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Azithromycin analogue CSY0073 attenuates lung inflammation induced by LPS challenge

British journal of pharmacology, 2014-04, Vol.171 (7), p.1783-1794

2014

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WS06.4 miR-9 and ANO1: Therapeutic targets in cystic fibrosis?

Journal of cystic fibrosis, 2015-06, Vol.14, p.S11-S11

2015

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Challenges of personalized medicine for cystic fibrosis

Archives de pédiatrie : organe officiel de la Société française de pédiatrie, 2015-07, Vol.22 (7), p.778-786

2015

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WS06.5 New insights into the pathogenesis and potential therapies of cystic fibrosis (CF) bone disease

Journal of cystic fibrosis, 2016-06, Vol.15, p.S10-S10

2016

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WS17.03 An alternative mutation agnostic therapy for cystic fibrosis with oligonucleotide antisense

Journal of cystic fibrosis, 2023-06, Vol.22, p.S32-S32

2023

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Morphological analysis of the trachea and pattern of breathing in βENaC-Tg mice

Respiratory physiology & neurobiology, 2011-09, Vol.178 (2), p.346-348

2011

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628 A new oligonucleotide antisense therapy for all patients with cystic fibrosis

Journal of cystic fibrosis, 2022-10, Vol.21, p.S346-S346

2022

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ePS05.5 Cystic fibrosis bone disease: Is the CFTR corrector C18 an option for therapy?

Journal of cystic fibrosis, 2015-06, Vol.14, p.S51-S51

2015

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WS6.7 Cystic fibrosis and bone disease: defective osteoblast maturation with the F508del mutation in CFTR

Journal of cystic fibrosis, 2014-06, Vol.13, p.S13-S13

2014

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WS15.4 Study of the chloride channel ANO1 in cystic fibrosis context

Journal of cystic fibrosis, 2012-06, Vol.11, p.S33-S33

2012

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Développement d’une approche antisens pour activer le canal alternatif TMEM16a chez les patients atteints de mucoviscidose

Revue des maladies respiratoires, 2023-02, Vol.40 (2), p.132-132

2023

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Fluticasone reduces IL-6 and IL-8 production of cystic fibrosis bronchial epithelial cells via IKK-β kinase pathway

The European respiratory journal, 2003-04, Vol.21 (4), p.574-581

2003

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Étude du potentiel thérapeutique du TSB ANO1 dans la mucoviscidose

Revue des maladies respiratoires, 2022-02, Vol.39 (2), p.124-124

2022

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Histone deacetylases (HDACs) and IFRD1 in CF airway epithelial cell models

Journal of cystic fibrosis, 2010, Vol.9, p.S46-S46

2010

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Étude du rôle du facteur de virulence ExoY de Pseudomonas aeruginosa dans la réponse des cellules pulmonaires des patients atteints de mucoviscidose

Revue des maladies respiratoires, 2023-02, Vol.40 (2), p.135-136

2023

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153 Regulation of corticosteroid binding globulin (CBG) in the inflammatory context of cystic fibrosis

Journal of cystic fibrosis, 2012-06, Vol.11, p.S95-S95

2012

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Airway epithelial cell inflammatory signalling in cystic fibrosis

The international journal of biochemistry & cell biology, 2008, Vol.40 (9), p.1703-1715

2008

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Azithromycin is not able to decrease inflammatory process in cystic fibrosis human bronchial epithelial cells

Journal of cystic fibrosis, 2009, Vol.8, p.S54-S54

2009

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Abnormal TRAF4 and TMEM16a expression during tracheal development in CFTR-deficient mice

Journal of cystic fibrosis, 2010, Vol.9, p.S16-S16

2010

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Hepatic corticosteroid-binding globulin expression in CF patients

Journal of cystic fibrosis, 2010, Vol.9, p.S19-S19

2010

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Azithromycin is not able to decrease inflammatory process in cystic fibrosis cells

Journal of cystic fibrosis, 2008-06, Vol.7, p.S54-S54

2008

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Weak anti-inflammatory effects of glucocorticoids on CF bronchial epithelial cells

Journal of cystic fibrosis, 2009, Vol.8, p.S54-S54

2009

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Involvement of toll-like receptor 4 in the inflammatory reaction induced by hydroxyapatite particles

Biomaterials, 2007-01, Vol.28 (3), p.400-404

2007

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A new candidate biomarker of inflammation in CF airway: Amphiregulin

Journal of cystic fibrosis, 2008-06, Vol.7, p.S56-S56

2008

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Defective CFTR Cl− channel enhances proteasomal degradation and reduces NF-κB activity in lung epithelial cells exposed to oxidative stress

Journal of cystic fibrosis, 2008-06, Vol.7, p.S52-S52

2008

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Azithromycin analogue CSY0073 attenuates lung inflammation induced by LPS challenge

WS06.4 miR-9 and ANO1: Therapeutic targets in cystic fibrosis?

Challenges of personalized medicine for cystic fibrosis

WS06.5 New insights into the pathogenesis and potential therapies of cystic fibrosis (CF) bone disease

WS17.03 An alternative mutation agnostic therapy for cystic fibrosis with oligonucleotide antisense

Morphological analysis of the trachea and pattern of breathing in βENaC-Tg mice

628 A new oligonucleotide antisense therapy for all patients with cystic fibrosis

ePS05.5 Cystic fibrosis bone disease: Is the CFTR corrector C18 an option for therapy?

WS6.7 Cystic fibrosis and bone disease: defective osteoblast maturation with the F508del mutation in CFTR

WS15.4 Study of the chloride channel ANO1 in cystic fibrosis context

Développement d’une approche antisens pour activer le canal alternatif TMEM16a chez les patients atteints de mucoviscidose

Fluticasone reduces IL-6 and IL-8 production of cystic fibrosis bronchial epithelial cells via IKK-β kinase pathway

Étude du potentiel thérapeutique du TSB ANO1 dans la mucoviscidose

Histone deacetylases (HDACs) and IFRD1 in CF airway epithelial cell models

Étude du rôle du facteur de virulence ExoY de Pseudomonas aeruginosa dans la réponse des cellules pulmonaires des patients atteints de mucoviscidose

153 Regulation of corticosteroid binding globulin (CBG) in the inflammatory context of cystic fibrosis

Airway epithelial cell inflammatory signalling in cystic fibrosis

Azithromycin is not able to decrease inflammatory process in cystic fibrosis human bronchial epithelial cells

Abnormal TRAF4 and TMEM16a expression during tracheal development in CFTR-deficient mice

Hepatic corticosteroid-binding globulin expression in CF patients

Azithromycin is not able to decrease inflammatory process in cystic fibrosis cells

Weak anti-inflammatory effects of glucocorticoids on CF bronchial epithelial cells

Involvement of toll-like receptor 4 in the inflammatory reaction induced by hydroxyapatite particles

A new candidate biomarker of inflammation in CF airway: Amphiregulin

Defective CFTR Cl− channel enhances proteasomal degradation and reduces NF-κB activity in lung epithelial cells exposed to oxidative stress

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