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Perforin and granzymes: function, dysfunction and human pathology

Nature reviews. Immunology, 2015-06, Vol.15 (6), p.388-400

2015

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Intratumoral Copper Modulates PD-L1 Expression and Influences Tumor Immune Evasion

Cancer research (Chicago, Ill.), 2020-10, Vol.80 (19), p.4129-4144

2020

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TRAIL+ NK Cells Control CD4+ T Cell Responses during Chronic Viral Infection to Limit Autoimmunity

Immunity (Cambridge, Mass.), 2014-10, Vol.41 (4), p.646-656

2014

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Failed CTL/NK cell killing and cytokine hypersecretion are directly linked through prolonged synapse time

The Journal of experimental medicine, 2015-03, Vol.212 (3), p.307-317

2015

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Lipid order and charge protect killer T cells from accidental death

Nature communications, 2019-11, Vol.10 (1), p.5396-13, Article 5396

2019

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Functional significance of the perforin/granzyme cell death pathway

Nature reviews. Immunology, 2002-10, Vol.2 (10), p.735-747

2002

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Revisiting T Cell Tolerance as a Checkpoint Target for Cancer Immunotherapy

Frontiers in immunology, 2020-09, Vol.11, p.589641-589641

2020

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Reprogrammed CRISPR-Cas13b suppresses SARS-CoV-2 replication and circumvents its mutational escape through mismatch tolerance

Nature communications, 2021-07, Vol.12 (1), p.1-16, Article 4270

2021

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Conformational changes during pore formation by the perforin-related protein pleurotolysin

PLoS biology, 2015-02, Vol.13 (2), p.e1002049

2015

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Perforin forms transient pores on the target cell plasma membrane to facilitate rapid access of granzymes during killer cell attack

Blood, 2013-04, Vol.121 (14), p.2659-2668

2013

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Real-time visualization of perforin nanopore assembly

Nature nanotechnology, 2017-05, Vol.12 (5), p.467-473

2017

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Perforin: structure, function, and role in human immunopathology

Immunological reviews, 2010-05, Vol.235 (1), p.35-54

2010

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Agonist immunotherapy restores T cell function following MEK inhibition improving efficacy in breast cancer

Nature communications, 2017-09, Vol.8 (1), p.606-18, Article 606

2017

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Perforinopathy: a spectrum of human immune disease caused by defective perforin delivery or function

Frontiers in immunology, 2013-12, Vol.4, p.441-441

2013

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The structural basis for membrane binding and pore formation by lymphocyte perforin

Nature (London), 2010-11, Vol.468 (7322), p.447-451

2010

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PVRIG is a novel natural killer cell immune checkpoint receptor in acute myeloid leukemia

Haematologica (Roma), 2021-12, Vol.106 (12), p.3115-3124

2021

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Natural killer cells kill extracellular Pseudomonas aeruginosa using contact-dependent release of granzymes B and H

PLoS pathogens, 2022-02, Vol.18 (2), p.e1010325

2022

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Differential effects of BTK inhibitors ibrutinib and zanubrutinib on NK-cell effector function in patients with mantle cell lymphoma

Haematologica (Roma), 2020-02, Vol.105 (2), p.e76-e79

2020

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Functional crosstalk between type I and II interferon through the regulated expression of STAT1

PLoS biology, 2010-04, Vol.8 (4), p.e1000361-e1000361

2010

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The immunostimulatory effect of lenalidomide on NK-cell function is profoundly inhibited by concurrent dexamethasone therapy

Blood, 2011-02, Vol.117 (5), p.1605-1613

2011

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Antigen-driven EGR2 expression is required for exhausted CD8 + T cell stability and maintenance

Nature communications, 2021-05, Vol.12 (1), p.2782-2782, Article 2782

2021

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Functional dissection of the granzyme family: cell death and inflammation

Immunological reviews, 2010-05, Vol.235 (1), p.73-92

2010

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Myeloma natural killer cells are exhausted and have impaired regulation of activation

Haematologica (Roma), 2021-09, Vol.106 (9), p.2522-2526

2021

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MACPF/CDC family of pore-forming toxins

Cellular microbiology, 2008-09, Vol.10 (9), p.1765-1774

2008

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The efficacy of combination treatment with elotuzumab and lenalidomide is dependent on crosstalk between natural killer cells, monocytes and myeloma cells

Haematologica (Roma), 2023-01, Vol.108 (1), p.83-97

2023

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Perforin and granzymes: function, dysfunction and human pathology

Intratumoral Copper Modulates PD-L1 Expression and Influences Tumor Immune Evasion

TRAIL+ NK Cells Control CD4+ T Cell Responses during Chronic Viral Infection to Limit Autoimmunity

Failed CTL/NK cell killing and cytokine hypersecretion are directly linked through prolonged synapse time

Lipid order and charge protect killer T cells from accidental death

Functional significance of the perforin/granzyme cell death pathway

Revisiting T Cell Tolerance as a Checkpoint Target for Cancer Immunotherapy

Reprogrammed CRISPR-Cas13b suppresses SARS-CoV-2 replication and circumvents its mutational escape through mismatch tolerance

Conformational changes during pore formation by the perforin-related protein pleurotolysin

Perforin forms transient pores on the target cell plasma membrane to facilitate rapid access of granzymes during killer cell attack

Real-time visualization of perforin nanopore assembly

Perforin: structure, function, and role in human immunopathology

Agonist immunotherapy restores T cell function following MEK inhibition improving efficacy in breast cancer

Perforinopathy: a spectrum of human immune disease caused by defective perforin delivery or function

The structural basis for membrane binding and pore formation by lymphocyte perforin

PVRIG is a novel natural killer cell immune checkpoint receptor in acute myeloid leukemia

Natural killer cells kill extracellular Pseudomonas aeruginosa using contact-dependent release of granzymes B and H

Differential effects of BTK inhibitors ibrutinib and zanubrutinib on NK-cell effector function in patients with mantle cell lymphoma

Functional crosstalk between type I and II interferon through the regulated expression of STAT1

The immunostimulatory effect of lenalidomide on NK-cell function is profoundly inhibited by concurrent dexamethasone therapy

Antigen-driven EGR2 expression is required for exhausted CD8 + T cell stability and maintenance

Functional dissection of the granzyme family: cell death and inflammation

Myeloma natural killer cells are exhausted and have impaired regulation of activation

MACPF/CDC family of pore-forming toxins

The efficacy of combination treatment with elotuzumab and lenalidomide is dependent on crosstalk between natural killer cells, monocytes and myeloma cells

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