light Chain 2 during Global Myocardial Ischemia and Reperfusion Injury

Circulation (New York, N.Y.), 2011-11, Vol.124 (suppl_21)

2011

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Erk1-Mediated Development of Left Ventricular Cardiac Hypertrophy in a P21 Activated Kinase-1 (Pak1) Knockout Mouse Model

Biophysical journal, 2011-02, Vol.100 (3), p.289a-289a

2011

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p21-activated kinase improves cardiac contractility during ischemia-reperfusion concomitant with changes in troponin-T and myosin light chain 2 phosphorylation

American Journal of Physiology: Cell Physiology, 2012-01, Vol.302 (1), p.H224-H230

2012

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p21-activated kinase improves cardiac contractility during ischemia-reperfusion concomitant with changes in troponin-T and myosin light chain 2 phosphorylation

American journal of physiology. Heart and circulatory physiology, 2012, Vol.71 (1)

2012

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Pak1 as a Novel Therapeutic Target for Anti-Hypertrophic Treatment in the Heart

Circulation (New York, N.Y.), 2011-11, Vol.124 (24), p.2702-2715

2011

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Pakl as a Novel Therapeutic Target for Antihypertrophic Treatment in the Heart

Circulation (New York, N.Y.), 2011, Vol.124 (24), p.2702-2715

2011

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Cholesterol regulation of PIP(2): why cell type is so important

Frontiers in physiology, 2012, Vol.3, p.492-492

2012

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Ranolazine Improves Cardiac Diastolic Dysfunction Through Modulation of Myofilament Calcium Sensitivity

P21-activated kinase in inflammatory and cardiovascular disease

Pak1 as a novel therapeutic target for antihypertrophic treatment in the heart

The β-arrestin-biased ligand TRV120023 inhibits angiotensin II-induced cardiac hypertrophy while preserving enhanced myofilament response to calcium

Tetrahydrobiopterin improves diastolic dysfunction by reversing changes in myofilament properties

Ablation of p21-activated kinase-1 in mice promotes isoproterenol-induced cardiac hypertrophy in association with activation of Erk1/2 and inhibition of protein phosphatase 2A

Src Homology 2 Domain-containing Phosphatase 2 (Shp2) Is a Component of the A-kinase-anchoring Protein (AKAP)-Lbc Complex and Is Inhibited by Protein Kinase A (PKA) under Pathological Hypertrophic Conditions in the Heart

The C-terminus of the long AKAP13 isoform (AKAP-Lbc) is critical for development of compensatory cardiac hypertrophy

Metabolic efficiency promotes protection from pressure overload in hearts expressing slow skeletal troponin I

p21-activated kinase improves cardiac contractility during ischemia-reperfusion concomitant with changes in troponin-T and myosin light chain 2 phosphorylation

Post-translational modifications of myofilament proteins involved in length-dependent prolongation of relaxation in rabbit right ventricular myocardium

Limiting sarcolemmal Na+ entry during resuscitation from ventricular fibrillation prevents excess mitochondrial Ca2+ accumulation and attenuates myocardial injury

Cholesterol regulation of PIP2: why cell type is so important

Tetrahydrobiopterin Improves Diastolic Heart Failure by Increasing Myofilament Calcium Sensitivity

A‐kinase anchoring protein (AKAP)‐Lbc coordinates protein kinase A (PKA) phosphorylation and inhibition of Src homology 2 domain‐containing phosphatase 2 (Shp2)

Inhibition of Angiotensin II-Induced Ventricular Hypertrophy and Fibrosis by Modulation of Pak1 Activity

The [Beta]-arrestin-biased ligand TRV120023 inhibits angiotensin II-induced cardiac hypertrophy while preserving enhanced myofilament response to calcium

Post-Translational Modifications of Myofilament Proteins Involved in Length-Dependent Prolongation of Relaxation in Rabbit Right Ventricular Myocardium

Abstract 8840: P21-Activated Kinase Improves Myocardial Performance through Modulation of Troponin-T and Myosin Light Chain 2 during Global Myocardial Ischemia and Reperfusion Injury

Erk1-Mediated Development of Left Ventricular Cardiac Hypertrophy in a P21 Activated Kinase-1 (Pak1) Knockout Mouse Model

p21-activated kinase improves cardiac contractility during ischemia-reperfusion concomitant with changes in troponin-T and myosin light chain 2 phosphorylation

p21-activated kinase improves cardiac contractility during ischemia-reperfusion concomitant with changes in troponin-T and myosin light chain 2 phosphorylation

Pak1 as a Novel Therapeutic Target for Anti-Hypertrophic Treatment in the Heart

Pakl as a Novel Therapeutic Target for Antihypertrophic Treatment in the Heart

Cholesterol regulation of PIP(2): why cell type is so important

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